Toshihito Seki
- Hepatology top 0.2%
- Epidemiology top 2%
- Molecular Biology top 5%
- Oncology top 2%
- Surgery top 5%
- Co-authors
- Kyoichi InoueKazuichi OkazakiTaiichi NakagawaKoichi MatsuzakiMasayuki WakabayashiKatsunori YoshidaNoriyo YamashikiToru Tamai
- Topics
- Hepatocellular Carcinoma Treatment and Prognosis (40 papers)Liver Disease Diagnosis and Treatment (27 papers)Pancreatic and Hepatic Oncology Research (18 papers)
- Cited by
- HepatologyEpidemiologyOncology
- Journals
- Proceedings of the National Academy of SciencesApplied and Environmental MicrobiologyHepatology
- Partner nations
- JapanUnited StatesGermany
In The Last Decade
Toshihito Seki
117 papers receiving 4.5k citations
Hit Papers
Peers
Comparison fields: 5 of 123
- Hepatology 2.4k
- Epidemiology 1.5k
- Molecular Biology 1.4k
- Oncology 1.2k
- Surgery 782
Countries citing papers authored by Toshihito Seki
This map shows the geographic impact of Toshihito Seki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Toshihito Seki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Toshihito Seki more than expected).
Fields of papers citing papers by Toshihito Seki
This network shows the impact of papers produced by Toshihito Seki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Toshihito Seki. The network helps show where Toshihito Seki may publish in the future.
Co-authorship network of co-authors of Toshihito Seki
This figure shows the co-authorship network connecting the top 25 collaborators of Toshihito Seki. A scholar is included among the top collaborators of Toshihito Seki based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Toshihito Seki. Toshihito Seki is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 3 | |
| 3 | 113 | |
| 4 | 0 | |
| 5 | 36 | |
| 6 | 120 | |
| 7 | 1 | |
| 8 | 2 | |
| 9 | 1 | |
| 10 | 2 | |
| 11 | 27 | |
| 12 | 1 | |
| 13 | 5 | |
| 14 | 2 | |
| 15 | 1 | |
| 16 | 1 | |
| 17 | 0 | |
| 18 | 22 | |
| 19 | 1 | |
| 20 | 1 |
About Toshihito Seki
Toshihito Seki is a scholar working on Hepatology, Pharmacology and Oncology, having authored 127 papers that have together received 4.7k indexed citations. Recurring topics across this work include Hepatocellular Carcinoma Treatment and Prognosis (40 papers), Liver Disease Diagnosis and Treatment (27 papers) and Pancreatic and Hepatic Oncology Research (18 papers). The work is most often cited by research in Hepatology (2.4k citations), Epidemiology (1.5k citations) and Oncology (1.2k citations). Toshihito Seki has collaborated with scholars based in Japan, United States and Germany. Frequent co-authors include Kyoichi Inoue, Kazuichi Okazaki, Taiichi Nakagawa, Koichi Matsuzaki, Masayuki Wakabayashi, Katsunori Yoshida, Noriyo Yamashiki, Toru Tamai, Jun–ichi Fujisawa and Akira Nishimura. Their work appears in journals such as Proceedings of the National Academy of Sciences, Applied and Environmental Microbiology and Hepatology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.