Wangsen Cao

4.2k total citations
70 papers, 3.4k citations indexed

About

Wangsen Cao is a scholar working on Molecular Biology, Nephrology and Genetics. According to data from OpenAlex, Wangsen Cao has authored 70 papers receiving a total of 3.4k indexed citations (citations by other indexed papers that have themselves been cited), including 52 papers in Molecular Biology, 12 papers in Nephrology and 11 papers in Genetics. Recurrent topics in Wangsen Cao's work include Genomics, phytochemicals, and oxidative stress (9 papers), Parathyroid Disorders and Treatments (9 papers) and Histone Deacetylase Inhibitors Research (9 papers). Wangsen Cao is often cited by papers focused on Genomics, phytochemicals, and oxidative stress (9 papers), Parathyroid Disorders and Treatments (9 papers) and Histone Deacetylase Inhibitors Research (9 papers). Wangsen Cao collaborates with scholars based in China, United States and Bangladesh. Wangsen Cao's co-authors include Shasha Yin, Charles J. Lowenstein, Clare Bao, Sylvain Doré, Wei Ai, Fang Chen, Wenjun Lin, Qing Jiang, Zhihong Liu and Qi Gao and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Wangsen Cao

70 papers receiving 3.4k citations

Author Peers

Peers are selected by citation overlap in the author's most active subfields. citations · hero ref

Author Last Decade Papers Cites
Wangsen Cao 2.1k 434 410 401 376 70 3.4k
Phuong Le 2.1k 1.0× 310 0.7× 458 1.1× 314 0.8× 197 0.5× 75 4.0k
Pamela M. Martin 2.7k 1.3× 329 0.8× 777 1.9× 415 1.0× 145 0.4× 95 4.7k
Jean‐Loup Bascands 1.9k 0.9× 536 1.2× 313 0.8× 184 0.5× 718 1.9× 155 4.7k
Kyu‐Sang Park 1.7k 0.8× 259 0.6× 721 1.8× 248 0.6× 476 1.3× 131 3.9k
Takashi Moriguchi 2.2k 1.1× 642 1.5× 332 0.8× 294 0.7× 138 0.4× 38 3.5k
Hisao Seo 2.3k 1.1× 439 1.0× 641 1.6× 619 1.5× 279 0.7× 194 5.2k
John S.D. Chan 2.0k 0.9× 473 1.1× 435 1.1× 300 0.7× 709 1.9× 132 4.6k
Matthias Baumann 1.2k 0.6× 470 1.1× 244 0.6× 167 0.4× 156 0.4× 123 3.4k
M.J. López-Armada 1.1k 0.5× 503 1.2× 412 1.0× 349 0.9× 415 1.1× 60 3.0k
Laurent Baud 1.3k 0.6× 775 1.8× 519 1.3× 218 0.5× 720 1.9× 115 3.8k

Countries citing papers authored by Wangsen Cao

Since Specialization
Citations

This map shows the geographic impact of Wangsen Cao's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wangsen Cao with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wangsen Cao more than expected).

Fields of papers citing papers by Wangsen Cao

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Wangsen Cao. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wangsen Cao. The network helps show where Wangsen Cao may publish in the future.

Co-authorship network of co-authors of Wangsen Cao

This figure shows the co-authorship network connecting the top 25 collaborators of Wangsen Cao. A scholar is included among the top collaborators of Wangsen Cao based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Wangsen Cao. Wangsen Cao is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Li, Li, Bin Zhang, Junaid Wazir, et al.. (2025). S1PR3-driven positive feedback loop sustains STAT3 activation and keratinocyte hyperproliferation in psoriasis. Cell Death and Disease. 16(1). 31–31. 4 indexed citations
3.
Wei, Lulu, Ranran Wang, Li Li, et al.. (2024). Disrupted methionine cycle triggers muscle atrophy in cancer cachexia through epigenetic regulation of REDD1. Cell Metabolism. 37(2). 460–476.e8. 16 indexed citations
4.
Meng, Xiangrui, et al.. (2024). Longikaurin A induces ferroptosis and inhibits glioblastoma progression through DNA methylation - Mediated GPX4 suppression. European Journal of Pharmacology. 984. 177061–177061. 3 indexed citations
5.
Zhang, Qiong, Yi Zhang, Tingyu Wang, et al.. (2023). Bromodomain-containing protein 4 activates androgen receptor transcription and promotes ovarian fibrosis in PCOS. Cell Reports. 42(9). 113090–113090. 18 indexed citations
6.
Xu, Zhou, Xinran Li, Yuyang Zhang, et al.. (2022). LRPPRC inhibits autophagy and promotes foam cell formation in atherosclerosis. FEBS Journal. 289(23). 7545–7560. 7 indexed citations
7.
Ma, Xiaojie, Xiaowu Dong, Yao−Zhong Xu, et al.. (2022). Identification of AP-1 as a Critical Regulator of Glutathione Peroxidase 4 (GPX4) Transcriptional Suppression and Acinar Cell Ferroptosis in Acute Pancreatitis. Antioxidants. 12(1). 100–100. 23 indexed citations
8.
Zhu, Xiaobo, Jian Dong, Fang Chen, et al.. (2022). Reversal of Epigenetic Peroxisome Proliferator-Activated Receptor-γ Suppression by Diacerein Alleviates Oxidative Stress and Osteoarthritis in Mice. Antioxidants and Redox Signaling. 37(1-3). 40–53. 12 indexed citations
9.
Niu, Mengyuan, Li Li, Zhonglan Su, et al.. (2021). An integrative transcriptome study reveals Ddit4/Redd1 as a key regulator of cancer cachexia in rodent models. Cell Death and Disease. 12(7). 652–652. 21 indexed citations
10.
Dong, Jian, Kaijia Zhang, Gaocai Li, et al.. (2021). CDDO-Im ameliorates osteoarthritis and inhibits chondrocyte apoptosis in mice via enhancing Nrf2-dependent autophagy. Acta Pharmacologica Sinica. 43(7). 1793–1802. 23 indexed citations
11.
Chen, Xingren, Xiaobo Zhu, Wei Ai, et al.. (2021). Nrf2 epigenetic derepression induced by running exercise protects against osteoporosis. Bone Research. 9(1). 15–15. 74 indexed citations
12.
Zhu, Xiaobo, Fang Chen, Ke Lu, et al.. (2019). PPARγ preservation via promoter demethylation alleviates osteoarthritis in mice. Annals of the Rheumatic Diseases. 78(10). 1420–1429. 93 indexed citations
13.
Yin, Shasha, Qin Zhang, Jun Yang, et al.. (2017). TGFβ-incurred epigenetic aberrations of miRNA and DNA methyltransferase suppress Klotho and potentiate renal fibrosis. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1864(7). 1207–1216. 76 indexed citations
14.
Lin, Wenjun, Qin Zhang, Lin Liu, et al.. (2017). Klotho restoration via acetylation of Peroxisome Proliferation–Activated Receptor γ reduces the progression of chronic kidney disease. Kidney International. 92(3). 669–679. 67 indexed citations
15.
Zhang, Qin, Lin Liu, Wenjun Lin, et al.. (2016). Rhein reverses Klotho repression via promoter demethylation and protects against kidney and bone injuries in mice with chronic kidney disease. Kidney International. 91(1). 144–156. 103 indexed citations
16.
Zhang, Qin, Shasha Yin, Lin Liu, Zhihong Liu, & Wangsen Cao. (2016). Rhein reversal of DNA hypermethylation-associated Klotho suppression ameliorates renal fibrosis in mice. Scientific Reports. 6(1). 34597–34597. 56 indexed citations
17.
Zhang, Bin, Sijing Xie, Zhonglan Su, et al.. (2016). Heme oxygenase-1 induction attenuates imiquimod-induced psoriasiform inflammation by negative regulation of Stat3 signaling. Scientific Reports. 6(1). 21132–21132. 42 indexed citations
18.
Cao, Wangsen, Clare Bao, Elizaveta Padalko, & Charles J. Lowenstein. (2008). Acetylation of mitogen-activated protein kinase phosphatase-1 inhibits Toll-like receptor signaling. The Journal of Experimental Medicine. 205(6). 1491–1503. 169 indexed citations
20.
Cao, Wangsen, David F. Claxton, Christine A. Kelley, et al.. (1997). CBFβ-SMMHC, expressed in M4Eo AML, reduced CBF DNA-binding and inhibited the G1 to S cell cycle transition at the restriction point in myeloid and lymphoid cells. Oncogene. 15(11). 1315–1327. 71 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026