Wenwen Wei

1.6k total citations · 1 hit paper
51 papers, 1.1k citations indexed

About

Wenwen Wei is a scholar working on Molecular Biology, Oncology and Immunology. According to data from OpenAlex, Wenwen Wei has authored 51 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Molecular Biology, 14 papers in Oncology and 14 papers in Immunology. Recurrent topics in Wenwen Wei's work include Acute Myeloid Leukemia Research (9 papers), CAR-T cell therapy research (8 papers) and Immune Cell Function and Interaction (7 papers). Wenwen Wei is often cited by papers focused on Acute Myeloid Leukemia Research (9 papers), CAR-T cell therapy research (8 papers) and Immune Cell Function and Interaction (7 papers). Wenwen Wei collaborates with scholars based in China, United States and New Zealand. Wenwen Wei's co-authors include Wanpeng Xi, Bo Zhang, Kunsong Chen, Jiyuan Shen, Ian Ferguson, Changjie Xu, Xudong Zhao, Dong Yang, Bin Sun and Xin Wang and has published in prestigious journals such as Journal of Clinical Investigation, Blood and Journal of Agricultural and Food Chemistry.

In The Last Decade

Wenwen Wei

50 papers receiving 1.1k citations

Hit Papers

NKG2D-CAR T cells eliminate senescent cells in aged mice ... 2023 2026 2024 2025 2023 25 50 75 100

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Wenwen Wei China 15 505 334 144 143 137 51 1.1k
Niaz Mahmood Canada 15 557 1.1× 109 0.3× 252 1.8× 153 1.1× 84 0.6× 38 1.2k
Jing Cheng China 16 400 0.8× 125 0.4× 59 0.4× 61 0.4× 41 0.3× 34 877
Gongbo Li United States 20 333 0.7× 176 0.5× 38 0.3× 397 2.8× 205 1.5× 56 969
Jonghwa Won South Korea 17 359 0.7× 76 0.2× 60 0.4× 191 1.3× 172 1.3× 52 822
Yan Zhu China 16 648 1.3× 379 1.1× 333 2.3× 165 1.2× 47 0.3× 50 1.1k
Jiaping Xue United States 17 614 1.2× 236 0.7× 115 0.8× 92 0.6× 106 0.8× 23 977
Sivaprakasam Balasubramanian United States 21 817 1.6× 62 0.2× 152 1.1× 261 1.8× 130 0.9× 30 1.4k
Arvind Pandey India 16 579 1.1× 114 0.3× 158 1.1× 183 1.3× 72 0.5× 28 1.1k

Countries citing papers authored by Wenwen Wei

Since Specialization
Citations

This map shows the geographic impact of Wenwen Wei's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wenwen Wei with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wenwen Wei more than expected).

Fields of papers citing papers by Wenwen Wei

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Wenwen Wei. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wenwen Wei. The network helps show where Wenwen Wei may publish in the future.

Co-authorship network of co-authors of Wenwen Wei

This figure shows the co-authorship network connecting the top 25 collaborators of Wenwen Wei. A scholar is included among the top collaborators of Wenwen Wei based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Wenwen Wei. Wenwen Wei is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Deng, Yue, Xixi Liu, Yang Xiao, et al.. (2025). IL-17–producing γδ T cells in the tumor microenvironment promote radioresistance in mice. Journal of Clinical Investigation. 135(24). 1 indexed citations
3.
Wang, Wenjing, et al.. (2024). Unveiling the mechanism of ultraviolet-induced degradation in silicon heterojunction solar cells. Solar Energy Materials and Solar Cells. 276. 113062–113062. 7 indexed citations
4.
Wang, Jing, Ye He, Mengyuan Zhang, et al.. (2024). The degree of risk factor and accumulation effect for large niche in individuals after cesarean section. BMC Pregnancy and Childbirth. 24(1). 38–38. 2 indexed citations
5.
Li, Jie, Bin Zhang, Zhiyuan Zhou, et al.. (2024). Stabilization of KPNB1 by deubiquitinase USP7 promotes glioblastoma progression through the YBX1-NLGN3 axis. Journal of Experimental & Clinical Cancer Research. 43(1). 28–28. 11 indexed citations
6.
Ye, Wenle, Xin Huang, Jiansong Huang, et al.. (2023). Glutamate dehydrogenase 1: A novel metabolic target in inhibiting acute myeloid leukaemia progression. British Journal of Haematology. 202(3). 566–577. 10 indexed citations
7.
Zhang, Yi, Wenwen Wei, Bin Zhang, et al.. (2023). STI-8591, a Novel Tyrosine Kinase Inhibitor, Shows Excellent Antileukemic Activity in FLT3-Mutated (mut) Acute Myeloid Leukemia (AML). Blood. 142(Supplement 1). 5010–5010.
8.
Wei, Wenwen, Dong Yang, Bin Sun, et al.. (2023). SECTM1-based CAR T cells enriched with CD7-low/negative subsets exhibit efficacy in CD7-positive malignancies. Blood Advances. 7(13). 2941–2951. 6 indexed citations
9.
Yang, Dong, Bin Sun, Shirong Li, et al.. (2023). NKG2D-CAR T cells eliminate senescent cells in aged mice and nonhuman primates. Science Translational Medicine. 15(709). eadd1951–eadd1951. 114 indexed citations breakdown →
10.
Mao, Shihui, Qian Yu, Wenwen Wei, et al.. (2023). FLOT1 knockdown inhibits growth of AML cells through triggering apoptosis and pyroptosis. Annals of Hematology. 102(3). 583–595. 7 indexed citations
11.
Mao, Shihui, Jiajia Pan, Wenwen Wei, et al.. (2023). CPT1B, a metabolic molecule, is also an independent risk factor in CN-AML. Cancer Biomarkers. 37(3). 133–145. 4 indexed citations
12.
Li, Fenglin, Qing Ling, Ying Chen, et al.. (2023). Dihydropyrimidinase‐like 2 can serve as a novel therapeutic target and prognostic biomarker in acute myeloid leukemia. Cancer Medicine. 12(7). 8319–8330. 8 indexed citations
13.
Wei, Wenwen, Jiajia Pan, Jinghan Wang, et al.. (2023). circSLC25A13 acts as a ceRNA to regulate AML progression via miR‐616‐3p/ADCY2 axis. Molecular Carcinogenesis. 62(10). 1546–1562. 4 indexed citations
14.
Chen, Xi, et al.. (2022). Immune Checkpoint Inhibitors in Peripheral T-Cell Lymphoma. Frontiers in Pharmacology. 13. 869488–869488. 18 indexed citations
15.
Lin, Xiangjie, Jinghan Wang, Xin Huang, et al.. (2021). Global, regional, and national burdens of leukemia from 1990 to 2017: a systematic analysis of the global burden of disease 2017 study. Aging. 13(7). 10468–10489. 35 indexed citations
16.
Li, Fenglin, Xiang Zhang, Shihui Mao, et al.. (2021). MAP4K1 functions as a tumor promotor and drug mediator for AML via modulation of DNA damage/repair system and MAPK pathway. EBioMedicine. 69. 103441–103441. 10 indexed citations
17.
Wei, Wenwen, Xin Wang, Fangyu Zhou, et al.. (2021). Gestational Diabetes Mellitus: The Genetic Susceptibility Behind the Disease. Hormone and Metabolic Research. 53(8). 489–498. 19 indexed citations
18.
19.
Xue, Cong, Wenwen Wei, Peng Sun, et al.. (2018). Pretreatment anti-Mullerian hormone-based nomogram predicts menstruation status after chemotherapy for premenopausal women with hormone receptor-positive early breast cancer. Breast Cancer Research and Treatment. 173(3). 619–628. 10 indexed citations
20.
Shen, Zhijun, et al.. (2017). Association of choroidal thickness with early stages of diabetic retinopathy in type 2 diabetes. International Journal of Ophthalmology. 10(4). 613–618. 21 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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