Urška Repnik
- Physiology top 1%
- Calcium signaling and nucleotide metabolism 10
- Immunology top 5%
- Immunotherapy and Immune Responses 7
- Immune cells in cancer 6
- Endocrinology top 2%
- Cell Biology top 5%
- Cellular transport and secretion 11
- Infectious Diseases top 5%
- Tuberculosis Research and Epidemiology 6
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- Autophagy in Disease and Therapy 7
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- Cell death mechanisms and regulation 6
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- Protease and Inhibitor Mechanisms 5
- Co-authors
- Boris TurkVito TürkMaruša Hafner ČesenVeronika StokaGareth GriffithsMatjaž JerasMaximiliano G. GutiérrezMiomir Knežević
- Cited by
- PhysiologyImmunologyEndocrinology
In The Last Decade
Urška Repnik
67 papers receiving 3.6k citations
Peers
Comparison fields: 5 of 140
- Physiology 277
- Immunology 888
- Endocrinology 207
- Cell Biology 452
- Infectious Diseases 490
Countries citing papers authored by Urška Repnik
This map shows the geographic impact of Urška Repnik's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Urška Repnik with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Urška Repnik more than expected).
Fields of papers citing papers by Urška Repnik
This network shows the impact of papers produced by Urška Repnik. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Urška Repnik. The network helps show where Urška Repnik may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Urška Repnik, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 0 | |
| 2 | 2024 | 6 | |
| 3 | 2023 | 7 | |
| 4 | 2023 | 2 | |
| 5 | 2021 | 13 | |
| 6 | 2021 | 10 | |
| 7 | 2019 | 39 | |
| 8 | 2019 | 62 | |
| 9 | 2018 | 64 | |
| 10 | 2017 | 113 | |
| 11 | 2017 | 69 | |
| 12 | 2015 | 29 | |
| 13 | 2015 | 12 | |
| 14 | 2014 | 169 | |
| 15 | 2012 | 30 | |
| 16 | 2011 | 336 | |
| 17 | 2010 | 44 | |
| 18 | 2010 | 154 | |
| 19 | 2008 | 67 | |
| 20 | 2005 | 45 |
About Urška Repnik
Urška Repnik is a scholar working on Physiology, Endocrinology, Immunology, Parasitology and Cell Biology, having authored 68 papers that have together received 3.7k indexed citations. Recurring topics across this work include Cellular transport and secretion (11 papers), Calcium signaling and nucleotide metabolism (10 papers), Immunotherapy and Immune Responses (7 papers), Autophagy in Disease and Therapy (7 papers), Cell death mechanisms and regulation (6 papers), Tuberculosis Research and Epidemiology (6 papers), Immune cells in cancer (6 papers) and Protease and Inhibitor Mechanisms (5 papers). The work is most often cited by research in Physiology (277 citations), Immunology (888 citations), Endocrinology (207 citations), Cell Biology (452 citations) and Infectious Diseases (490 citations). Urška Repnik has collaborated with scholars based in Norway, Germany and Slovenia. Frequent co-authors include Boris Turk, Vito Türk, Maruša Hafner Česen, Veronika Stoka, Gareth Griffiths, Matjaž Jeras, Maximiliano G. Gutiérrez, Miomir Knežević, Rok Romih and Saška Ivanova. Their work appears in journals such as Journal of Biological Chemistry, PLoS Pathogens, Cold Spring Harbor Protocols, Biological Chemistry and Mitochondrion.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.