W. Andrew Yeudall
- Otorhinolaryngology top 1%
- Head and Neck Cancer Studies 8
- Periodontics top 1%
- Oncology top 2%
- Cancer-related Molecular Pathways 33
- Chemokine receptors and signaling 7
- Cancer Research top 5%
- Molecular Biology top 5%
- TGF-β signaling in diseases 10
- RNA Interference and Gene Delivery 9
- Cancer-related gene regulation 8
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- Cancer Research and Treatments 8
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- Dendrimers and Hyperbranched Polymers 6
W. Andrew Yeudall
89 papers receiving 3.0k citations
Peers
Comparison fields: 5 of 109
- Otorhinolaryngology 287
- Periodontics 290
- Oncology 1.2k
- Cancer Research 434
- Molecular Biology 1.5k
Countries citing papers authored by W. Andrew Yeudall
This map shows the geographic impact of W. Andrew Yeudall's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by W. Andrew Yeudall with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites W. Andrew Yeudall more than expected).
Fields of papers citing papers by W. Andrew Yeudall
This network shows the impact of papers produced by W. Andrew Yeudall. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by W. Andrew Yeudall. The network helps show where W. Andrew Yeudall may publish in the future.
Co-authorship network
The 25 scholars most cited alongside W. Andrew Yeudall, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2024 | 3 | |
| 2 | 2022 | 5 | |
| 3 | 2020 | 3 | |
| 4 | 2018 | 5 | |
| 5 | 2018 | 15 | |
| 6 | 2017 | 51 | |
| 7 | 2017 | 36 | |
| 8 | 2012 | 20 | |
| 9 | 2011 | 13 | |
| 10 | 2008 | 72 | |
| 11 | 2008 | 43 | |
| 12 | 2005 | 24 | |
| 13 | 2004 | 57 | |
| 14 | Cell cycle control during osteoblast differentiation and post-confluence growth regulation by c-fos | 2001 | 1 |
| 15 | 1997 | 18 | |
| 16 | Sensitivity to transforming growth factor beta 1-induced growth arrest is common in human squamous cell carcinoma cell lines: c-MYC down-regulation and p21waf1 induction are important early events. | 1996 | 53 |
| 17 | 1993 | 22 | |
| 18 | 1993 | 44 | |
| 19 | 1992 | 46 | |
| 20 | 1991 | 81 |
About W. Andrew Yeudall
W. Andrew Yeudall is a scholar working on Otorhinolaryngology, Oncology and Periodontics, having authored 90 papers that have together received 3.0k indexed citations. Recurring topics across this work include Cancer-related Molecular Pathways (33 papers), TGF-β signaling in diseases (10 papers), RNA Interference and Gene Delivery (9 papers), Cancer-related gene regulation (8 papers), Cancer Research and Treatments (8 papers), Head and Neck Cancer Studies (8 papers), Chemokine receptors and signaling (7 papers) and Dendrimers and Hyperbranched Polymers (6 papers). The work is most often cited by research in Otorhinolaryngology (287 citations), Periodontics (290 citations) and Oncology (1.2k citations). W. Andrew Yeudall has collaborated with scholars based in United States, United Kingdom and France. Frequent co-authors include Hu Yang, Vyomesh Patel, Judit Jakus, John F. Ensley, J. Silvio Gutkind, Sinem E. Sahingur, Quan Yuan, Leyuan Xu, Keith C. Robbins and Hui‐Xin Wang. Their work appears in journals such as Journal of Biological Chemistry, Nature Communications and Cancer Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.