Tomáš Stopka

3.3k citations
78 papers · 2.5k indexed · h-index 28
  • Hematology top 1%
    • Acute Myeloid Leukemia Research 25
  • Genetics top 2%
    • Chronic Lymphocytic Leukemia Research 7
    • MicroRNA in disease regulation 9
    • Epigenetics and DNA Methylation 21
    • Genomics and Chromatin Dynamics 16
    • Chromatin Remodeling and Cancer 8
    • RNA modifications and cancer 8
  • Immunology top 10%
    • Erythrocyte Function and Pathophysiology 6

Tomáš Stopka

73 papers receiving 2.4k citations

Peers

Tomáš Stopka
Comparison fields: 5 of 107
  • Hematology 737
  • Genetics 413
  • Cancer Research 560
  • Molecular Biology 1.6k
  • Immunology 296
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Kinuko Ohneda Japan
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Citations per year

Countries citing papers authored by Tomáš Stopka

Since Specialization
Citations

This map shows the geographic impact of Tomáš Stopka's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tomáš Stopka with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tomáš Stopka more than expected).

Fields of papers citing papers by Tomáš Stopka

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tomáš Stopka. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tomáš Stopka. The network helps show where Tomáš Stopka may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Tomáš Stopka, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Tomáš Stopka Line = papers co-authored together Tomáš Stopka links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20243
2 20241
3 20241
4 20242
5 20233
6 20200
7 20184
8 20178
9 201643
10 201323
11 201236
12 2011116
13 2010133
14 200921
15 2007129
16 200691
17
Developmentally Regulated Tissue–specific Expression of the Mouse A–crystallin Requires Establishment of a Broad H3K9 Acetylation Domain Including the Upstream Region DCR1, Activated via FGF2 Signaling
20051
18 2005119
19 200385
20 200291

About Tomáš Stopka

Tomáš Stopka is a scholar working on Hematology, Genetics and Cancer Research, having authored 78 papers that have together received 2.5k indexed citations. Recurring topics across this work include Acute Myeloid Leukemia Research (25 papers), Epigenetics and DNA Methylation (21 papers), Genomics and Chromatin Dynamics (16 papers), MicroRNA in disease regulation (9 papers), Chromatin Remodeling and Cancer (8 papers), RNA modifications and cancer (8 papers), Chronic Lymphocytic Leukemia Research (7 papers) and Erythrocyte Function and Pathophysiology (6 papers). The work is most often cited by research in Hematology (737 citations), Genetics (413 citations) and Cancer Research (560 citations). Tomáš Stopka has collaborated with scholars based in Czechia, United States and United Kingdom. Frequent co-authors include Arthur I. Skoultchi, Pavel Burda, Jaroslav F. Prchal, Peter Laslo, Josef T. Prchal, Bruce A. Julian, Michal Mrug, Petra Bašová, E Nečas and Vít Pospíšil. Their work appears in journals such as Blood, Leukemia, International Journal of Molecular Sciences, Experimental Hematology and Cells.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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