Toril Tefre

632 total citations
11 papers, 526 citations indexed

About

Toril Tefre is a scholar working on Molecular Biology, Pharmacology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Toril Tefre has authored 11 papers receiving a total of 526 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 3 papers in Pharmacology and 2 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Toril Tefre's work include DNA Repair Mechanisms (3 papers), Pharmacogenetics and Drug Metabolism (3 papers) and Protein Kinase Regulation and GTPase Signaling (2 papers). Toril Tefre is often cited by papers focused on DNA Repair Mechanisms (3 papers), Pharmacogenetics and Drug Metabolism (3 papers) and Protein Kinase Regulation and GTPase Signaling (2 papers). Toril Tefre collaborates with scholars based in Norway, United States and Sweden. Toril Tefre's co-authors include Jeffrey R. Idle, Ann K. Daly, Vidar M. Steen, Anne‐Lise Børresen, Ole A. Andreassen, Aage Haugen, David Ryberg, Vidar Skaug, Daniel W. Nebert and Steinar Aamdal and has published in prestigious journals such as Nucleic Acids Research, Environmental Health Perspectives and British Journal of Cancer.

In The Last Decade

Toril Tefre

11 papers receiving 516 citations

Peers

Countries citing papers authored by Toril Tefre

Since Specialization

Citations

This map shows the geographic impact of Toril Tefre's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Toril Tefre with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Toril Tefre more than expected).

Fields of papers citing papers by Toril Tefre

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Toril Tefre. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Toril Tefre. The network helps show where Toril Tefre may publish in the future.

Co-authorship network of co-authors of Toril Tefre

This figure shows the co-authorship network connecting the top 25 collaborators of Toril Tefre. A scholar is included among the top collaborators of Toril Tefre based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Toril Tefre. Toril Tefre is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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11 of 11 papers shown

#	Work	Indexed citations
1	Differences in serum levels of CB-153 and p,p′-DDE, and reproductive parameters between men living south and north in Norway 2011 ·Reproductive Toxicology ·Trine B. Haugen, Toril Tefre, Gunilla Malm, Bo Jönsson, Lars Rylander, Lars Hagmar, (unknown), (unknown), Thomas Sæther, Yngve Figenschau, Aleksander Giwercman	27
2	Linkage disequilibrium pattern of the ATM gene in breast cancer patients and controls; association of SNPs and haplotypes to radio-sensitivity and post-lumpectomy local recurrence 2007 ·Radiation Oncology ·Hege Edvardsen, Toril Tefre, (unknown), Phuong Vu, Bruce G. Haffty, Sophie D. Fosså, Vessela N. Kristensen, A.L. Børresen-Dale	30
3	Detection of the poor metabolizer-associated CYP2D6(D) gene deletion allele by long-PCR technology 1995 ·Pharmacogenetics ·Vidar M. Steen, Ole A. Andreassen, Ann K. Daly, Toril Tefre, Anne‐Lise Børresen, Jeffrey R. Idle, (unknown)	212
4	Genotyping of the CYP 2D6 gene in Norwegian lung cancer patients and controls 1994 ·Pharmacogenetics ·Toril Tefre, Ann K. Daly, Martin Armstrong, Julian Leathart, Jeffrey R. Idle, (unknown), (unknown)	28
5	Allele diversity of the H-ras-1 variable number of tandem repeats in Norwegian lung cancer patients. 1992 ·Environmental Health Perspectives ·David Ryberg, Toril Tefre, Vidar Skaug, Lodve Stangeland, Steinar Øvrebø, Anne Naalsund, A L Børresen, Aage Haugen	4
6	Allele Diversity of the H-ras-1 Variable Number of Tandem Repeats in Norwegian Lung Cancer Patients 1992 ·Environmental Health Perspectives ·David Ryberg, Toril Tefre, Vidar Skaug, Lodve Stangeland, Steinar Øvrebø, Anne Naalsund, Anne‐Lise Børresen, Aage Haugen	1
7	Human CYP1A1 (cytochrome P1450) gene: lack of association between the Msp I restriction fragment length polymorphism and incidence of lung cancer in a Norwegian population 1991 ·Pharmacogenetics ·Toril Tefre, David Ryberg, Aage Haugen, Daniel W. Nebert, Vidar Skaug, (unknown), (unknown)	136
8	Studies of the L-myc DNA polymorphism and relation to metastasis in Norwegian lung cancer patients 1990 ·British Journal of Cancer ·Toril Tefre, A-L Børresen, Steinar Aamdal, Anton Brögger	38
9	Pstl Polymorphism at the human P₁450 gene on chromosome 15 1990 ·Nucleic Acids Research ·Aage Haugen, James C. Willey, A L Børresen, Toril Tefre	7
10	Ha-ras-1 alleles in Norwegian lung cancer patients 1990 ·Human Genetics ·David Ryberg, Toril Tefre, (unknown), Vidar Skaug, Lodve Stangeland, Anne Naalsund, (unknown), (unknown), Aage Haugen	14
11	Inhibition of very-low-density lipoprotein secretion by chloroquine, verapamil and monensin takes place in the Golgi complex 1987 ·Biochimica et Biophysica Acta (BBA) - Molecular Cell Research ·Arild C. Rustan, Jon Ø. Nossen, Toril Tefre, Christian A. Drevon	29

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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