Weinstein Ib

2.2k total citations
52 papers, 1.9k citations indexed

About

Weinstein Ib is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Weinstein Ib has authored 52 papers receiving a total of 1.9k indexed citations (citations by other indexed papers that have themselves been cited), including 23 papers in Molecular Biology, 12 papers in Oncology and 9 papers in Cancer Research. Recurrent topics in Weinstein Ib's work include Cancer-related Molecular Pathways (6 papers), DNA and Nucleic Acid Chemistry (5 papers) and Carcinogens and Genotoxicity Assessment (5 papers). Weinstein Ib is often cited by papers focused on Cancer-related Molecular Pathways (6 papers), DNA and Nucleic Acid Chemistry (5 papers) and Carcinogens and Genotoxicity Assessment (5 papers). Weinstein Ib collaborates with scholars based in United States, South Korea and Israel. Weinstein Ib's co-authors include Dezider Grünberger, Nadir Arber, Thomas R. Sutter, Doriano Fabbro, Silu Wang, Peter R. Holt, Melissa D. Begg, Yu Zhang, Hanina Hibshoosh and Ira Schieren and has published in prestigious journals such as Gastroenterology, British Journal of Cancer and Molecular Pharmacology.

In The Last Decade

Weinstein Ib

52 papers receiving 1.7k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Weinstein Ib United States 25 1.3k 625 386 169 155 52 1.9k
Brent C. Behrens United States 15 1.2k 0.9× 768 1.2× 222 0.6× 205 1.2× 73 0.5× 19 2.1k
Karen G. Louie United States 9 1.0k 0.8× 672 1.1× 188 0.5× 162 1.0× 57 0.4× 12 1.7k
Shigeki Tsuchida Japan 30 2.3k 1.8× 631 1.0× 358 0.9× 175 1.0× 158 1.0× 116 3.2k
G. Timothy Bowden United States 29 1.2k 1.0× 429 0.7× 477 1.2× 85 0.5× 235 1.5× 57 2.2k
Sabina Carla Righetti Italy 19 1.2k 1.0× 761 1.2× 252 0.7× 144 0.9× 105 0.7× 28 1.8k
Renza Vento Italy 34 1.9k 1.5× 731 1.2× 863 2.2× 148 0.9× 149 1.0× 97 2.8k
Dennis S. France United States 21 1.6k 1.2× 524 0.8× 437 1.1× 192 1.1× 115 0.7× 50 2.6k
Pamela A. Havre United States 20 897 0.7× 379 0.6× 267 0.7× 120 0.7× 85 0.5× 33 1.4k
Alan J. Townsend United States 33 1.9k 1.5× 756 1.2× 322 0.8× 157 0.9× 67 0.4× 60 2.7k
G. Tim Bowden United States 26 1.1k 0.9× 568 0.9× 562 1.5× 90 0.5× 231 1.5× 40 2.0k

Countries citing papers authored by Weinstein Ib

Since Specialization
Citations

This map shows the geographic impact of Weinstein Ib's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Weinstein Ib with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Weinstein Ib more than expected).

Fields of papers citing papers by Weinstein Ib

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Weinstein Ib. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Weinstein Ib. The network helps show where Weinstein Ib may publish in the future.

Co-authorship network of co-authors of Weinstein Ib

This figure shows the co-authorship network connecting the top 25 collaborators of Weinstein Ib. A scholar is included among the top collaborators of Weinstein Ib based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Weinstein Ib. Weinstein Ib is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Harris, N., Marjorie Pick, V Deutsch, et al.. (1998). c-K-ras transformed rat enterocytes (R1) are more sensitive than normal enterocytes (IEC18) to growth inhibition and appptosis-induced by sulindac sulfone and nimesulid. Gastroenterology. 114. A607–A607. 3 indexed citations
2.
Shirin, Haim, et al.. (1998). H pylori induces p21cip1 & inhibits G1/S progression while inducing apoptosis in synchronized gastric epithelial cells. Gastroenterology. 114. A287–A287. 1 indexed citations
3.
Arber, Nadir, Hanina Hibshoosh, Thomas R. Sutter, et al.. (1996). Increased expression of cyclin D1 is an early event in multistage colorectal carcinogenesis. Gastroenterology. 110(3). 669–674. 277 indexed citations
4.
Livneh, Etta, et al.. (1996). Linking protein kinase C to the cell cycle: ectopic expression of PKC eta in NIH3T3 cells alters the expression of cyclins and Cdk inhibitors and induces adipogenesis.. PubMed. 12(7). 1545–55. 64 indexed citations
5.
Fabbro, Doriano, et al.. (1993). The epsilon isoform of protein kinase C is an oncogene when overexpressed in rat fibroblasts.. PubMed. 8(8). 2095–104. 208 indexed citations
6.
Brandt‐Rauf, Paul W., et al.. (1992). A peptide from the GAP-binding domain of the ras-p21 protein and azatyrosine block ras-induced maturation of Xenopus oocytes.. PubMed. 11(4). 1373–8. 15 indexed citations
7.
Shinozuka, Hisashi, et al.. (1991). Changes in expression of cellular oncogenes and endogenous retrovirus-like sequences during hepatocarcinogenesis induced by a peroxisome proliferator. British Journal of Cancer. 64(5). 815–820. 29 indexed citations
8.
O’Driscoll, Kevin R., et al.. (1991). 1 alpha,25-dihydroxyvitamin D3-induced regulation of protein kinase C gene expression during HL-60 cell differentiation.. PubMed. 2(4). 187–94. 37 indexed citations
9.
Neugut, AI, et al.. (1990). Utilizing television to promote a community colon cancer screening program.. PubMed. 339. 311–23. 2 indexed citations
10.
LoGerfo, P, et al.. (1987). Altered levels of protein kinase C and Ca2+-dependent protein kinases in human colon carcinomas.. PubMed. 47(8). 2036–9. 111 indexed citations
11.
Ib, Weinstein. (1985). Cell culture studies on the mechanism of action of chemical carcinogens and tumor promoters.. PubMed. 10. 177–87. 4 indexed citations
12.
Gattoni‐Celli, Sebastiano, et al.. (1985). Genetic targets in multistage carcinogenesis.. PubMed. 9. 29–40. 2 indexed citations
13.
Ib, Weinstein, Sebastiano Gattoni‐Celli, Paul T. Kirschmeier, et al.. (1984). Initial cellular targets and eventual genomic changes in multistage carcinogenesis.. PubMed. 277–97. 4 indexed citations
14.
Pestka, S, et al.. (1984). The human interferons: from the past and into the future.. PubMed. 37. 261–81. 5 indexed citations
15.
Ib, Weinstein, et al.. (1981). Enhancement of macrophage-induced cytotoxicity by phorbol ester tumor promoters.. PubMed. 41(11 Pt 1). 4523–8. 25 indexed citations
16.
Ib, Weinstein. (1980). Estimation of distributions of time to appearance of tumor and time to death from tumor after appearance in mice fed 2-acetylaminofluorene.. PubMed. 3(3 Spec No). 89–101. 26 indexed citations
17.
Ib, Weinstein. (1980). Cell culture systems for studying multifactor interactions in carcinogenesis.. PubMed. 8. 149–64. 4 indexed citations
18.
Ib, Weinstein. (1978). Current concepts on mechanisms of chemical carcinogenesis.. PubMed. 54(4). 366–83. 26 indexed citations
19.
Pulkrabek, Peter, et al.. (1977). Differential excision from DNA of the C-8 and N2 guanosine adducts of N-acetyl-2-aminofluorene by single strand-specific endonucleases.. PubMed. 37(10). 3756–60. 91 indexed citations
20.
Ib, Weinstein, et al.. (1965). Miracil D: An Inhibitor of Ribonucleic Acid Synthesis in Bacillus subtilis. Molecular Pharmacology. 1(3). 297–305. 30 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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