Vivi Kasim

2.6k total citations · 1 hit paper
62 papers, 1.9k citations indexed

About

Vivi Kasim is a scholar working on Molecular Biology, Cancer Research and Oncology. According to data from OpenAlex, Vivi Kasim has authored 62 papers receiving a total of 1.9k indexed citations (citations by other indexed papers that have themselves been cited), including 53 papers in Molecular Biology, 26 papers in Cancer Research and 19 papers in Oncology. Recurrent topics in Vivi Kasim's work include RNA modifications and cancer (12 papers), Cancer-related Molecular Pathways (11 papers) and Cancer, Hypoxia, and Metabolism (11 papers). Vivi Kasim is often cited by papers focused on RNA modifications and cancer (12 papers), Cancer-related Molecular Pathways (11 papers) and Cancer, Hypoxia, and Metabolism (11 papers). Vivi Kasim collaborates with scholars based in China, Japan and Thailand. Vivi Kasim's co-authors include Shourong Wu, Makoto Miyagishi, Can Huang, Rendy Hosea, Hezhao Zhao, Yanjun Li, Mami Yasukawa, Kenkichi Masutomi, Yoshiko Maida and Naoko Okamoto and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Nucleic Acids Research.

In The Last Decade

Vivi Kasim

57 papers receiving 1.8k citations

Hit Papers

The two sides of chromosomal instability: drivers and bra... 2024 2026 2025 2024 20 40 60

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Vivi Kasim China 24 1.3k 528 253 253 205 62 1.9k
Wen-Jia Chen China 23 996 0.8× 523 1.0× 345 1.4× 128 0.5× 192 0.9× 101 1.6k
Efrosyni Paraskeva Greece 24 1.4k 1.1× 757 1.4× 178 0.7× 261 1.0× 197 1.0× 43 2.1k
Peizhen Hu China 22 817 0.6× 461 0.9× 297 1.2× 168 0.7× 316 1.5× 55 1.8k
Kasper Thorsen Denmark 28 1.8k 1.4× 826 1.6× 312 1.2× 199 0.8× 257 1.3× 43 2.7k
Yongde Luo United States 29 1.9k 1.5× 403 0.8× 203 0.8× 193 0.8× 197 1.0× 62 2.5k
Kamiya Mehla United States 19 939 0.7× 623 1.2× 377 1.5× 246 1.0× 165 0.8× 27 1.6k
Katarzyna Augoff Poland 19 1.0k 0.8× 723 1.4× 299 1.2× 91 0.4× 164 0.8× 35 1.7k
Hong Shu China 22 780 0.6× 309 0.6× 245 1.0× 96 0.4× 177 0.9× 94 1.4k
Alessandro Carrer United States 15 1.4k 1.1× 751 1.4× 387 1.5× 324 1.3× 99 0.5× 22 2.1k

Countries citing papers authored by Vivi Kasim

Since Specialization
Citations

This map shows the geographic impact of Vivi Kasim's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Vivi Kasim with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Vivi Kasim more than expected).

Fields of papers citing papers by Vivi Kasim

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Vivi Kasim. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Vivi Kasim. The network helps show where Vivi Kasim may publish in the future.

Co-authorship network of co-authors of Vivi Kasim

This figure shows the co-authorship network connecting the top 25 collaborators of Vivi Kasim. A scholar is included among the top collaborators of Vivi Kasim based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Vivi Kasim. Vivi Kasim is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Hosea, Rendy, et al.. (2024). The two sides of chromosomal instability: drivers and brakes in cancer. Signal Transduction and Targeted Therapy. 9(1). 75–75. 72 indexed citations breakdown →
2.
Li, Wenfang, Can Huang, Lei Zhang, et al.. (2024). p52-ZER6/IGF1R axis maintains cancer stem cell population to promote cancer progression by enhancing pro-survival mitophagy. Oncogene. 43(27). 2115–2131. 8 indexed citations
3.
Hosea, Rendy, et al.. (2023). NeuroD1-GPX4 signaling leads to ferroptosis resistance in hepatocellular carcinoma. PLoS Genetics. 19(12). e1011098–e1011098. 9 indexed citations
4.
Tang, Yu, Qiu Li, Lei Zhang, et al.. (2023). The p52-ZER6/G6PD axis alters aerobic glycolysis and promotes tumor progression by activating the pentose phosphate pathway. Oncogenesis. 12(1). 17–17. 13 indexed citations
5.
Hosea, Rendy, et al.. (2023). Targeting Transcription Factor YY1 for Cancer Treatment: Current Strategies and Future Directions. Cancers. 15(13). 3506–3506. 34 indexed citations
6.
Li, Juan, Xinxin Luo, Zhuolin Li, et al.. (2023). YY2/PHGDH axis suppresses tumorigenesis by inhibiting tumor cell de novo serine biosynthesis. Biomedicine & Pharmacotherapy. 165. 115006–115006. 6 indexed citations
7.
Wang, Yi‐Cheng, et al.. (2023). Salidroside facilitates therapeutic angiogenesis in diabetic hindlimb ischemia by inhibiting ferroptosis. Biomedicine & Pharmacotherapy. 159. 114245–114245. 17 indexed citations
8.
Wu, Shourong, et al.. (2022). Intramuscular injection of sotagliflozin promotes neovascularization in diabetic mice through enhancing skeletal muscle cells paracrine function. Acta Pharmacologica Sinica. 43(10). 2636–2650. 13 indexed citations
9.
Kasim, Vivi, et al.. (2022). Therapeutic angiogenesis-based strategy for peripheral artery disease. Theranostics. 12(11). 5015–5033. 40 indexed citations
10.
Wang, Yi‐Cheng, et al.. (2022). SGLT2 inhibitor empagliflozin promotes revascularization in diabetic mouse hindlimb ischemia by inhibiting ferroptosis. Acta Pharmacologica Sinica. 44(6). 1161–1174. 27 indexed citations
11.
Hosea, Rendy, et al.. (2020). The biological implications of Yin Yang 1 in the hallmarks of cancer. Theranostics. 10(9). 4183–4200. 92 indexed citations
13.
Lei, Ke, Wenfang Li, Can Huang, et al.. (2019). Neurogenic differentiation factor 1 promotes colorectal cancer cell proliferation and tumorigenesis by suppressing the p53/p21 axis. Cancer Science. 111(1). 175–185. 22 indexed citations
14.
Huang, Can, Shourong Wu, Wenfang Li, et al.. (2019). Zinc-finger protein p52-ZER6 accelerates colorectal cancer cell proliferation and tumour progression through promoting p53 ubiquitination. EBioMedicine. 48. 248–263. 25 indexed citations
15.
Zhang, Jianqi, et al.. (2019). Tyrosol Facilitates Neovascularization by Enhancing Skeletal Muscle Cells Viability and Paracrine Function in Diabetic Hindlimb Ischemia Mice. Frontiers in Pharmacology. 10. 909–909. 19 indexed citations
16.
Wu, Shourong, Huimin Wang, Yanjun Li, et al.. (2018). Transcription Factor YY1 Promotes Cell Proliferation by Directly Activating the Pentose Phosphate Pathway. Cancer Research. 78(16). 4549–4562. 115 indexed citations
17.
Ji, Hong, Can Huang, Shourong Wu, & Vivi Kasim. (2018). XBP1-s promotes colorectal cancer cell proliferation by inhibiting TAp73 transcriptional activity. Biochemical and Biophysical Research Communications. 508(1). 203–209. 19 indexed citations
18.
Huang, Can, Shourong Wu, Hong Ji, et al.. (2017). Identification of XBP1-u as a novel regulator of the MDM2/p53 axis using an shRNA library. Science Advances. 3(10). e1701383–e1701383. 44 indexed citations
19.
20.
Wu, Shourong, Vivi Kasim, Mitsunobu R. Kano, et al.. (2013). Transcription Factor YY1 Contributes to Tumor Growth by Stabilizing Hypoxia Factor HIF-1α in a p53-Independent Manner. Cancer Research. 73(6). 1787–1799. 67 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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