Wei Xiang

3.2k total citations
90 papers, 2.4k citations indexed

About

Wei Xiang is a scholar working on Molecular Biology, Cancer Research and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Wei Xiang has authored 90 papers receiving a total of 2.4k indexed citations (citations by other indexed papers that have themselves been cited), including 44 papers in Molecular Biology, 15 papers in Cancer Research and 11 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Wei Xiang's work include Cancer-related molecular mechanisms research (8 papers), Epigenetics and DNA Methylation (7 papers) and Autophagy in Disease and Therapy (7 papers). Wei Xiang is often cited by papers focused on Cancer-related molecular mechanisms research (8 papers), Epigenetics and DNA Methylation (7 papers) and Autophagy in Disease and Therapy (7 papers). Wei Xiang collaborates with scholars based in China, United States and Australia. Wei Xiang's co-authors include Fenghua Tao, Hongbing Deng, Yanxiang Cheng, Xue‐Hai Zhu, Huifeng Zheng, Yumin Du, Xiaowen Shi, Songshan Huang, Hai Tao and Lijun Xu and has published in prestigious journals such as Circulation, Nature Communications and Hepatology.

In The Last Decade

Wei Xiang

87 papers receiving 2.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Wei Xiang China 28 1.1k 454 343 323 266 90 2.4k
Kun Zhao China 28 1.2k 1.0× 361 0.8× 190 0.6× 175 0.5× 205 0.8× 163 2.8k
Jingjing Ye China 30 1.2k 1.1× 338 0.7× 356 1.0× 662 2.0× 125 0.5× 128 2.9k
Kaijuan Wang China 24 862 0.8× 498 1.1× 174 0.5× 240 0.7× 93 0.3× 105 2.0k
Yong Sook Kim South Korea 28 1.2k 1.0× 374 0.8× 455 1.3× 560 1.7× 408 1.5× 74 2.9k
Yue Zhou China 27 1.2k 1.0× 300 0.7× 654 1.9× 684 2.1× 188 0.7× 77 3.1k
Yuan Yang China 32 1.6k 1.4× 456 1.0× 146 0.4× 196 0.6× 102 0.4× 267 3.8k
Jialiang Wang China 27 1.1k 1.0× 449 1.0× 274 0.8× 523 1.6× 98 0.4× 81 2.8k
Ziying Yang China 21 1.4k 1.2× 618 1.4× 278 0.8× 173 0.5× 273 1.0× 66 2.2k

Countries citing papers authored by Wei Xiang

Since Specialization
Citations

This map shows the geographic impact of Wei Xiang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wei Xiang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wei Xiang more than expected).

Fields of papers citing papers by Wei Xiang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Wei Xiang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wei Xiang. The network helps show where Wei Xiang may publish in the future.

Co-authorship network of co-authors of Wei Xiang

This figure shows the co-authorship network connecting the top 25 collaborators of Wei Xiang. A scholar is included among the top collaborators of Wei Xiang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Wei Xiang. Wei Xiang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Xiang, Wei, Jie Liu, Wei Li, et al.. (2025). Design, synthesis, and structure-activity relationship of 5,7- dimethylbenzo[d]thiazoles as novel Kv7.2/7.3 activators with antiepileptic effects. European Journal of Medicinal Chemistry. 292. 117660–117660. 1 indexed citations
2.
Ma, Yuheng, Xi Liu, Yuhuan Xie, et al.. (2025). Synthesis and characterization of celecoxib peroxide: crystal structure, theoretical analysis, thermochemistry and bond dissociation energy. Acta Crystallographica Section C Structural Chemistry. 81(8). 488–496.
3.
4.
Lyu, Lei, Rui Min, Fuxin Zheng, et al.. (2024). Prognostic value of inflammation and immune-related gene NOD2 in clear cell renal cell carcinoma. Human Cell. 37(3). 782–800. 1 indexed citations
5.
6.
Ni, Li, Satadru K. Lahiri, Jiali Nie, et al.. (2021). Genetic inhibition of nuclear factor of activated T-cell c2 prevents atrial fibrillation in CREM transgenic mice. Cardiovascular Research. 118(13). 2805–2818. 18 indexed citations
7.
Luo, Yue, Tao Hai, Jin Lin, Wei Xiang, & Weichun Guo. (2019). CDKN2B-AS1 Exerts Oncogenic Role in Osteosarcoma by Promoting Cell Proliferation and Epithelial to Mesenchymal Transition. Cancer Biotherapy and Radiopharmaceuticals. 35(1). 58–65. 12 indexed citations
8.
Xiang, Wei, Ting Jiang, Xiaoxia Hao, et al.. (2019). Primary cilia and autophagy interaction is involved in mechanical stress mediated cartilage development via ERK/mTOR axis. Life Sciences. 218. 308–313. 27 indexed citations
9.
10.
Wang, Yongsheng, Yinliang Qi, Wei Xiang, et al.. (2018). Hyperbaric oxygen rescues lung cancer cells from chemical hypoxia-induced low differentiation and apoptosis resistance. Experimental Lung Research. 44(8-9). 417–423. 9 indexed citations
11.
Xiang, Wei, et al.. (2017). Basic fibroblast growth factor increases IFT88 expression in chondrocytes. Molecular Medicine Reports. 16(5). 6590–6599. 5 indexed citations
12.
Sun, Yue, Yaping Ye, Peng Zhang, et al.. (2017). Iguratimod prevents ovariectomy-induced bone loss and suppresses osteoclastogenesis via inhibition of peroxisome proliferator-activated receptor-γ. Molecular Medicine Reports. 16(6). 8200–8208. 23 indexed citations
13.
Wu, Xinchao, Dong Liu, Dan Tao, et al.. (2016). BRD4 Regulates EZH2 Transcription through Upregulation of C-MYC and Represents a Novel Therapeutic Target in Bladder Cancer. Molecular Cancer Therapeutics. 15(5). 1029–1042. 87 indexed citations
14.
Cai, Yin, Miao Liu, Fengyun Sun, Chunlin Li, & Wei Xiang. (2016). [Influencing factors of non-point source pollution of watershed based on boosted regression tree algorithm.]. PubMed. 27(3). 911–919. 1 indexed citations
15.
Deng, Ke‐Qiong, Aibing Wang, Yan‐Xiao Ji, et al.. (2016). Suppressor of IKKɛ is an essential negative regulator of pathological cardiac hypertrophy. Nature Communications. 7(1). 11432–11432. 60 indexed citations
16.
Huang, Xin, Shilong Huang, Fei Xu, et al.. (2015). Dose-dependent inhibitory effects of zoledronic acid on osteoblast viability and function in vitro. Molecular Medicine Reports. 13(1). 613–622. 54 indexed citations
17.
Xu, Hui, et al.. (2014). Preparation and Characterization of Mesoporous Activated Carbon from Alligator Weed with H<sub>3</sub>PO<sub>4</sub>. Advanced materials research. 936. 868–874. 3 indexed citations
18.
Zhang, Yan, Xiaoxiong Liu, Zhi‐Gang She, et al.. (2014). Interferon regulatory factor 9 is an essential mediator of heart dysfunction and cell death following myocardial ischemia/reperfusion injury. Basic Research in Cardiology. 109(5). 434–434. 50 indexed citations
19.
Liu, Hongyun, Youbin Deng, Kun Liu, et al.. (2013). Left ventricular systolic strain of the cardiac allograft evaluated with three-dimensional speckle tracking echocardiography. Journal of Huazhong University of Science and Technology [Medical Sciences]. 33(5). 765–769. 2 indexed citations
20.
Xiang, Wei, Ye Cao, Xiaosheng Wang, et al.. (2012). Somal and Dendritic Development of Human CA3 Pyramidal Neurons F rom Midgestation to Middle Childhood: A Quantitative Golgi Study. The Anatomical Record. 296(1). 123–132. 24 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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