William A. Truitt

3.1k total citations
46 papers, 2.4k citations indexed

About

William A. Truitt is a scholar working on Cellular and Molecular Neuroscience, Cognitive Neuroscience and Social Psychology. According to data from OpenAlex, William A. Truitt has authored 46 papers receiving a total of 2.4k indexed citations (citations by other indexed papers that have themselves been cited), including 25 papers in Cellular and Molecular Neuroscience, 17 papers in Cognitive Neuroscience and 11 papers in Social Psychology. Recurrent topics in William A. Truitt's work include Neurotransmitter Receptor Influence on Behavior (19 papers), Neuroscience and Neuropharmacology Research (12 papers) and Stress Responses and Cortisol (11 papers). William A. Truitt is often cited by papers focused on Neurotransmitter Receptor Influence on Behavior (19 papers), Neuroscience and Neuropharmacology Research (12 papers) and Stress Responses and Cortisol (11 papers). William A. Truitt collaborates with scholars based in United States, United Kingdom and Poland. William A. Truitt's co-authors include Lique M. Coolen, Anantha Shekhar, Philip L. Johnson, Stephanie D. Fitz, Amy D. Dietrich, Kevin E. McKenna, Julien Allard, Andrei I. Molosh, Tammy J. Sajdyk and Donald G. Rainnie and has published in prestigious journals such as Science, Nature Medicine and Journal of Neuroscience.

In The Last Decade

William A. Truitt

46 papers receiving 2.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
William A. Truitt United States 25 888 758 690 511 500 46 2.4k
Irina Antonijevic Germany 28 667 0.8× 947 1.2× 723 1.0× 503 1.0× 590 1.2× 62 3.1k
Franco J. Vaccarino Canada 35 941 1.1× 2.4k 3.1× 897 1.3× 304 0.6× 796 1.6× 103 4.6k
Christopher V. Dayas Australia 31 1.1k 1.3× 1.1k 1.5× 1.1k 1.6× 469 0.9× 778 1.6× 66 3.6k
Sérgio Tufik Brazil 33 2.1k 2.4× 738 1.0× 1.5k 2.1× 1.1k 2.2× 168 0.3× 75 3.5k
László Lénárd Hungary 26 636 0.7× 1.2k 1.6× 675 1.0× 132 0.3× 357 0.7× 141 2.9k
Jim R. Fadel United States 35 1.7k 1.9× 1.0k 1.3× 1.2k 1.8× 744 1.5× 303 0.6× 74 3.0k
Wayne G. Brake Canada 29 619 0.7× 1.2k 1.5× 219 0.3× 182 0.4× 1.1k 2.3× 61 3.7k
S. Hansen Sweden 29 329 0.4× 741 1.0× 394 0.6× 167 0.3× 1.2k 2.3× 57 2.4k
Daina Economidou Italy 29 1.0k 1.1× 2.5k 3.2× 376 0.5× 237 0.5× 375 0.8× 34 3.2k
Stewart D. Clark United States 20 1.1k 1.2× 1.2k 1.6× 552 0.8× 362 0.7× 262 0.5× 44 2.1k

Countries citing papers authored by William A. Truitt

Since Specialization
Citations

This map shows the geographic impact of William A. Truitt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by William A. Truitt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites William A. Truitt more than expected).

Fields of papers citing papers by William A. Truitt

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by William A. Truitt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by William A. Truitt. The network helps show where William A. Truitt may publish in the future.

Co-authorship network of co-authors of William A. Truitt

This figure shows the co-authorship network connecting the top 25 collaborators of William A. Truitt. A scholar is included among the top collaborators of William A. Truitt based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with William A. Truitt. William A. Truitt is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Bernabe, Cristian, Izabela Facco Caliman, Andrei I. Molosh, et al.. (2024). Identification of a novel perifornical-hypothalamic-area-projecting serotonergic system that inhibits innate panic and conditioned fear responses. Translational Psychiatry. 14(1). 60–60. 6 indexed citations
2.
Hauser, Sheketha R., Gerald A. Deehan, Robert A. Waeiss, et al.. (2023). Inhibitory and excitatory alcohol-seeking cues distinct roles in behavior, neurochemistry, and mesolimbic pathway in alcohol preferring (P) rats. Drug and Alcohol Dependence. 246. 109858–109858. 3 indexed citations
3.
Hauser, Sheketha R., Patrick J. Mulholland, William A. Truitt, et al.. (2021). Adolescent Intermittent Ethanol (AIE) Enhances the Dopaminergic Response to Ethanol within the Mesolimbic Pathway during Adulthood: Alterations in Cholinergic/Dopaminergic Genes Expression in the Nucleus Accumbens Shell. International Journal of Molecular Sciences. 22(21). 11733–11733. 8 indexed citations
4.
Burke, Andrew R., et al.. (2021). Role of Basolateral Amygdalar Somatostatin 2 Receptors in a Rat Model of Chronic Anxiety. Neuroscience. 477. 40–49. 4 indexed citations
5.
Hauser, Sheketha R., Simon N. Katner, Robert A. Waeiss, et al.. (2020). Selective breeding for high alcohol preference is associated with increased sensitivity to cannabinoid reward within the nucleus accumbens shell. Pharmacology Biochemistry and Behavior. 197. 173002–173002. 2 indexed citations
6.
Hauser, Sheketha R., Gerald A. Deehan, Robert A. Waeiss, et al.. (2019). Conditioned stimuli affect ethanol-seeking by female alcohol-preferring (P) rats: the role of repeated-deprivations, cue-pretreatment, and cue-temporal intervals. Psychopharmacology. 236(9). 2835–2846. 7 indexed citations
7.
Deehan, Gerald A., Sheketha R. Hauser, Robert A. Waeiss, et al.. (2015). Co-administration of ethanol and nicotine: the enduring alterations in the rewarding properties of nicotine and glutamate activity within the mesocorticolimbic system of female alcohol-preferring (P) rats. Psychopharmacology. 232(23). 4293–4302. 25 indexed citations
8.
Truitt, William A., Sheketha R. Hauser, Gerald A. Deehan, et al.. (2014). Ethanol and nicotine interaction within the posterior ventral tegmental area in male and female alcohol-preferring rats: evidence of synergy and differential gene activation in the nucleus accumbens shell. Psychopharmacology. 232(3). 639–649. 35 indexed citations
9.
Toalston, Jamie E., Gerald A. Deehan, Sheketha R. Hauser, et al.. (2014). Reinforcing Properties and Neurochemical Response of Ethanol within the Posterior Ventral Tegmental Area Are Enhanced in Adulthood by Periadolescent Ethanol Consumption. Journal of Pharmacology and Experimental Therapeutics. 351(2). 317–326. 25 indexed citations
10.
Molosh, Andrei I., Tammy J. Sajdyk, William A. Truitt, et al.. (2013). NPY Y1 Receptors Differentially Modulate GABAA and NMDA Receptors via Divergent Signal-Transduction Pathways to Reduce Excitability of Amygdala Neurons. Neuropsychopharmacology. 38(7). 1352–1364. 53 indexed citations
11.
Deehan, Gerald A., Sheketha R. Hauser, Jessica A. Wilden, William A. Truitt, & Zachary A. Rodd. (2013). Elucidating the biological basis for the reinforcing actions of alcohol in the mesolimbic dopamine system: the role of active metabolites of alcohol. Frontiers in Behavioral Neuroscience. 7. 104–104. 32 indexed citations
12.
Johnson, Philip L., Andrei I. Molosh, Stephanie D. Fitz, William A. Truitt, & Anantha Shekhar. (2012). Orexin, stress, and anxiety/panic states. Progress in brain research. 198. 133–161. 185 indexed citations
13.
Ding, Zheng‐Ming, Simon N. Katner, Zachary A. Rodd, et al.. (2012). Repeated exposure of the posterior ventral tegmental area to nicotine increases the sensitivity of local dopamine neurons to the stimulating effects of ethanol. Alcohol. 46(3). 217–223. 18 indexed citations
14.
Johnson, Philip L., Brian C. Samuels, Stephanie D. Fitz, et al.. (2012). Orexin 1 receptors are a novel target to modulate panic responses and the panic brain network. Physiology & Behavior. 107(5). 733–742. 95 indexed citations
15.
Molosh, Andrei I., Philip L. Johnson, Rachel C. Dirks, et al.. (2012). Orexin-A induces anxiety-like behavior through interactions with glutamatergic receptors in the bed nucleus of the stria terminalis of rats. Physiology & Behavior. 107(5). 726–732. 95 indexed citations
16.
Lukkes, Jodi L., Daniel R. Staub, Amy D. Dietrich, et al.. (2011). Topographical distribution of corticotropin-releasing factor type 2 receptor-like immunoreactivity in the rat dorsal raphe nucleus: co-localization with tryptophan hydroxylase. Neuroscience. 183. 47–63. 29 indexed citations
17.
Johnson, Philip L., William A. Truitt, Stephanie D. Fitz, et al.. (2009). A key role for orexin in panic anxiety. Nature Medicine. 16(1). 111–115. 316 indexed citations
18.
Johnson, Philip L., C.A. Lowry, William A. Truitt, & Anantha Shekhar. (2008). Disruption of GABAergic tone in the dorsomedial hypothalamus attenuates responses in a subset of serotonergic neurons in the dorsal raphe nucleus following lactate-induced panic. Journal of Psychopharmacology. 22(6). 642–652. 68 indexed citations
19.
Truitt, William A., et al.. (2003). Progesterone attenuates the effect of the 5-HT1A receptor agonist, 8-OH-DPAT, and of mild restraint on lordosis behavior. Brain Research. 974(1-2). 202–211. 29 indexed citations
20.
Maswood, Sharmin, et al.. (1999). Estrous cycle modulation of extracellular serotonin in mediobasal hypothalamus: role of the serotonin transporter and terminal autoreceptors. Brain Research. 831(1-2). 146–154. 94 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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