Valentina Salvi
- Immunology top 5%
- Immunotherapy and Immune Responses 13
- Immune Response and Inflammation 10
- interferon and immune responses 5
- Immune Cell Function and Interaction 5
- T-cell and B-cell Immunology 5
- Cancer Research top 10%
- MicroRNA in disease regulation 5
- Oncology top 10%
- Chemokine receptors and signaling 4
- Rheumatology top 5%
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- Angiogenesis and VEGF in Cancer 4
- Co-authors
- Silvano SozzaniDaniela BosisioAnnalisa Del PreteFrancesca SozioMattia LaffranchiAlessandra SorianiLaura TiberioMarco Presta
- Cited by
- ImmunologyCancer ResearchOncology
- Partner nations
- ItalyUnited StatesSwitzerland
In The Last Decade
Valentina Salvi
49 papers receiving 1.9k citations
Hit Papers
Peers
Comparison fields: 5 of 123
- Immunology 834
- Cancer Research 254
- Oncology 436
- Complementary and Manual Therapy 28
- Rheumatology 190
Countries citing papers authored by Valentina Salvi
This map shows the geographic impact of Valentina Salvi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Valentina Salvi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Valentina Salvi more than expected).
Fields of papers citing papers by Valentina Salvi
This network shows the impact of papers produced by Valentina Salvi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Valentina Salvi. The network helps show where Valentina Salvi may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Valentina Salvi, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2024 | 8 | |
| 2 | 2024 | 5 | |
| 3 | 2024 | 0 | |
| 4 | 2024 | 11 | |
| 5 | 2024 | 5 | |
| 6 | 2023 | 7 | |
| 7 | 2023 | 7 | |
| 8 | Dendritic cell subsets in cancer immunity and tumor antigen sensingbreakdown → | 2023 | 354 |
| 9 | 2023 | 4 | |
| 10 | 2022 | 4 | |
| 11 | 2022 | 4 | |
| 12 | 2021 | 108 | |
| 13 | 2020 | 49 | |
| 14 | 2020 | 4 | |
| 15 | 2019 | 30 | |
| 16 | 2018 | 22 | |
| 17 | 2017 | 69 | |
| 18 | 2017 | 39 | |
| 19 | 2017 | 70 | |
| 20 | 2013 | 46 |
About Valentina Salvi
Valentina Salvi is a scholar working on Immunology, Cancer Research and Immunology and Allergy, having authored 50 papers that have together received 1.9k indexed citations. Recurring topics across this work include Immunotherapy and Immune Responses (13 papers), Immune Response and Inflammation (10 papers), interferon and immune responses (5 papers), MicroRNA in disease regulation (5 papers), Immune Cell Function and Interaction (5 papers), T-cell and B-cell Immunology (5 papers), Angiogenesis and VEGF in Cancer (4 papers) and Chemokine receptors and signaling (4 papers). The work is most often cited by research in Immunology (834 citations), Cancer Research (254 citations) and Oncology (436 citations). Valentina Salvi has collaborated with scholars based in Italy, United States and Switzerland. Frequent co-authors include Silvano Sozzani, Daniela Bosisio, Annalisa Del Prete, Francesca Sozio, Mattia Laffranchi, Alessandra Soriani, Laura Tiberio, Marco Presta, Stefania Mitola and Daria Leali. Their work appears in journals such as Blood, The Journal of Immunology and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.