W. Krone

2.1k total citations
72 papers, 1.7k citations indexed

About

W. Krone is a scholar working on Molecular Biology, Neurology and Cell Biology. According to data from OpenAlex, W. Krone has authored 72 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 39 papers in Molecular Biology, 34 papers in Neurology and 12 papers in Cell Biology. Recurrent topics in W. Krone's work include Neurofibromatosis and Schwannoma Cases (30 papers), Neuroblastoma Research and Treatments (12 papers) and Sarcoma Diagnosis and Treatment (10 papers). W. Krone is often cited by papers focused on Neurofibromatosis and Schwannoma Cases (30 papers), Neuroblastoma Research and Treatments (12 papers) and Sarcoma Diagnosis and Treatment (10 papers). W. Krone collaborates with scholars based in Germany, United States and Poland. W. Krone's co-authors include Michael Schmid, Walther Vogel, Günter Assum, K. Bross, Ingrid Eisenbarth, Dieter Kaufmann, Wolfgang Engel, Hildegard Kehrer‐Sawatzki, Sven Hoffmeyer and U. Wolf and has published in prestigious journals such as Nature, The Lancet and Diabetes.

In The Last Decade

W. Krone

69 papers receiving 1.6k citations

Author Peers

Peers are selected by citation overlap in the author's most active subfields. citations · hero ref

Author Last Decade Papers Cites
W. Krone 856 536 467 373 201 72 1.7k
Jay W. Ellison 1.2k 1.4× 351 0.7× 1.2k 2.7× 210 0.6× 249 1.2× 52 2.6k
Aslıhan Tolun 1.4k 1.6× 138 0.3× 936 2.0× 87 0.2× 228 1.1× 81 2.3k
Toshiyuki Habu 1.5k 1.8× 328 0.6× 341 0.7× 254 0.7× 113 0.6× 38 2.1k
Giorgio Bernardi 1.3k 1.5× 73 0.1× 767 1.6× 173 0.5× 75 0.4× 11 2.1k
Nicole Van Regemorter 1.2k 1.4× 135 0.3× 532 1.1× 54 0.1× 107 0.5× 57 2.0k
H. Kruyer 388 0.5× 270 0.5× 251 0.5× 40 0.1× 397 2.0× 20 930
Gurbax S. Sekhon 596 0.7× 187 0.3× 522 1.1× 124 0.3× 42 0.2× 42 1.1k
S Malcolm 1.2k 1.4× 96 0.2× 1.1k 2.4× 118 0.3× 44 0.2× 71 2.3k
Jennifer S. Fang 1.1k 1.3× 108 0.2× 414 0.9× 114 0.3× 176 0.9× 34 2.0k
Jenny Douglas 1.1k 1.3× 81 0.2× 681 1.5× 72 0.2× 93 0.5× 21 1.6k

Countries citing papers authored by W. Krone

Since Specialization
Citations

This map shows the geographic impact of W. Krone's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by W. Krone with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites W. Krone more than expected).

Fields of papers citing papers by W. Krone

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by W. Krone. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by W. Krone. The network helps show where W. Krone may publish in the future.

Co-authorship network of co-authors of W. Krone

This figure shows the co-authorship network connecting the top 25 collaborators of W. Krone. A scholar is included among the top collaborators of W. Krone based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with W. Krone. W. Krone is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Krone, W. & U. Wolf. (2009). Chromosome variation and gene action. Hereditas. 86(1). 31–35.
2.
Laudes, Matthias, F. Oberhäuser, K.-J. Walgenbach, et al.. (2009). Comparison of Phenotypes in Male and Female Individuals of a New Family with Dunnigan Type of Familial Partial Lipodystrophy Due to a Lamin A/CR482WMutation. Hormone and Metabolic Research. 41(5). 414–417. 6 indexed citations
3.
Schumann, C, et al.. (2005). Autoimmune Polyglandular Syndrome Associated with Idiopathic Giant Cell Myocarditis. Experimental and Clinical Endocrinology & Diabetes. 113(5). 302–307. 17 indexed citations
4.
Eisenbarth, Ingrid, G Vogel, W. Krone, Walther Vogel, & Günter Assum. (2000). An Isochore Transition in the NF1 Gene Region Coincides with a Switch in the Extent of Linkage Disequilibrium. The American Journal of Human Genetics. 67(4). 873–880. 49 indexed citations
5.
Eisenbarth, Ingrid, Kim S. Beyer, W. Krone, & Günter Assum. (2000). Toward a Survey of Somatic Mutation of the NF1 Gene in Benign Neurofibromas of Patients with Neurofibromatosis Type 1. The American Journal of Human Genetics. 66(2). 393–401. 61 indexed citations
6.
Häeussler, Jüergen, et al.. (2000). Tumor Antigen HuR Binds Specifically to One of Five Protein-Binding Segments in the 3′-Untranslated Region of the Neurofibromin Messenger RNA. Biochemical and Biophysical Research Communications. 267(3). 726–732. 20 indexed citations
8.
Hoffmeyer, Sven, et al.. (1998). Nearby Stop Codons in Exons of the Neurofibromatosis Type 1 Gene Are Disparate Splice Effectors. The American Journal of Human Genetics. 62(2). 269–277. 45 indexed citations
9.
Kehrer‐Sawatzki, Hildegard, et al.. (1997). A third neurofibromatosis type 1 (NF1) pseudogene at chromosome 15q11.2. Human Genetics. 100(5-6). 595–600. 21 indexed citations
10.
Hoffmeyer, Sven, et al.. (1995). On unequal allelic expression of the neurofibromin gene in neurofibromatosis type 1. Human Molecular Genetics. 4(8). 1267–1272. 40 indexed citations
11.
Eisenbarth, Ingrid, et al.. (1995). Analysis of an alternatively spliced exon of the neurofibromatosis type 1 gene in cultured melanocytes from patients with neurofibromatosis 1. Archives of Dermatological Research. 287(5). 413–416. 3 indexed citations
12.
Hoffmeyer, Sven, et al.. (1994). A deletion in the 5 ′-region of the neurofibromatosis type 1 (NF1) gene. Human Genetics. 94(1). 97–100. 11 indexed citations
13.
Motejlek, K, Günter Assum, W. Krone, & Albrecht K. Kleinschmidt. (1991). The size of small polydisperse circular DNA (spcDNA) in angiofibroma-derived cell cultures from patients with tuberous sclerosis (TSC) differs from that in fibroblasts. Human Genetics. 87(1). 6–10. 10 indexed citations
14.
Kaufmann, Dieter, et al.. (1991). Increased melanogenesis in cultured epidermal melanocytes from patients with neurofibromatosis 1 (NF1). Human Genetics. 87(2). 144–150. 36 indexed citations
15.
Stark, Markus, et al.. (1991). A small deletion and an adjacent base exchange in a potential stem-loop region of the neurofibromatosis 1 gene. Human Genetics. 87(6). 685–7. 20 indexed citations
16.
Dietrich, Claudia U., et al.. (1990). Cytogenetic studies in tuberous sclerosis. Cancer Genetics and Cytogenetics. 45(2). 161–177. 13 indexed citations
17.
Kaufmann, Dieter, et al.. (1989). A cell culture study on melanocytes from patients with neurofibromatosis-1. Archives of Dermatological Research. 281(7). 510–513. 17 indexed citations
18.
Schmid, Michael & W. Krone. (1976). The relationship of a specific chromosomal region to the development of the acrosome. Chromosoma. 56(4). 327–347. 33 indexed citations
19.
Hoehn, Holger, et al.. (1971). In vitro alteration of association patterns of human acrocentric chromosomes. Human Genetics. 11(2). 146–154. 8 indexed citations
20.
Engel, Wolfgang, W. Krone, & U. Wolf. (1967). Die wirkung von thioguanin, hydroxylamin unde 5-bromdesoxyuridin auf menschliche chromosomen in vitro. Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis. 4(3). 353–368. 30 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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