Tomoya Terashima

1.8k total citations
53 papers, 1.4k citations indexed

About

Tomoya Terashima is a scholar working on Cellular and Molecular Neuroscience, Neurology and Physiology. According to data from OpenAlex, Tomoya Terashima has authored 53 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Cellular and Molecular Neuroscience, 15 papers in Neurology and 14 papers in Physiology. Recurrent topics in Tomoya Terashima's work include Pain Mechanisms and Treatments (12 papers), Nerve injury and regeneration (12 papers) and Neuroinflammation and Neurodegeneration Mechanisms (8 papers). Tomoya Terashima is often cited by papers focused on Pain Mechanisms and Treatments (12 papers), Nerve injury and regeneration (12 papers) and Neuroinflammation and Neurodegeneration Mechanisms (8 papers). Tomoya Terashima collaborates with scholars based in Japan, United States and United Kingdom. Tomoya Terashima's co-authors include Hideto Kojima, Miwako Katagi, Hitoshi Yasuda, Lawrence Chan, Hiromichi Kawai, Junko Okano, Hiroshi Maegawa, Yuki Nakae, Kengo Maeda and Masashi Fujitani and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Clinical Investigation and Nature Communications.

In The Last Decade

Tomoya Terashima

48 papers receiving 1.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Tomoya Terashima Japan 21 432 334 271 236 178 53 1.4k
Haixia Lü China 24 840 1.9× 287 0.9× 315 1.2× 246 1.0× 81 0.5× 82 1.9k
Murat Digicaylioglu United States 19 768 1.8× 228 0.7× 316 1.2× 313 1.3× 195 1.1× 28 1.9k
Inja Lim South Korea 20 594 1.4× 259 0.8× 242 0.9× 223 0.9× 83 0.5× 45 1.3k
Hitomi Kurinami Japan 25 600 1.4× 668 2.0× 311 1.1× 372 1.6× 101 0.6× 38 1.9k
Yu-Ping Peng China 26 657 1.5× 205 0.6× 431 1.6× 647 2.7× 318 1.8× 80 2.0k
Joanna Lewin‐Kowalik Poland 22 460 1.1× 416 1.2× 495 1.8× 170 0.7× 187 1.1× 106 1.6k
Suelen Adriani Marques Brazil 15 197 0.5× 315 0.9× 354 1.3× 149 0.6× 58 0.3× 25 893
Cha‐Gyun Jung Japan 24 837 1.9× 303 0.9× 343 1.3× 214 0.9× 189 1.1× 56 1.6k
Kudret Türeyen Türkiye 18 371 0.9× 145 0.4× 278 1.0× 440 1.9× 155 0.9× 26 1.2k

Countries citing papers authored by Tomoya Terashima

Since Specialization
Citations

This map shows the geographic impact of Tomoya Terashima's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tomoya Terashima with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tomoya Terashima more than expected).

Fields of papers citing papers by Tomoya Terashima

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tomoya Terashima. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tomoya Terashima. The network helps show where Tomoya Terashima may publish in the future.

Co-authorship network of co-authors of Tomoya Terashima

This figure shows the co-authorship network connecting the top 25 collaborators of Tomoya Terashima. A scholar is included among the top collaborators of Tomoya Terashima based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tomoya Terashima. Tomoya Terashima is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kitamura, A., et al.. (2025). Brain natriuretic peptide changes as a potential biomarker for paroxysmal atrial fibrillation in acute ischemic stroke cases. Journal of Stroke and Cerebrovascular Diseases. 34(12). 108470–108470.
2.
Tsuji, Shunichiro, et al.. (2025). Pro-inflammatory microglia-targeted peptide therapy ameliorates neonatal hypoxic-ischemic encephalopathy in mice. Molecular Therapy. 33(7). 3177–3194.
3.
Kojima, Hideto, Tomoya Terashima, Miwako Katagi, et al.. (2023). Expression of proinflammatory cytokines and proinsulin by bone marrow-derived cells for fracture healing in long-term diabetic mice. BMC Musculoskeletal Disorders. 24(1). 585–585. 5 indexed citations
4.
Terashima, Tomoya, Shunichiro Tsuji, Miwako Katagi, et al.. (2022). Ambient Temperature Is Correlated With the Severity of Neonatal Hypoxic-Ischemic Brain Injury via Microglial Accumulation in Mice. Frontiers in Pediatrics. 10. 883556–883556. 2 indexed citations
6.
Katagi, Miwako, Tomoya Terashima, Natsuko Ohashi, et al.. (2021). Malfunctioning CD106-positive, short-term hematopoietic stem cells trigger diabetic neuropathy in mice by cell fusion. Communications Biology. 4(1). 575–575. 7 indexed citations
7.
Ohashi, Natsuko, Tomoya Terashima, Miwako Katagi, et al.. (2021). GLT1 gene delivery based on bone marrow-derived cells ameliorates motor function and survival in a mouse model of ALS. Scientific Reports. 11(1). 12803–12803. 9 indexed citations
8.
9.
Terashima, Tomoya, Nobuhiro Ogawa, Toshiyuki Sato, et al.. (2019). Advanced Technology for Gene Delivery with Homing Peptides to Spinal Cord through Systemic Circulation in Mice. Molecular Therapy — Methods & Clinical Development. 13. 474–483. 2 indexed citations
10.
Terashima, Tomoya, Nobuhiro Ogawa, Yuki Nakae, et al.. (2018). Gene Therapy for Neuropathic Pain through siRNA-IRF5 Gene Delivery with Homing Peptides to Microglia. Molecular Therapy — Nucleic Acids. 11. 203–215. 52 indexed citations
11.
Yamashita, Hirofumi, Nobukatsu Sawamoto, Hiromichi Kawai, et al.. (2017). Charcot–Marie–Tooth disease type 2A with an autosomal-recessive inheritance: the first report of an adult-onset disease. Journal of Human Genetics. 63(1). 89–92. 10 indexed citations
12.
Okano, Junko, Hideto Kojima, Miwako Katagi, et al.. (2015). Epidermis–dermis junction as a novel location for bone marrow-derived cells to reside in response to ionizing radiation. Biochemical and Biophysical Research Communications. 461(4). 695–701. 5 indexed citations
13.
Terashima, Tomoya, et al.. (2014). Bone marrow-derived TNF-α causes diabetic neuropathy in mice. Diabetologia. 58(2). 402–410. 28 indexed citations
14.
Ogawa, Nobuhiro, Hiromichi Kawai, Tomoya Terashima, et al.. (2014). Gene Therapy for Neuropathic Pain by Silencing of TNF-α Expression with Lentiviral Vectors Targeting the Dorsal Root Ganglion in Mice. PLoS ONE. 9(3). e92073–e92073. 49 indexed citations
15.
Tamiya, Gen, Satoshi Makino, Makiko Hayashi, et al.. (2014). A Mutation of COX6A1 Causes a Recessive Axonal or Mixed Form of Charcot-Marie-Tooth Disease. The American Journal of Human Genetics. 95(3). 294–300. 63 indexed citations
16.
Kojima, Hideto, Lawrence Chan, Tomoya Terashima, et al.. (2013). Haematopoietic cells produce BDNF and regulate appetite upon migration to the hypothalamus. Nature Communications. 4(1). 1526–1526. 33 indexed citations
17.
Takemura, Y, Shinji Imai, Hideto Kojima, et al.. (2012). Brain-Derived Neurotrophic Factor from Bone Marrow-Derived Cells Promotes Post-Injury Repair of Peripheral Nerve. PLoS ONE. 7(9). e44592–e44592. 36 indexed citations
18.
Li, Rongying, Antoni Paul, Kerry W.S. Ko, et al.. (2011). Interleukin-7 induces recruitment of monocytes/macrophages to endothelium. European Heart Journal. 33(24). 3114–3123. 48 indexed citations
19.
Chan, Lawrence, et al.. (2011). Pathogenesis of diabetic neuropathy: bad to the bone. Annals of the New York Academy of Sciences. 1240(1). 70–76. 23 indexed citations
20.
Terashima, Tomoya, et al.. (2009). DRG-targeted helper-dependent adenoviruses mediate selective gene delivery for therapeutic rescue of sensory neuronopathies in mice. Journal of Clinical Investigation. 119(7). 2100–112. 27 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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