Tomoh Masaki

1.3k citations
13 papers · 1.2k indexed · h-index 11
Topics
Nitric Oxide and Endothelin Effects (10 papers)Receptor Mechanisms and Signaling (5 papers)Ion channel regulation and function (4 papers)
Partner nations
JapanUnited States

In The Last Decade

Tomoh Masaki

13 papers receiving 1.1k citations

Peers

Tomoh Masaki
Comparison fields: 5 of 78
  • Physiology 763
  • Cardiology and Cardiovascular Medicine 495
  • Molecular Biology 409
  • Immunology 139
  • Cellular and Molecular Neuroscience 129
Replace David G. Harrison with:
David G. Harrison United States
Mark Pullen United States
Y Hirata Japan
Michel Skaf United States
M. Nishikibe Japan
Adriana V. Treuer Chile
Paul Fransen Belgium
K. Yasunari Japan
Alex L. Loeb United States
M. Ihara Japan
Tomoh Masaki relative to David G. Harrison United States David G. Harrison's profile →
Citations per field
00.5×1.6×
David G. Harrison · 1×
Citations per year

Countries citing papers authored by Tomoh Masaki

Since Specialization
Citations

This map shows the geographic impact of Tomoh Masaki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tomoh Masaki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tomoh Masaki more than expected).

Fields of papers citing papers by Tomoh Masaki

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tomoh Masaki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tomoh Masaki. The network helps show where Tomoh Masaki may publish in the future.

Co-authorship network of co-authors of Tomoh Masaki

This figure shows the co-authorship network connecting the top 25 collaborators of Tomoh Masaki. A scholar is included among the top collaborators of Tomoh Masaki based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tomoh Masaki. Tomoh Masaki is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
#WorkIndexed citations
1 90
2 6
3 30
4 54
5 14
6 19
7 48
8 9
9 68
10 304
11 213
12 49
13 255

About Tomoh Masaki

Tomoh Masaki is a scholar working on Physiology, Biochemistry and Cardiology and Cardiovascular Medicine, having authored 13 papers that have together received 1.2k indexed citations. Recurring topics across this work include Nitric Oxide and Endothelin Effects (10 papers), Receptor Mechanisms and Signaling (5 papers) and Ion channel regulation and function (4 papers). The work is most often cited by research in Physiology (763 citations), Cardiology and Cardiovascular Medicine (495 citations) and Endocrine and Autonomic Systems (67 citations). Tomoh Masaki has collaborated with scholars based in Japan and United States. Frequent co-authors include Masashi Yanagisawa, Nobuhiro Suzuki, Chieko Kitada, Hirokazu Matsumoto, Masahiko Fujino, Y Hirata, Toshiaki Emori, T Imai, Fumiaki Marumo and Takeshi Sakurai. Their work appears in journals such as Biochemical and Biophysical Research Communications, Kidney International and Hypertension.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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