Twan van den Beucken

3.7k citations

45 papers · 2.5k indexed · 1 hit paper · h-index 22

Co-authors: Bradly G. Wouters Marianne Koritzinsky Philippe Lambin Kasper M.A. Rouschop Kim G.M. Savelkouls Jan Willem Voncken Michaël G. Magagnin Ludwig J. Dubois
Topics: Cancer, Hypoxia, and Metabolism (13 papers)RNA modifications and cancer (9 papers)Endoplasmic Reticulum Stress and Disease (8 papers)
Cited by: Cancer Research Cell Biology Physiology
Journals: Proceedings of the National Academy of Sciences Journal of Biological Chemistry Journal of Clinical Investigation
Partner nations: Netherlands Canada United States

In The Last Decade

Twan van den Beucken

45 papers receiving 2.5k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

The unfolded protein response protects human tumor cells during hypoxia through regulation of the autophagy genes MAP1LC3B and ATG5

2009 654 citations Kasper M.A. Rouschop, Twan van den Beucken et al. profile →

Peers

Countries citing papers authored by Twan van den Beucken

Since Specialization

Citations

This map shows the geographic impact of Twan van den Beucken's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Twan van den Beucken with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Twan van den Beucken more than expected).

Fields of papers citing papers by Twan van den Beucken

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Twan van den Beucken. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Twan van den Beucken. The network helps show where Twan van den Beucken may publish in the future.

Co-authorship network of co-authors of Twan van den Beucken

This figure shows the co-authorship network connecting the top 25 collaborators of Twan van den Beucken. A scholar is included among the top collaborators of Twan van den Beucken based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Twan van den Beucken. Twan van den Beucken is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Immersive VR exploration of human cells: Experiences of second-year Bachelor of Health Sciences students using 360-degree video in PBL 2025 ·Research Publications (Maastricht University) ·Nynke de Jong,Herman E. Popeijus,Tanja C. Adam,Twan van den Beucken,Stefan Jongen	1
2	Advances in drug-induced liver injury research: in vitro models, mechanisms, omics and gene modulation techniques 2024 ·Cell & Bioscience ·(unknown),Twan van den Beucken	6
3	IsoTools: a flexible workflow for long-read transcriptome sequencing analysis 2023 ·Bioinformatics ·Matthias Lienhard,Twan van den Beucken,Bernd Timmermann,(unknown),Stefan T. Börno,Florian Caiment,Martin Vingron,Ralf Herwig	20
4	Human-induced pluripotent stem cells as a model for studying sporadic Alzheimer’s disease 2020 ·Neurobiology of Learning and Memory ·(unknown),Günter Kenis,Twan van den Beucken	10
5	A cross-omics approach to investigate temporal gene expression regulation by 5-hydroxymethylcytosine via TBH-derived oxidative stress showed involvement of different regulatory kinases 2018 ·Toxicology in Vitro ·Jacob J. Briedé,Lize Deferme,(unknown),Sandra M.H. Claessen,Twan van den Beucken,J. Richard Wagner,Simone G. van Breda,Jos Kleinjans	4
6	Phosphorylation of eIF2α promotes cell survival in response to benzo[a]pyrene exposure 2018 ·Toxicology in Vitro ·(unknown),Jos Kleinjans,Twan van den Beucken	3
7	Identification of essential transcription factors for adequate DNA damage response after benzo(a)pyrene and aflatoxin B1 exposure by combining transcriptomics with functional genomics 2017 ·Toxicology ·(unknown),Terezinha Souza,Danyel Jennen,Jos Kleinjans,Twan van den Beucken	32
8	Inferring transcription factor activity from microarray data reveals novel targets for toxicological investigations 2017 ·Toxicology ·Terezinha Souza,Twan van den Beucken,Jos Kleinjans,Danyel Jennen	2
9	Quantitative analysis of ChIP-seq data uncovers dynamic and sustained H3K4me3 and H3K27me3 modulation in cancer cells under hypoxia 2016 ·Epigenetics & Chromatin ·Michiel Adriaens,Peggy Prickaerts,Michelle Chan‐Seng‐Yue,Twan van den Beucken,Vivian E.H. Dahlmans,Lars Eijssen,Tim Beck,Bradly G. Wouters,Jan Willem Voncken,Chris T. Evelo	20
10	Hypoxia increases genome-wide bivalent epigenetic marking by specific gain of H3K27me3 2016 ·Epigenetics & Chromatin ·Peggy Prickaerts,Michiel Adriaens,Twan van den Beucken,Elizabeth Koch,Ludwig J. Dubois,Vivian E.H. Dahlmans,(unknown),Chris T. Evelo,Michelle Chan‐Seng‐Yue,Bradly G. Wouters,Jan Willem Voncken	62
11	Hypoxia promotes stem cell phenotypes and poor prognosis through epigenetic regulation of DICER. : Hypoxia promotes stem cell phenotypes and poor prognosis through epigenetic regulation of DICER. 2014 ·Nature Communications ·Twan van den Beucken,Elizabeth Koch,Kenneth C. Chu,Rajesha Rupaimoole,Peggy Prickaerts,Michiel Adriaens,Jan Willem Voncken,Adrian L. Harris,Francesca M. Buffa,Syed Haider,Maud H. W. Starmans,Cindy Q. Yao,Mircea Ivan,Cristina Ivan,Chad V. Pecot,Paul C. Boutros,Anil K. Sood,Marianne Koritzinsky,Bradly G. Wouters	1
12	Cigarette Smoke Extract Induces a Phenotypic Shift in Epithelial Cells; Involvement of HIF1α in Mesenchymal Transition 2014 ·PLoS ONE ·(unknown),Niki L. Reynaert,Twan van den Beucken,Harry R. Gosker,Chiel C. de Theije,Anne Verhamme,Ken R. Bracke,Emiel F.�M. Wouters,Mieke A. Dentener	36
13	Hypoxia promotes stem cell phenotypes and poor prognosis through epigenetic regulation of DICER 2014 ·Nature Communications ·Twan van den Beucken,Elizabeth Koch,Kenneth C. Chu,Rajesha Rupaimoole,Peggy Prickaerts,Michiel Adriaens,Jan Willem Voncken,Adrian L. Harris,Francesca M. Buffa,Syed Haider,Maud H. W. Starmans,Cindy Q. Yao,Mircea Ivan,Cristina Ivan,Chad V. Pecot,Paul C. Boutros,Anil K. Sood,Marianne Koritzinsky,Bradly G. Wouters	185
14	Hypoxia disrupts the Fanconi anemia pathway and sensitizes cells to chemotherapy through regulation of UBE2T 2011 ·Radiotherapy and Oncology ·(unknown),Twan van den Beucken,Alice Meng,(unknown),John Thoms,Robert G. Bristow,Bradly G. Wouters	31
15	Translational control is a major contributor to hypoxia induced gene expression 2011 ·Radiotherapy and Oncology ·Twan van den Beucken,Michaël G. Magagnin,Barry Jutten,Renaud Seigneuric,Philippe Lambin,Marianne Koritzinsky,Bradly G. Wouters	36
16	Deregulation of cap-dependent mRNA translation increases tumour radiosensitivity through reduction of the hypoxic fraction 2011 ·Radiotherapy and Oncology ·Kasper M.A. Rouschop,Ludwig J. Dubois,Marco B.E. Schaaf,Twan van den Beucken,Natasja G. Lieuwes,Tom G. Keulers,Kim G.M. Savelkouls,Johan Bussink,Albert J. van der Kogel,Marianne Koritzinsky,Bradly G. Wouters	20
17	Proteomic analysis of gene expression following hypoxia and reoxygenation reveals proteins involved in the recovery from endoplasmic reticulum and oxidative stress 2007 ·Radiotherapy and Oncology ·Michaël G. Magagnin,Kjell Sergeant,Twan van den Beucken,Kasper M.A. Rouschop,Barry Jutten,Renaud Seigneuric,Philippe Lambin,Bart Devreese,Marianne Koritzinsky,Bradly G. Wouters	16
18	Phosphorylation of eIF2α is required for mRNA translation inhibition and survival during moderate hypoxia 2007 ·Radiotherapy and Oncology ·Marianne Koritzinsky,Kasper M.A. Rouschop,Twan van den Beucken,Michaël G. Magagnin,Kim G.M. Savelkouls,Philippe Lambin,Bradly G. Wouters	48
19	Translational control of gene expression during hypoxia 2006 ·Cancer Biology & Therapy ·Twan van den Beucken,Marianne Koritzinsky,Bradly G. Wouters	113
20	The hypoxic proteome is influenced by gene-specific changes in mRNA translation 2005 ·Radiotherapy and Oncology ·Marianne Koritzinsky,Renaud Seigneuric,Michaël G. Magagnin,Twan van den Beucken,Philippe Lambin,Bradly G. Wouters	92

About Twan van den Beucken

Twan van den Beucken is a scholar working on Cancer Research, Cell Biology and Physiology, having authored 45 papers that have together received 2.5k indexed citations. Recurring topics across this work include Cancer, Hypoxia, and Metabolism (13 papers), RNA modifications and cancer (9 papers) and Endoplasmic Reticulum Stress and Disease (8 papers). The work is most often cited by research in Cancer Research (940 citations), Cell Biology (681 citations) and Physiology (107 citations). Twan van den Beucken has collaborated with scholars based in Netherlands, Canada and United States. Frequent co-authors include Bradly G. Wouters, Marianne Koritzinsky, Philippe Lambin, Kasper M.A. Rouschop, Kim G.M. Savelkouls, Jan Willem Voncken, Michaël G. Magagnin, Ludwig J. Dubois, Johan Bussink and Albert J. van der Kogel. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Journal of Clinical Investigation.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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