Glenda M. Halliday
- Neurology top 0.01%
- Parkinson's Disease Mechanisms and Treatments 312
- Neurological disorders and treatments 122
- Amyotrophic Lateral Sclerosis Research 93
- Neurological diseases and metabolism 72
- Neurology top 0.01%
- Parkinson's Disease Mechanisms and Treatments 312
- Neurological disorders and treatments 122
- Amyotrophic Lateral Sclerosis Research 93
- Neurological diseases and metabolism 72
- Cellular and Molecular Neuroscience top 0.02%
- Nuclear Receptors and Signaling 55
- Neuroscience and Neuropharmacology Research 47
- Physiology top 0.02%
- Alzheimer's disease research and treatments 202
- Psychiatry and Mental health top 0.1%
- Dementia and Cognitive Impairment Research 79
- Co-authors
- Jillian J. KrilAnthony E. LangWerner PoeweJosé Á. ObesoJohn G. MorrisCharles H. AdlerM. A. HelyC. Warren Olanow
- Partner nations
- AustraliaUnited StatesUnited Kingdom
In The Last Decade
Glenda M. Halliday
641 papers receiving 50.3k citations
Hit Papers
Peers
Comparison fields: 5 of 211
- Neurology 30.2k
- Neurology 8.9k
- Cellular and Molecular Neuroscience 13.7k
- Physiology 13.4k
- Psychiatry and Mental health 5.9k
Countries citing papers authored by Glenda M. Halliday
This map shows the geographic impact of Glenda M. Halliday's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Glenda M. Halliday with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Glenda M. Halliday more than expected).
Fields of papers citing papers by Glenda M. Halliday
This network shows the impact of papers produced by Glenda M. Halliday. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Glenda M. Halliday. The network helps show where Glenda M. Halliday may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Glenda M. Halliday, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 3 | |
| 2 | 2025 | 1 | |
| 3 | 2025 | 1 | |
| 4 | 2025 | 0 | |
| 5 | 2024 | 9 | |
| 6 | 2024 | 5 | |
| 7 | 2023 | 2 | |
| 8 | 2022 | 17 | |
| 9 | 2022 | 3 | |
| 10 | 2022 | 31 | |
| 11 | 2022 | 18 | |
| 12 | 2021 | 38 | |
| 13 | 2020 | 11 | |
| 14 | 2020 | 49 | |
| 15 | Evidence for α-synuclein prions causing multiple system atrophy in humans with parkinsonismbreakdown → | 2015 | 542 |
| 16 | 2014 | 341 | |
| 17 | 2013 | 23 | |
| 18 | 2008 | 267 | |
| 19 | 2006 | 4 | |
| 20 | 2000 | 126 |
About Glenda M. Halliday
Glenda M. Halliday is a scholar working on Neurology, Neurology and Cellular and Molecular Neuroscience, having authored 661 papers that have together received 51.2k indexed citations. Recurring topics across this work include Parkinson's Disease Mechanisms and Treatments (312 papers), Alzheimer's disease research and treatments (202 papers), Neurological disorders and treatments (122 papers), Amyotrophic Lateral Sclerosis Research (93 papers), Dementia and Cognitive Impairment Research (79 papers), Neurological diseases and metabolism (72 papers), Nuclear Receptors and Signaling (55 papers) and Neuroscience and Neuropharmacology Research (47 papers). The work is most often cited by research in Neurology (30.2k citations), Neurology (8.9k citations) and Cellular and Molecular Neuroscience (13.7k citations). Glenda M. Halliday has collaborated with scholars based in Australia, United States and United Kingdom. Frequent co-authors include Jillian J. Kril, Anthony E. Lang, Werner Poewe, José Á. Obeso, John G. Morris, Charles H. Adler, M. A. Hely, C. Warren Olanow, Klaus Seppi and Caroline M. Tanner. Their work appears in journals such as Movement Disorders, Acta Neuropathologica, Brain, Neurology and Neurobiology of Aging.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.