Tomoya Nishimura

814 citations
17 papers · 246 · h-index 8

Impact in

    • Hereditary Neurological Disorders
    • Genetic Neurodegenerative Diseases
    • Neurological diseases and metabolism
    • Botulinum Toxin and Related Neurological Disorders
    • Amyotrophic Lateral Sclerosis Research

Papers in

Tomoya Nishimura

15 papers receiving 244 citations

Peers

Tomoya Nishimura
Comparison fields: 5 of 62
  • Cellular and Molecular Neuroscience 111
  • Neurology 50
  • Developmental Neuroscience 21
  • Neurology 62
  • Genetics 22
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Katrin Meuer Germany
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Claudia Stendel Germany
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Citations per field
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Citations per year

Countries citing papers authored by Tomoya Nishimura

Since Specialization
Citations

This map shows the geographic impact of Tomoya Nishimura's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tomoya Nishimura with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tomoya Nishimura more than expected).

Fields of papers citing papers by Tomoya Nishimura

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tomoya Nishimura. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tomoya Nishimura. The network helps show where Tomoya Nishimura may publish in the future.

Co-authors

The 25 scholars most cited alongside Tomoya Nishimura, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Tomoya Nishimura Line = papers co-authored together Tomoya Nishimura links everyone, so they are left out of the graph.

All Works

17 of 17 papers shown
#Work
1 199466
2 199343
3 199630
4 199720
5 199719
6 199917
7 199516
8 199315
9 19966
10 20203
11 20243
12 19953
13 20213
14 20231
15 19961
16 20240
17
[A case of spinal muscular atrophy with marked calf hypertrophy and adolescent onset].
20000

About Tomoya Nishimura

Tomoya Nishimura is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience, Neurology, Neurology and Genetics, having authored 17 papers that have together received 246 indexed citations. Recurring topics across this work include Hereditary Neurological Disorders (6 papers), Neurological diseases and metabolism (5 papers), Botulinum Toxin and Related Neurological Disorders (3 papers), RNA Research and Splicing (2 papers), Neurogenetic and Muscular Disorders Research (2 papers), Methane Hydrates and Related Phenomena (2 papers), Cephalopods and Marine Biology (1 paper) and Signaling Pathways in Disease (1 paper). The work is most often cited by research in Cellular and Molecular Neuroscience (111 citations), Neurology (50 citations), Developmental Neuroscience (21 citations), Neurology (62 citations) and Genetics (22 citations). Tomoya Nishimura has collaborated with scholars based in Japan and United States. Frequent co-authors include Takehiko Yanagihara, Harutoshi Fujimura, Hiroaki Yoshikawa, Saburo Sakoda, Yoichi Yamamoto, Y Ogawa, Tadamitsu Kishimoto, Eri Adachi, Satoshi Ueno and Yoshinaga Saeki. Their work appears in journals such as Acta Neuropathologica, Microbes and Environments, Journal of the Neurological Sciences, Pharmaceutical Research and FEBS Letters.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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