Doron Melamed

3.7k total citations
61 papers, 2.6k citations indexed

About

Doron Melamed is a scholar working on Immunology, Molecular Biology and Oncology. According to data from OpenAlex, Doron Melamed has authored 61 papers receiving a total of 2.6k indexed citations (citations by other indexed papers that have themselves been cited), including 44 papers in Immunology, 11 papers in Molecular Biology and 8 papers in Oncology. Recurrent topics in Doron Melamed's work include T-cell and B-cell Immunology (38 papers), Immune Cell Function and Interaction (36 papers) and Immunotherapy and Immune Responses (17 papers). Doron Melamed is often cited by papers focused on T-cell and B-cell Immunology (38 papers), Immune Cell Function and Interaction (36 papers) and Immunotherapy and Immune Responses (17 papers). Doron Melamed collaborates with scholars based in Israel, United States and Germany. Doron Melamed's co-authors include Aharon Friedman, David Nemazee, John C. Cambier, Robert J. Benschop, Keren Zohar, E.D. Heller, Efrat Edry, Elias Toubi, Ayelet Snir and Aharon Kessel and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Doron Melamed

61 papers receiving 2.5k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Doron Melamed Israel 31 1.7k 453 251 205 186 61 2.6k
Craig Hammerberg United States 35 1.7k 1.0× 750 1.7× 293 1.2× 302 1.5× 283 1.5× 79 4.1k
Carmen Sánchez‐Torres Mexico 28 1.7k 1.0× 614 1.4× 333 1.3× 93 0.5× 227 1.2× 63 2.7k
Dominique Gatto Australia 17 2.0k 1.2× 681 1.5× 243 1.0× 178 0.9× 165 0.9× 20 2.8k
Jean-Claude Guillemot France 14 843 0.5× 513 1.1× 238 0.9× 120 0.6× 87 0.5× 19 1.8k
Paul E. Baker United States 23 1.5k 0.9× 593 1.3× 336 1.3× 346 1.7× 192 1.0× 41 2.5k
Lynn Puddington United States 33 2.1k 1.2× 774 1.7× 310 1.2× 110 0.5× 238 1.3× 53 3.2k
A. J. Snijders Netherlands 20 1.8k 1.1× 381 0.8× 280 1.1× 202 1.0× 101 0.5× 40 2.5k
Masayoshi Kohase Japan 23 1.3k 0.8× 637 1.4× 461 1.8× 165 0.8× 210 1.1× 51 2.4k
Franz Petry Germany 28 1.7k 1.1× 460 1.0× 127 0.5× 239 1.2× 81 0.4× 60 3.0k
Didier Leturcq United States 18 1.6k 1.0× 588 1.3× 178 0.7× 163 0.8× 118 0.6× 20 2.8k

Countries citing papers authored by Doron Melamed

Since Specialization
Citations

This map shows the geographic impact of Doron Melamed's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Doron Melamed with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Doron Melamed more than expected).

Fields of papers citing papers by Doron Melamed

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Doron Melamed. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Doron Melamed. The network helps show where Doron Melamed may publish in the future.

Co-authorship network of co-authors of Doron Melamed

This figure shows the co-authorship network connecting the top 25 collaborators of Doron Melamed. A scholar is included among the top collaborators of Doron Melamed based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Doron Melamed. Doron Melamed is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Labi, Verena, et al.. (2019). miR-17∼92 in lymphocyte development and lymphomagenesis. Cancer Letters. 446. 73–80. 11 indexed citations
2.
Labi, Verena, Rostislav Novak, Renaud Gaujoux, et al.. (2016). A c-Myc/miR17-92/Pten Axis Controls PI3K-Mediated Positive and Negative Selection in B Cell Development and Reconstitutes CD19 Deficiency. Cell Reports. 16(2). 419–431. 40 indexed citations
3.
Kessel, Aharon, Tharwat Haj, Regina Peri, et al.. (2011). Human CD19+CD25high B regulatory cells suppress proliferation of CD4+ T cells and enhance Foxp3 and CTLA-4 expression in T-regulatory cells. Autoimmunity Reviews. 11(9). 670–677. 247 indexed citations
4.
Meyron‐Holtz, Esther G., et al.. (2011). Alginate encapsulated cells secreting Fas‐ligand reduce lymphoma carcinogenicity. Cancer Science. 103(1). 116–124. 8 indexed citations
5.
Waisman, Ari, Manfred Kraus, Jane Seagal, et al.. (2007). IgG1 B cell receptor signaling is inhibited by CD22 and promotes the development of B cells whose survival is less dependent on Igα/β. The Journal of Experimental Medicine. 204(4). 747–758. 105 indexed citations
6.
Melamed, Doron. (2006). Modulation of matrix metalloproteinase-9 (MMP-9) secretion in B lymphopoiesis. International Immunology. 18(9). 1355–1362. 14 indexed citations
7.
8.
Melamed, Doron, et al.. (2004). Class switch recombination in B lymphopoiesis: a potential pathway for B cell autoimmunity. Autoimmunity Reviews. 3(6). 464–469. 8 indexed citations
9.
Aït‐Azzouzene, Djemel, Patrick Skog, Marc W. Retter, et al.. (2004). Tolerance‐induced receptor selection: scope, sensitivity, locus specificity, and relationship to lymphocyte‐positive selection. Immunological Reviews. 197(1). 219–230. 15 indexed citations
10.
Zohar, Keren, et al.. (2004). Modification of Ligand-independent B Cell Receptor Tonic Signals Activates Receptor Editing in Immature B Lymphocytes. Journal of Biological Chemistry. 279(14). 13418–13424. 32 indexed citations
11.
Melamed, Doron, et al.. (2003). Differential c-Myc Responsiveness to B Cell Receptor Ligation in B Cell-Negative Selection. The Journal of Immunology. 171(5). 2446–2452. 14 indexed citations
12.
Shivtiel, Shoham, et al.. (2002). Impaired Light Chain Allelic Exclusion and Lack of Positive Selection in Immature B Cells Expressing Incompetent Receptor Deficient of CD19. The Journal of Immunology. 168(11). 5596–5604. 53 indexed citations
13.
Shivtiel, Shoham, et al.. (2002). Receptor editing in CD45-deficient immature B cells. European Journal of Immunology. 32(8). 2264–2264. 10 indexed citations
14.
Seagal, Jane & Doron Melamed. (2002). Role of Receptor Revision in Forming a B Cell Repertoire. Clinical Immunology. 105(1). 1–8. 11 indexed citations
15.
Melamed, Doron, et al.. (2000). Unexpected Autoantibody Production in Membrane Ig-μ-Deficient/ lpr Mice. The Journal of Immunology. 165(8). 4353–4358. 39 indexed citations
16.
Nemazee, David, Valérie Kouskoff, W. Hertz, et al.. (2000). B-Cell-Receptor-Dependent Positive and Negative Selection in Immature B Cells. Current topics in microbiology and immunology. 245(2). 57–71. 35 indexed citations
17.
18.
Halevy, Orna, et al.. (1994). Retinoic Acid Receptor-α Gene Expression Is Modulated by Dietary Vitamin A and by Retinoic Acid in Chicken T Lymphocytes. Journal of Nutrition. 124(11). 2139–2146. 25 indexed citations
19.
Melamed, Doron & Aharon Friedman. (1993). Direct evidence for anergy in T lymphocytes tolerized by oral administration of ovalbumin. European Journal of Immunology. 23(4). 935–942. 203 indexed citations
20.
Melamed, Doron & Aharon Friedman. (1993). Modification of the Immune Response by Oral Tolerance: Antigen Requirements and Interaction with Immunogenic Stimuli. Cellular Immunology. 146(2). 412–420. 30 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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