Vikas Sud

999 citations
10 papers · 765 indexed · 1 hit paper · h-index 7

Vikas Sud

9 papers receiving 755 citations

Hit Papers

Neutrophil extracellular traps promote inflammation and d...3762018202620202023100200300

Peers

Vikas Sud
Comparison fields: 5 of 83
  • Hepatology 135
  • Immunology 323
  • Epidemiology 319
  • Physiology 136
  • Immunology and Allergy 31
Replace Anissa A. Widjaja with:
Anissa A. Widjaja Singapore
Antje Mohs Germany
Hui‐Wei Chen United States
Masaki Honda Japan
Aysha Ali United Kingdom
Sacha Robert France
Einav Vax Israel
Shiva Kumar Shanmukhappa United States
Rie Irie Japan
Ryan S. McMahan United States
Vikas Sud relative to Anissa A. Widjaja Singapore Anissa A. Widjaja's profile →
Citations per field
00.5×20×40×52×
Anissa A. Widjaja · 1×
Citations per year

Countries citing papers authored by Vikas Sud

Since Specialization
Citations

This map shows the geographic impact of Vikas Sud's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Vikas Sud with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Vikas Sud more than expected).

Fields of papers citing papers by Vikas Sud

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Vikas Sud. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Vikas Sud. The network helps show where Vikas Sud may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Vikas Sud, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Vikas Sud Line = papers co-authored together Vikas Sud links everyone, so they are left out of the graph.

All Works

10 of 10 papers shown
#Work
1 20250
2 201816
3
Neutrophil extracellular traps promote inflammation and development of hepatocellular carcinoma in nonalcoholic steatohepatitisbreakdown →
2018376
4 201836
5 201795
6 2017210
7 201712
8 20142
9 20141
10 201217

About Vikas Sud

Vikas Sud is a scholar working on Hepatology, Virology, Immunology, Urology and Ophthalmology, having authored 10 papers that have together received 765 indexed citations. Recurring topics across this work include Immune Response and Inflammation (2 papers), Immune cells in cancer (2 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (2 papers), IL-33, ST2, and ILC Pathways (2 papers), Liver Disease and Transplantation (1 paper), Renal cell carcinoma treatment (1 paper), Venomous Animal Envenomation and Studies (1 paper) and Eosinophilic Esophagitis (1 paper). The work is most often cited by research in Hepatology (135 citations), Immunology (323 citations), Epidemiology (319 citations), Physiology (136 citations) and Immunology and Allergy (31 citations). Vikas Sud has collaborated with scholars based in United States, India and China. Frequent co-authors include Allan Tsung, Hai Huang, Dirk J. van der Windt, Hongji Zhang, Samer Tohme, Patricia Loughran, Hamza O. Yazdani, Richard L. Simmons, Julie Goswami and Patrick R. Varley. Their work appears in journals such as Gene Expression, The Journal of Immunology, Journal of Hepatology, Oncotarget and European Journal of Surgical Oncology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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