Tomas Kavanagh

666 total citations · 1 hit paper
15 papers, 366 citations indexed

About

Tomas Kavanagh is a scholar working on Physiology, Molecular Biology and Psychiatry and Mental health. According to data from OpenAlex, Tomas Kavanagh has authored 15 papers receiving a total of 366 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Physiology, 10 papers in Molecular Biology and 3 papers in Psychiatry and Mental health. Recurrent topics in Tomas Kavanagh's work include Alzheimer's disease research and treatments (10 papers), RNA regulation and disease (3 papers) and Dementia and Cognitive Impairment Research (3 papers). Tomas Kavanagh is often cited by papers focused on Alzheimer's disease research and treatments (10 papers), RNA regulation and disease (3 papers) and Dementia and Cognitive Impairment Research (3 papers). Tomas Kavanagh collaborates with scholars based in Australia, United States and Japan. Tomas Kavanagh's co-authors include Eleanor Drummond, Thomas Wısnıewskı, Geoffrey Pires, Beatrix Ueberheide, Manor Askenazi, Glenda M. Halliday, James D. Mills, Michael Janitz, Woojin S. Kim and Bei Jun Chen and has published in prestigious journals such as Nature Communications, PLoS ONE and The FASEB Journal.

In The Last Decade

Tomas Kavanagh

13 papers receiving 364 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Compilation of reported protein changes in the brain in Alzheimer’s disease

2023 81 citations Manor Askenazi, Tomas Kavanagh et al. Nature Communications profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Tomas Kavanagh Australia	9	209	189	55	44	42	15	366
Adam Cantlon United States	6	170 0.8×	174 0.9×	66 1.2×	73 1.7×	97 2.3×	6	314
Jan Verheijen United States	6	220 1.1×	155 0.8×	27 0.5×	47 1.1×	30 0.7×	8	357
Ang Xing China	7	118 0.6×	132 0.7×	50 0.9×	57 1.3×	25 0.6×	10	321
Lenora Higginbotham United States	8	217 1.0×	241 1.3×	39 0.7×	70 1.6×	11 0.3×	14	411
Mei-Chen Liao United States	11	257 1.2×	315 1.7×	81 1.5×	118 2.7×	47 1.1×	26	540
Max Richter Germany	11	202 1.0×	159 0.8×	79 1.4×	39 0.9×	22 0.5×	14	433
Laura Beth McIntire United States	11	310 1.5×	193 1.0×	73 1.3×	70 1.6×	67 1.6×	24	544
Jiechao Zhou China	10	198 0.9×	83 0.4×	56 1.0×	54 1.2×	30 0.7×	11	343
Adeline E. Walsh United States	5	187 0.9×	152 0.8×	44 0.8×	103 2.3×	10 0.2×	6	383
Roger Willian de Lábio Brazil	12	238 1.1×	131 0.7×	28 0.5×	35 0.8×	37 0.9×	29	430

Countries citing papers authored by Tomas Kavanagh

Since Specialization

Citations

This map shows the geographic impact of Tomas Kavanagh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tomas Kavanagh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tomas Kavanagh more than expected).

Fields of papers citing papers by Tomas Kavanagh

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tomas Kavanagh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tomas Kavanagh. The network helps show where Tomas Kavanagh may publish in the future.

Co-authorship network of co-authors of Tomas Kavanagh

This figure shows the co-authorship network connecting the top 25 collaborators of Tomas Kavanagh. A scholar is included among the top collaborators of Tomas Kavanagh based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tomas Kavanagh. Tomas Kavanagh is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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15 of 15 papers shown

Kavanagh, Tomas, Evgeny Kanshin, Glenda M. Halliday, et al.. (2025). The interactome of tau phosphorylated at T217 in Alzheimer’s disease human brain tissue. Acta Neuropathologica. 149(1). 44–44.

Kavanagh, Tomas, et al.. (2025). Leveraging Proteomics to Explore the Molecular Mechanisms of Primary Tauopathies. Journal of Neurochemistry. 169(11). e70270–e70270.

Kavanagh, Tomas, et al.. (2025). Differences in the soluble and insoluble proteome between primary tauopathies. Alzheimer s & Dementia. 21(6). e70401–e70401. 1 indexed citations

Johnston, Caitlin, Tomas Kavanagh, Dominique Leitner, et al.. (2024). SMOC1 colocalizes with Alzheimer’s disease neuropathology and delays Aβ aggregation. Acta Neuropathologica. 148(1). 72–72. 10 indexed citations

Leitner, Dominique, Geoffrey Pires, Tomas Kavanagh, et al.. (2024). Similar brain proteomic signatures in Alzheimer’s disease and epilepsy. Acta Neuropathologica. 147(1). 27–27. 15 indexed citations

Leitner, Dominique, Tomas Kavanagh, Evgeny Kanshin, et al.. (2024). Differences in the cerebral amyloid angiopathy proteome in Alzheimer’s disease and mild cognitive impairment. Acta Neuropathologica. 148(1). 9–9. 7 indexed citations

Kavanagh, Tomas, et al.. (2024). hnRNP A1, hnRNP A2B1, and hnRNP K are dysregulated in tauopathies, but do not colocalize with tau pathology. Brain Pathology. 35(3). e13305–e13305. 8 indexed citations

Kavanagh, Tomas & Eleanor Drummond. (2024). Insights from a proteomic atlas of human Alzheimer’s disease brain tissue. Neural Regeneration Research. 20(3). 799–800. 1 indexed citations

Askenazi, Manor, Tomas Kavanagh, Geoffrey Pires, et al.. (2023). Compilation of reported protein changes in the brain in Alzheimer’s disease. Nature Communications. 14(1). 4466–4466. 81 indexed citations breakdown →

10.

Drummond, Eleanor, Tomas Kavanagh, Geoffrey Pires, et al.. (2022). The amyloid plaque proteome in early onset Alzheimer’s disease and Down syndrome. Acta Neuropathologica Communications. 10(1). 53–53. 75 indexed citations

11.

Kavanagh, Tomas, et al.. (2022). Tau interactome and RNA binding proteins in neurodegenerative diseases. Molecular Neurodegeneration. 17(1). 66–66. 51 indexed citations

12.

Montgomery, Magdalene K., Brenna Osborne, Amanda E. Brandon, et al.. (2019). Regulation of mitochondrial metabolism in murine skeletal muscle by the medium‐chain fatty acid receptor Gpr84. The FASEB Journal. 33(11). 12264–12276. 41 indexed citations

13.

Mills, James D., Tomas Kavanagh, Woojin S. Kim, et al.. (2015). High expression of long intervening non-coding RNA OLMALINC in the human cortical white matter is associated with regulation of oligodendrocyte maturation. Molecular Brain. 8(1). 2–2. 27 indexed citations

14.

Mills, James D., Tomas Kavanagh, Woojin S. Kim, et al.. (2013). Unique Transcriptome Patterns of the White and Grey Matter Corroborate Structural and Functional Heterogeneity in the Human Frontal Lobe. PLoS ONE. 8(10). e78480–e78480. 40 indexed citations

15.

Kavanagh, Tomas, James D. Mills, Woojin S. Kim, Glenda M. Halliday, & Michael Janitz. (2012). Pathway Analysis of the Human Brain Transcriptome in Disease. Journal of Molecular Neuroscience. 51(1). 28–36. 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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