W E Hathaway

683 citations
14 papers · 497 indexed · h-index 10

Impact in

  • Hematology top 5%
    • Blood Coagulation and Thrombosis Mechanisms
    • Platelet Disorders and Treatments
    • Hemophilia Treatment and Research
    • Venous Thromboembolism Diagnosis and Management

Papers in

    • Immune Response and Inflammation 3
    • Immunodeficiency and Autoimmune Disorders 2
    • Neutrophil, Myeloperoxidase and Oxidative Mechanisms 2

W E Hathaway

14 papers receiving 449 citations

Peers

W E Hathaway
Comparison fields: 5 of 65
  • Hematology 266
  • Internal Medicine 61
  • Genetics 72
  • Immunology and Allergy 37
  • Immunology 109
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Citations per field
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Citations per year

Countries citing papers authored by W E Hathaway

Since Specialization
Citations

This map shows the geographic impact of W E Hathaway's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by W E Hathaway with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites W E Hathaway more than expected).

Fields of papers citing papers by W E Hathaway

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by W E Hathaway. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by W E Hathaway. The network helps show where W E Hathaway may publish in the future.

Co-authors

The 25 scholars most cited alongside W E Hathaway, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with W E Hathaway Line = papers co-authored together W E Hathaway links everyone, so they are left out of the graph.

All Works

14 of 14 papers shown
#Work
1
Thrombosis in children receiving L-asparaginase. Determining patients at risk.
199332
2 199121
3 198624
4 198410
5 198412
6 19773
7 197621
8 1975316
9 19759
10 19741
11 197327
12 19689
13 19679
14 19653

About W E Hathaway

W E Hathaway is a scholar working on General Social Sciences, Immunology, Emergency Medical Services, Hematology and Nephrology, having authored 14 papers that have together received 497 indexed citations. Recurring topics across this work include Immune Response and Inflammation (3 papers), Blood Coagulation and Thrombosis Mechanisms (3 papers), Immunodeficiency and Autoimmune Disorders (2 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (2 papers), Neonatal Respiratory Health Research (2 papers), Pregnancy and preeclampsia studies (1 paper), Acute Myeloid Leukemia Research (1 paper) and Genetic Syndromes and Imprinting (1 paper). The work is most often cited by research in Hematology (266 citations), Internal Medicine (61 citations), Genetics (72 citations), Immunology and Allergy (37 citations) and Immunology (109 citations). W E Hathaway has collaborated with scholars based in United States and Thailand. Frequent co-authors include R P Rivers, William L. Weston, Lorrie F. Odom, Amy D. Shapiro, David S. Pearlman, C. Henry Kempe, Vincent A. Fulginiti, E. B. Reeve, Marilyn J. Manco‐Johnson and John H. Githens. Their work appears in journals such as Pediatric Research, The Journal of Pediatrics, British Journal of Haematology, American Journal of Hematology and Journal of Clinical Investigation.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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