Wenqi Zuo
- Cell Biology top 5%
- Microtubule and mitosis dynamics 3
- Biophysics top 10%
- Electromagnetic Fields and Biological Effects 3
-
- DNA Repair Mechanisms 3
- Protist diversity and phylogeny 3
-
- Hearing, Cochlea, Tinnitus, Genetics 8
-
- Cerebral Venous Sinus Thrombosis 4
- Vestibular and auditory disorders 4
-
- Noise Effects and Management 3
- Co-authors
- N. Ronald MorrisXin XiangGongshe HanBo LiuJun ZhangVladimir P. EfimovDonald A. WinkelmannYuanyuan Zhang
- Cited by
- Cell BiologyBiophysicsAging
- Journals
- SHILAP Revista de lepidopterología (1 paper)Current Biology (2 papers)BMC Bioinformatics (1 paper)
- Partner nations
- ChinaUnited StatesSaudi Arabia
In The Last Decade
Wenqi Zuo
21 papers receiving 453 citations
Peers
Comparison fields: 5 of 66
- Cell Biology 276
- Biophysics 42
- Aging 9
- Molecular Biology 335
- Sensory Systems 19
Countries citing papers authored by Wenqi Zuo
This map shows the geographic impact of Wenqi Zuo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wenqi Zuo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wenqi Zuo more than expected).
Fields of papers citing papers by Wenqi Zuo
This network shows the impact of papers produced by Wenqi Zuo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wenqi Zuo. The network helps show where Wenqi Zuo may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Wenqi Zuo, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 0 | |
| 2 | 2025 | 1 | |
| 3 | 2024 | 1 | |
| 4 | 2023 | 0 | |
| 5 | 2023 | 6 | |
| 6 | 2023 | 2 | |
| 7 | 2022 | 6 | |
| 8 | 2022 | 13 | |
| 9 | 2021 | 4 | |
| 10 | 2020 | 14 | |
| 11 | Integrative gene expression profiling reveals that dysregulated triple microRNAs confer paclitaxel resistance in non-small cell lung cancer via co-targeting MAPT | 2019 | 1 |
| 12 | 2019 | 15 | |
| 13 | 2019 | 12 | |
| 14 | 2017 | 9 | |
| 15 | 2017 | 13 | |
| 16 | 2015 | 29 | |
| 17 | 2015 | 14 | |
| 18 | 2001 | 149 | |
| 19 | 2000 | 90 | |
| 20 | 1999 | 74 |
About Wenqi Zuo
Wenqi Zuo is a scholar working on Sensory Systems, Biophysics and Neurology, having authored 23 papers that have together received 468 indexed citations. Recurring topics across this work include Hearing, Cochlea, Tinnitus, Genetics (8 papers), Cerebral Venous Sinus Thrombosis (4 papers), Vestibular and auditory disorders (4 papers), DNA Repair Mechanisms (3 papers), Electromagnetic Fields and Biological Effects (3 papers), Protist diversity and phylogeny (3 papers), Microtubule and mitosis dynamics (3 papers) and Noise Effects and Management (3 papers). The work is most often cited by research in Cell Biology (276 citations), Biophysics (42 citations) and Aging (9 citations). Wenqi Zuo has collaborated with scholars based in China, United States and Saudi Arabia. Frequent co-authors include N. Ronald Morris, Xin Xiang, Gongshe Han, Bo Liu, Jun Zhang, Vladimir P. Efimov, Donald A. Winkelmann, Yuanyuan Zhang, Weijia Kong and Xueyan Zhao. Their work appears in journals such as SHILAP Revista de lepidopterología, Current Biology and BMC Bioinformatics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.