Tomohisa Inada

1.2k total citations · 1 hit paper
13 papers, 970 citations indexed

About

Tomohisa Inada is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Physiology. According to data from OpenAlex, Tomohisa Inada has authored 13 papers receiving a total of 970 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 4 papers in Cellular and Molecular Neuroscience and 4 papers in Physiology. Recurrent topics in Tomohisa Inada's work include Axon Guidance and Neuronal Signaling (4 papers), Spaceflight effects on biology (3 papers) and Angiogenesis and VEGF in Cancer (3 papers). Tomohisa Inada is often cited by papers focused on Axon Guidance and Neuronal Signaling (4 papers), Spaceflight effects on biology (3 papers) and Angiogenesis and VEGF in Cancer (3 papers). Tomohisa Inada collaborates with scholars based in Japan, United States and Germany. Tomohisa Inada's co-authors include Toshio Suda, Fumio Arai, Osamu Ohneda, Kenneth Brasel, Tetsuo Sudo, Dirk Anderson, Takeshi Miyamoto, Takashi Miyata, Xiuqin Zhang and Yuichi Oike and has published in prestigious journals such as The Journal of Experimental Medicine, Blood and PLoS ONE.

In The Last Decade

Tomohisa Inada

13 papers receiving 942 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Commitment and Differentiation of Osteoclast Precursor Cells by the Sequential Expression of C-Fms and Receptor Activator of Nuclear Factor κb (Rank) Receptors

1999 575 citations Fumio Arai, Takeshi Miyamoto et al. The Journal of Experimental Medicine profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Tomohisa Inada Japan	9	750	362	236	152	135	13	970
Seiko Kyakumoto Japan	17	590 0.8×	175 0.5×	83 0.4×	77 0.5×	107 0.8×	47	946
D. W. Burton United States	23	599 0.8×	447 1.2×	124 0.5×	59 0.4×	24 0.2×	38	1.2k
Nicholas R. Hum United States	21	524 0.7×	316 0.9×	53 0.2×	183 1.2×	169 1.3×	50	1.1k
Elisheva Smith United States	13	900 1.2×	262 0.7×	32 0.1×	93 0.6×	74 0.5×	14	1.2k
Gabrielle Curinga United States	9	571 0.8×	176 0.5×	96 0.4×	63 0.4×	98 0.7×	11	1.4k
Alfred C. Aplin United States	19	568 0.8×	105 0.3×	75 0.3×	138 0.9×	178 1.3×	31	953
Jin Fan China	18	712 0.9×	89 0.2×	51 0.2×	302 2.0×	143 1.1×	30	1.2k
Álvaro Valín Spain	14	473 0.6×	218 0.6×	50 0.2×	80 0.5×	82 0.6×	22	697
Lucas C. Armstrong United States	14	639 0.9×	95 0.3×	40 0.2×	178 1.2×	62 0.5×	23	1.0k
D L Goad United States	10	543 0.7×	253 0.7×	24 0.1×	134 0.9×	49 0.4×	11	832

Countries citing papers authored by Tomohisa Inada

Since Specialization

Citations

This map shows the geographic impact of Tomohisa Inada's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tomohisa Inada with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tomohisa Inada more than expected).

Fields of papers citing papers by Tomohisa Inada

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tomohisa Inada. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tomohisa Inada. The network helps show where Tomohisa Inada may publish in the future.

Co-authorship network of co-authors of Tomohisa Inada

This figure shows the co-authorship network connecting the top 25 collaborators of Tomohisa Inada. A scholar is included among the top collaborators of Tomohisa Inada based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tomohisa Inada. Tomohisa Inada is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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13 of 13 papers shown

Shiba, Naoto, Hiroo Matsuse, Yoshio Takano, et al.. (2015). Electrically Stimulated Antagonist Muscle Contraction Increased Muscle Mass and Bone Mineral Density of One Astronaut - Initial Verification on the International Space Station. PLoS ONE. 10(8). e0134736–e0134736. 20 indexed citations

Shiba, Naoto, Hiroo Matsuse, Yoshio Takano, et al.. (2010). Development of a Training Method for Weightless Environment Using Both Electrical Stimulation and Voluntary Muscle Contraction. The Tohoku Journal of Experimental Medicine. 220(1). 83–93. 13 indexed citations

Karube, Kennosuke, Junji Suzumiya, Masataka Okamoto, et al.. (2007). Adult T-cell Lymphoma/Leukemia With Angioimmunoblastic T-cell Lymphomalike Features: Report of 11 Cases. The American Journal of Surgical Pathology. 31(2). 216–223. 21 indexed citations

Inoue, Takashi, et al.. (2007). Locomotion Assistance for the Person with Mobility Impairment: Fuzzy Control of Cycling Movement by Means of Surface Electrical-Stimulation. Conference proceedings. 8. 2420–2423. 6 indexed citations

Takeuchi, Ryo, et al.. (2006). A Countermeasure for Preventing Atrophy of Musculoskeletal System under Microgravity. 2006 SICE-ICASE International Joint Conference. 16. 5862–5867. 1 indexed citations

Shiba, Naoto, Takashi Maeda, Keisuke Hirota, et al.. (2004). STUDY OF THE DEVELOPMENT OF AN EFFECTIVE DEVICE FOR MAINTAINING SKELETAL MUSCLE IN SPACE. Biomechanisms. 17(0). 49–56. 1 indexed citations

Oike, Yuichi, Yasuhiro Ito, Koichi Hamada, et al.. (2002). Regulation of vasculogenesis and angiogenesis by EphB/ephrin-B2 signaling between endothelial cells and surrounding mesenchymal cells. Blood. 100(4). 1326–1333. 92 indexed citations

Suenobu, Souichi, Nobuyuki Takakura, Tomohisa Inada, et al.. (2002). A role of EphB4 receptor and its ligand, ephrin-B2, in erythropoiesis. Biochemical and Biophysical Research Communications. 293(3). 1124–1131. 59 indexed citations

Zhang, Xiuqin, Nobuyuki Takakura, Yuichi Oike, et al.. (2001). Stromal cells expressing ephrin-B2 promote the growth and sprouting of ephrin-B2+ endothelial cells. Blood. 98(4). 1028–1037. 60 indexed citations

10.

Arai, Fumio, Takeshi Miyamoto, Osamu Ohneda, et al.. (1999). Commitment and Differentiation of Osteoclast Precursor Cells by the Sequential Expression of C-Fms and Receptor Activator of Nuclear Factor κb (Rank) Receptors. The Journal of Experimental Medicine. 190(12). 1741–1754. 575 indexed citations breakdown →

11.

Inada, Tomohisa, Atsushi Iwama, Seiji Sakano, et al.. (1997). Selective Expression of the Receptor Tyrosine Kinase, HTK, on Human Erythroid Progenitor Cells. Blood. 89(8). 2757–2765. 41 indexed citations

12.

Sakano, Seiji, Tomohisa Inada, Atsushi Iwama, et al.. (1996). Characterization of a ligand for receptor protein-tyrosine kinase HTK expressed in immature hematopoietic cells.. PubMed. 13(4). 813–22. 76 indexed citations

13.

Inada, Tomohisa, Kenji Shirono, & Hiroyuki Tsuda. (1995). Hemolytic Anemia in a Patient with Subacute Bacterial Endocarditis Due to <i>Streptococcus sanguis</i>. Acta Haematologica. 94(2). 95–97. 5 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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