Walter E. Laug

4.3k citations
73 papers · 3.6k indexed · h-index 33
    • Cell Adhesion Molecules Research 17
    • Protease and Inhibitor Mechanisms 30
  • Hematology top 2%
    • Blood Coagulation and Thrombosis Mechanisms 12
  • Genetics top 2%
    • Glioma Diagnosis and Treatment 5
  • Oncology top 5%
    • Peptidase Inhibition and Analysis 6
    • Angiogenesis and VEGF in Cancer 11
    • Signaling Pathways in Disease 6
    • S100 Proteins and Annexins 5

Walter E. Laug

73 papers receiving 3.4k citations

Peers

Walter E. Laug
Comparison fields: 5 of 119
  • Immunology and Allergy 803
  • Cancer Research 1.3k
  • Hematology 511
  • Genetics 359
  • Oncology 747
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Countries citing papers authored by Walter E. Laug

Since Specialization
Citations

This map shows the geographic impact of Walter E. Laug's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Walter E. Laug with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Walter E. Laug more than expected).

Fields of papers citing papers by Walter E. Laug

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Walter E. Laug. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Walter E. Laug. The network helps show where Walter E. Laug may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Walter E. Laug, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Walter E. Laug Line = papers co-authored together Walter E. Laug links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20132
2 2008123
3 200712
4 2006107
5 200520
6 200410
7 200426
8 200374
9 200333
10 2002163
11 200215
12 199799
13 199627
14 199677
15 199680
16 199212
17 19907
18 199025
19 19883
20
Morphology, growth, chromosomal pattern and fibrinolytic activity of two new human neuroblastoma cell lines.
1977206

About Walter E. Laug

Walter E. Laug is a scholar working on Immunology and Allergy, Cancer Research and Hematology, having authored 73 papers that have together received 3.6k indexed citations. Recurring topics across this work include Protease and Inhibitor Mechanisms (30 papers), Cell Adhesion Molecules Research (17 papers), Blood Coagulation and Thrombosis Mechanisms (12 papers), Angiogenesis and VEGF in Cancer (11 papers), Peptidase Inhibition and Analysis (6 papers), Signaling Pathways in Disease (6 papers), Glioma Diagnosis and Treatment (5 papers) and S100 Proteins and Annexins (5 papers). The work is most often cited by research in Immunology and Allergy (803 citations), Cancer Research (1.3k citations) and Hematology (511 citations). Walter E. Laug has collaborated with scholars based in United States, Spain and Ireland. Frequent co-authors include Yves A. DeClerck, Hiroyuki Shimada, William F. Benedict, Peter A. Jones, Vazgen Khankaldyyan, Robert C. Seeger, Barbara M. Mueller, Takashi Taga, Tobey J. MacDonald and Ignacio Gonzales-Gomez. Their work appears in journals such as Neurosurgery, Thrombosis and Haemostasis, JNCI Journal of the National Cancer Institute, Molecular Imaging and Journal of Surgical Research.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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