Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies
if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the
same subfield and year (this is the minimum needed to enter the top 1%, not the average
within it), or reaches the top citation threshold in at least one of its specific research
topics.
Cloning of a complementary DNA for a protein-tyrosine kinase that specifically phosphorylates a negative regulatory site of p60c-src
1991563 citationsShigeyuki Nada, A MacAuley et al.Natureprofile →
Peers — A (Enhanced Table)
Peers by citation overlap · career bar shows stage (early→late)
cites ·
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This map shows the geographic impact of A MacAuley's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by A MacAuley with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites A MacAuley more than expected).
This network shows the impact of papers produced by A MacAuley. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by A MacAuley. The network helps show where A MacAuley may publish in the future.
Co-authorship network of co-authors of A MacAuley
This figure shows the co-authorship network connecting the top 25 collaborators of A MacAuley.
A scholar is included among the top collaborators of A MacAuley based on the total number of
citations received by their joint publications. Widths of edges
represent the number of papers authors have co-authored together.
Node borders
signify the number of papers an author published with A MacAuley. A MacAuley is excluded from
the visualization to improve readability, since they are connected to all nodes in the network.
Werb, Zena, John Ashkenas, A MacAuley, & Jane F. Wiesen. (1996). Extracellular matrix remodeling as a regulator of stromal-epithelial interactions during mammary gland development, involution and carcinogenesis.. PubMed. 29(9). 1087–97.55 indexed citations
7.
Werb, Zena, Carolyn J. Sympson, Caroline M. Alexander, et al.. (1996). Extracellular matrix remodeling and the regulation of epithelial-stromal interactions during differentiation and involution.. PubMed. 54. S68–74.84 indexed citations
8.
MacAuley, A, Masato Okada, Shigeyuki Nada, H. Nakagawa, & Jonathan A. Cooper. (1993). Phosphorylation of Src mutants at Tyr 527 in fibroblasts does not correlate with in vitro phosphorylation by CSK.. PubMed. 8(1). 117–24.29 indexed citations
9.
Nada, Shigeyuki, et al.. (1991). Cloning of a complementary DNA for a protein-tyrosine kinase that specifically phosphorylates a negative regulatory site of p60c-src. Nature. 351(6321). 69–72.563 indexed citations breakdown →
10.
MacAuley, A, et al.. (1990). Acidic residues at the carboxyl terminus of p60c-src are required for regulation of tyrosine kinase activity and transformation.. PubMed. 2(9). 828–40.7 indexed citations
11.
Kashishian, Adam, et al.. (1990). Properties of tripartite chimeras between Src and Lck.. PubMed. 5(10). 1463–70.5 indexed citations
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive
bibliographic database. While OpenAlex provides broad and valuable coverage of the global
research landscape, it—like all bibliographic datasets—has inherent limitations. These include
incomplete records, variations in author disambiguation, differences in journal indexing, and
delays in data updates. As a result, some metrics and network relationships displayed in
Rankless may not fully capture the entirety of a scholar's output or impact.