Travis W. Hein

4.3k total citations
87 papers, 3.6k citations indexed

About

Travis W. Hein is a scholar working on Physiology, Molecular Biology and Ophthalmology. According to data from OpenAlex, Travis W. Hein has authored 87 papers receiving a total of 3.6k indexed citations (citations by other indexed papers that have themselves been cited), including 47 papers in Physiology, 33 papers in Molecular Biology and 24 papers in Ophthalmology. Recurrent topics in Travis W. Hein's work include Nitric Oxide and Endothelin Effects (46 papers), Retinal Diseases and Treatments (17 papers) and Glaucoma and retinal disorders (15 papers). Travis W. Hein is often cited by papers focused on Nitric Oxide and Endothelin Effects (46 papers), Retinal Diseases and Treatments (17 papers) and Glaucoma and retinal disorders (15 papers). Travis W. Hein collaborates with scholars based in United States, Japan and Canada. Travis W. Hein's co-authors include Lih Kuo, Cuihua Zhang, James C. Liao, Wei Wang, Wenjuan Xu, Robert H. Rosa, Yi Ren, Kuang‐Tse Huang, Mark W. Vaughn and Luiz Belardinelli and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Circulation and Circulation Research.

In The Last Decade

Travis W. Hein

86 papers receiving 3.5k citations

Peers

Travis W. Hein
Emmanuel S. Buys United States
T A Brock United States
William I. Rosenblum United States
Mara Lorenzi United States
M. Paul Germany
Aqeela Afzal United States
Emmanuel S. Buys United States
Travis W. Hein
Citations per year, relative to Travis W. Hein Travis W. Hein (= 1×) peers Emmanuel S. Buys

Countries citing papers authored by Travis W. Hein

Since Specialization
Citations

This map shows the geographic impact of Travis W. Hein's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Travis W. Hein with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Travis W. Hein more than expected).

Fields of papers citing papers by Travis W. Hein

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Travis W. Hein. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Travis W. Hein. The network helps show where Travis W. Hein may publish in the future.

Co-authorship network of co-authors of Travis W. Hein

This figure shows the co-authorship network connecting the top 25 collaborators of Travis W. Hein. A scholar is included among the top collaborators of Travis W. Hein based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Travis W. Hein. Travis W. Hein is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Hein, Travis W., et al.. (2025). Activation of Smooth Muscle Kir2.1 Channels and Na+/K+-ATPase Mediates Dilation of Porcine Coronary Arterioles at Physiological Levels of Potassium. International Journal of Molecular Sciences. 26(6). 2654–2654. 1 indexed citations
2.
Rosa, Robert H., Min Zhao, Shu‐Huai Tsai, et al.. (2022). Intravitreal Administration of Stanniocalcin-1 Rescues Photoreceptor Degeneration with Reduced Oxidative Stress and Inflammation in a Porcine Model of Retinitis Pigmentosa. American Journal of Ophthalmology. 239. 230–243. 6 indexed citations
3.
Zhao, Min, et al.. (2019). Stanniocalcin-1 enhances ellipsoid zone intensity and cone function in the P23H rhodopsin transgenic pig. Investigative Ophthalmology & Visual Science. 60(9). 452–452. 1 indexed citations
4.
Hein, Travis W., Xin Xu, Yi Ren, et al.. (2019). Requisite roles of LOX-1, JNK, and arginase in diabetes-induced endothelial vasodilator dysfunction of porcine coronary arterioles. Journal of Molecular and Cellular Cardiology. 131. 82–90. 17 indexed citations
5.
Zhao, Min, Shu‐Huai Tsai, Wenjuan Xu, et al.. (2018). Data on SD-OCT image acquisition, ultrastructural features, and horizontal tissue shrinkage in the porcine retina. Data in Brief. 21. 1019–1025. 5 indexed citations
6.
Zhao, Min, Shu‐Huai Tsai, Wenjuan Xu, et al.. (2018). Correlation of spectral domain optical coherence tomography with histology and electron microscopy in the porcine retina. Experimental Eye Research. 177. 181–190. 37 indexed citations
7.
Thengchaisri, Naris, Travis W. Hein, Yi Ren, & Lih Kuo. (2015). Endothelin-1 impairs coronary arteriolar dilation: Role of p38 kinase-mediated superoxide production from NADPH oxidase. Journal of Molecular and Cellular Cardiology. 86. 75–84. 28 indexed citations
8.
Kuo, Lih & Travis W. Hein. (2013). Vasomotor Regulation of Coronary Microcirculation by Oxidative Stress: Role of Arginase. Frontiers in Immunology. 4. 237–237. 27 indexed citations
9.
Hayenga, Heather N., Jin Hu, Emily Wilson, et al.. (2012). Differential Progressive Remodeling of Coronary and Cerebral Arteries and Arterioles in an Aortic Coarctation Model of Hypertension. Frontiers in Physiology. 3. 420–420. 15 indexed citations
10.
Nagaoka, Taiji, Tomofumi Tani, Kenji Sogawa, et al.. (2012). Retinal arteriolar responses to acute severe elevation in systemic blood pressure in cats: Role of endothelium-derived factors. Experimental Eye Research. 103. 63–70. 16 indexed citations
11.
Lu, Guangrong, Travis W. Hein, & Lih Kuo. (2010). Rho Kinase-mediated Coronary Arteriolar Constriction to Endothelin-1: Mechanistic Implications for Cardiac Syndrome X. 1(2). 1 indexed citations
12.
Wang, Wei, Travis W. Hein, Cuihua Zhang, et al.. (2010). Oxidized Low-Density Lipoprotein Inhibits Nitric Oxide-Mediated Coronary Arteriolar Dilation by Up-regulating Endothelial Arginase I. Microcirculation. 18(1). 36–45. 35 indexed citations
14.
Hein, Travis W., Uma Shankar Singh, Jeannette Vásquez‐Vivar, et al.. (2009). Human C-reactive protein induces endothelial dysfunction and uncoupling of eNOS in vivo. Atherosclerosis. 206(1). 61–68. 129 indexed citations
15.
Olszewska-Pazdrak, Barbara, et al.. (2009). Chronic hypoxia attenuates VEGF signaling and angiogenic responses by downregulation of KDR in human endothelial cells. American Journal of Physiology-Cell Physiology. 296(5). C1162–C1170. 84 indexed citations
16.
Rosa, Robert H., et al.. (2006). Brimonidine evokes heterogeneous vasomotor response of retinal arterioles: diminished nitric oxide-mediated vasodilation when size goes small. American Journal of Physiology-Heart and Circulatory Physiology. 291(1). H231–H238. 1 indexed citations
17.
Hein, Travis W., Wenjuan Xu, & Lih Kuo. (2006). Dilation of Retinal Arterioles in Response to Lactate: Role of Nitric Oxide, Guanylyl Cyclase, and ATP-Sensitive Potassium Channels. Investigative Ophthalmology & Visual Science. 47(2). 693–693. 98 indexed citations
18.
Yuan, Zilong, Robert H. Rosa, Travis W. Hein, & Lih Kuo. (2005). Correlation of Tonometric and Direct Measurements of Intraocular Pressure in the Porcine Eye. Investigative Ophthalmology & Visual Science. 46(13). 3674–3674. 2 indexed citations
19.
Hein, Travis W., et al.. (2005). Requisite Roles of A2AReceptors, Nitric Oxide, and KATPChannels in Retinal Arteriolar Dilation in Response to Adenosine. Investigative Ophthalmology & Visual Science. 46(6). 2113–2113. 68 indexed citations
20.
Hein, Travis W., Wei Wang, Behyar Zoghi, Mariappan Muthuchamy, & Lih Kuo. (2001). Functional and Molecular Characterization of Receptor Subtypes Mediating Coronary Microvascular Dilation to Adenosine. Journal of Molecular and Cellular Cardiology. 33(2). 271–282. 73 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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