Tzuping Wei

1.5k total citations
26 papers, 1.2k citations indexed

About

Tzuping Wei is a scholar working on Physiology, Anesthesiology and Pain Medicine and Pharmacology. According to data from OpenAlex, Tzuping Wei has authored 26 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Physiology, 18 papers in Anesthesiology and Pain Medicine and 17 papers in Pharmacology. Recurrent topics in Tzuping Wei's work include Pain Mechanisms and Treatments (20 papers), Pain Management and Treatment (18 papers) and Musculoskeletal pain and rehabilitation (16 papers). Tzuping Wei is often cited by papers focused on Pain Mechanisms and Treatments (20 papers), Pain Management and Treatment (18 papers) and Musculoskeletal pain and rehabilitation (16 papers). Tzuping Wei collaborates with scholars based in United States, Germany and United Kingdom. Tzuping Wei's co-authors include Wade S. Kingery, Tian‐Zhi Guo, Wen‐Wu Li, Xiaoyou Shi, J. David Clark, David J. Clark, Liping Wang, Christopher R. Jacobs, Xiangqi Li and Rong Zhao and has published in prestigious journals such as Pain, Neuroscience and Journal of Bone and Mineral Research.

In The Last Decade

Tzuping Wei

26 papers receiving 1.2k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Tzuping Wei United States 20 797 493 371 312 180 26 1.2k
Xiangqi Li United States 25 854 1.1× 316 0.6× 249 0.7× 361 1.2× 478 2.7× 49 1.6k
Katie T. Freeman United States 18 650 0.8× 161 0.3× 191 0.5× 361 1.2× 392 2.2× 43 1.4k
Aaron P. Bloom United States 12 724 0.9× 218 0.4× 213 0.6× 369 1.2× 233 1.3× 14 1.3k
Molly A. Sevcik United States 12 967 1.2× 326 0.7× 153 0.4× 354 1.1× 249 1.4× 12 1.5k
Kyle G. Halvorson United States 14 693 0.9× 231 0.5× 131 0.4× 317 1.0× 226 1.3× 21 1.3k
Seena K. Ajit United States 24 482 0.6× 221 0.4× 180 0.5× 294 0.9× 939 5.2× 43 1.7k
David B. Mach United States 11 1.0k 1.3× 493 1.0× 233 0.6× 329 1.1× 420 2.3× 11 1.9k
Marielle J. Perreault United States 9 526 0.7× 435 0.9× 379 1.0× 115 0.4× 121 0.7× 12 947
Göran Lind Sweden 18 477 0.6× 313 0.6× 227 0.6× 298 1.0× 158 0.9× 34 1.2k
Nancy M. Luger United States 15 1.7k 2.2× 636 1.3× 351 0.9× 716 2.3× 635 3.5× 16 2.8k

Countries citing papers authored by Tzuping Wei

Since Specialization
Citations

This map shows the geographic impact of Tzuping Wei's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tzuping Wei with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tzuping Wei more than expected).

Fields of papers citing papers by Tzuping Wei

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tzuping Wei. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tzuping Wei. The network helps show where Tzuping Wei may publish in the future.

Co-authorship network of co-authors of Tzuping Wei

This figure shows the co-authorship network connecting the top 25 collaborators of Tzuping Wei. A scholar is included among the top collaborators of Tzuping Wei based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tzuping Wei. Tzuping Wei is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Guo, Tian‐Zhi, Xuanying Li, Wenwu Li, et al.. (2024). Autoantibodies cause nociceptive sensitization in a mouse model of degenerative osteoarthritis. Pain. 166(7). 1519–1531. 2 indexed citations
2.
Li, Wen‐Wu, et al.. (2023). Alpha-7 Nicotinic Acetylcholine Receptor Activation Inhibits Trauma Induced Pronociceptive Autoimmune Responses. Journal of Pain. 25(5). 104422–104422. 1 indexed citations
3.
4.
Wang, Liping, Saiyun Hou, Ilya Sabsovich, et al.. (2020). Mice lacking substance P have normal bone modeling but diminished bone formation, increased resorption, and accelerated osteopenia with aging. Bone. 144. 115806–115806. 9 indexed citations
5.
Guo, Tian‐Zhi, Tzuping Wei, Ting‐Ting Huang, Wade S. Kingery, & J. David Clark. (2018). Oxidative Stress Contributes to Fracture/Cast-Induced Inflammation and Pain in a Rat Model of Complex Regional Pain Syndrome. Journal of Pain. 19(10). 1147–1156. 29 indexed citations
6.
Wei, Tzuping, Tian‐Zhi Guo, Wen‐Wu Li, Wade S. Kingery, & J. David Clark. (2016). Acute versus chronic phase mechanisms in a rat model of CRPS. Journal of Neuroinflammation. 13(1). 14–14. 56 indexed citations
7.
Wang, Liping, Tian‐Zhi Guo, Tzuping Wei, et al.. (2016). Bisphosphonates Inhibit Pain, Bone Loss, and Inflammation in a Rat Tibia Fracture Model of Complex Regional Pain Syndrome. Anesthesia & Analgesia. 123(4). 1033–1045. 37 indexed citations
8.
Sun, Yundong, et al.. (2015). Substance P spinal signaling induces glial activation and nociceptive sensitization after fracture. Neuroscience. 310. 73–90. 63 indexed citations
9.
Shi, Xiaoyou, Tian‐Zhi Guo, Tzuping Wei, et al.. (2015). Facilitated spinal neuropeptide signaling and upregulated inflammatory mediator expression contribute to postfracture nociceptive sensitization. Pain. 156(10). 1852–1863. 32 indexed citations
10.
Guo, Tian‐Zhi, Tzuping Wei, Wen‐Wu Li, et al.. (2014). Immobilization Contributes to Exaggerated Neuropeptide Signaling, Inflammatory Changes, and Nociceptive Sensitization After Fracture in Rats. Journal of Pain. 15(10). 1033–1045. 59 indexed citations
11.
Li, Wen‐Wu, Xiaoyou Shi, Liping Wang, et al.. (2013). Epidermal adrenergic signaling contributes to inflammation and pain sensitization in a rat model of complex regional pain syndrome. Pain. 154(8). 1224–1236. 54 indexed citations
12.
Wei, Tzuping, Tian‐Zhi Guo, Wen‐Wu Li, et al.. (2012). Keratinocyte expression of inflammatory mediators plays a crucial role in substance P-induced acute and chronic pain. Journal of Neuroinflammation. 9(1). 181–181. 50 indexed citations
13.
Wang, Liping, Rong Zhao, Xiaoyou Shi, et al.. (2009). Substance P stimulates bone marrow stromal cell osteogenic activity, osteoclast differentiation, and resorption activity in vitro. Bone. 45(2). 309–320. 137 indexed citations
14.
Li, Wen‐Wu, Tian‐Zhi Guo, De‐Yong Liang, et al.. (2009). The NALP1 inflammasome controls cytokine production and nociception in a rat fracture model of complex regional pain syndrome. Pain. 147(1). 277–286. 63 indexed citations
15.
Wei, Tzuping, Wen‐Wu Li, Tian‐Zhi Guo, et al.. (2009). Post-junctional facilitation of Substance P signaling in a tibia fracture rat model of complex regional pain syndrome type I. Pain. 144(3). 278–286. 74 indexed citations
16.
Wei, Tzuping, Ilya Sabsovich, Tian‐Zhi Guo, et al.. (2008). Pentoxifylline attenuates nociceptive sensitization and cytokine expression in a tibia fracture rat model of complex regional pain syndrome. European Journal of Pain. 13(3). 253–262. 54 indexed citations
17.
Sabsovich, Ilya, Tzuping Wei, Tian‐Zhi Guo, et al.. (2008). Effect of anti-NGF antibodies in a rat tibia fracture model of complex regional pain syndrome type I. Pain. 138(1). 47–60. 96 indexed citations
18.
Sabsovich, Ilya, Tian‐Zhi Guo, Tzuping Wei, et al.. (2007). TNF signaling contributes to the development of nociceptive sensitization in a tibia fracture model of complex regional pain syndrome type I. Pain. 137(3). 507–519. 80 indexed citations
19.
Guo, Tian‐Zhi, Tzuping Wei, & Wade S. Kingery. (2006). Glucocorticoid inhibition of vascular abnormalities in a tibia fracture rat model of complex regional pain syndrome type I. Pain. 121(1). 158–167. 44 indexed citations
20.
Guo, Tian‐Zhi, Tzuping Wei, J. David Clark, et al.. (2005). Capsaicin-Sensitive Sensory Neurons Contribute to the Maintenance of Trabecular Bone Integrity. Journal of Bone and Mineral Research. 20(2). 257–267. 136 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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