Tom Were

2.0k total citations
58 papers, 1.4k citations indexed

About

Tom Were is a scholar working on Public Health, Environmental and Occupational Health, Immunology and Epidemiology. According to data from OpenAlex, Tom Were has authored 58 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 23 papers in Public Health, Environmental and Occupational Health, 19 papers in Immunology and 18 papers in Epidemiology. Recurrent topics in Tom Were's work include Malaria Research and Control (22 papers), Complement system in diseases (12 papers) and HIV Research and Treatment (8 papers). Tom Were is often cited by papers focused on Malaria Research and Control (22 papers), Complement system in diseases (12 papers) and HIV Research and Treatment (8 papers). Tom Were collaborates with scholars based in Kenya, United States and United Kingdom. Tom Were's co-authors include Douglas J. Perkins, John Michael Ong’echa, Collins Ouma, James B. Hittner, Gregory C. Davenport, John Vulule, Prakasha Kempaiah, Christopher Keller, R.O Otieno and Robert E. Ferrell and has published in prestigious journals such as SHILAP Revista de lepidopterología, Journal of Clinical Microbiology and The Journal of Infectious Diseases.

In The Last Decade

Tom Were

52 papers receiving 1.4k citations

Author Peers

Peers are selected by citation overlap in the author's most active subfields. citations · hero ref

Author Last Decade Papers Cites
Tom Were 880 558 204 192 185 58 1.4k
Yacouba Cissoko 832 0.9× 253 0.5× 175 0.9× 152 0.8× 186 1.0× 47 1.2k
Oscar Kai 1.3k 1.5× 674 1.2× 132 0.6× 185 1.0× 118 0.6× 29 1.6k
Doris Luckner 824 0.9× 408 0.7× 126 0.6× 196 1.0× 99 0.5× 25 1.3k
Lígia Antunes Gonçalves 673 0.8× 300 0.5× 168 0.8× 300 1.6× 217 1.2× 31 1.3k
Alloys S. S. Orago 840 1.0× 465 0.8× 187 0.9× 233 1.2× 150 0.8× 55 1.4k
Arlene E. Dent 873 1.0× 558 1.0× 118 0.6× 217 1.1× 160 0.9× 61 1.5k
Gehad ElGhazali 694 0.8× 696 1.2× 190 0.9× 168 0.9× 176 1.0× 82 1.6k
Léopold G. Lehman 932 1.1× 345 0.6× 119 0.6× 207 1.1× 104 0.6× 37 1.2k
John Michael Ong’echa 1.2k 1.4× 808 1.4× 236 1.2× 267 1.4× 196 1.1× 74 2.1k
Amagana Dolo 1.3k 1.5× 504 0.9× 186 0.9× 309 1.6× 120 0.6× 67 1.8k

Countries citing papers authored by Tom Were

Since Specialization
Citations

This map shows the geographic impact of Tom Were's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tom Were with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tom Were more than expected).

Fields of papers citing papers by Tom Were

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tom Were. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tom Were. The network helps show where Tom Were may publish in the future.

Co-authorship network of co-authors of Tom Were

This figure shows the co-authorship network connecting the top 25 collaborators of Tom Were. A scholar is included among the top collaborators of Tom Were based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tom Were. Tom Were is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Were, Tom, et al.. (2025). Giardia duodenalis in Human Health: A Comprehensive Review of its Epidemiology, Transmission, Pathophysiology, Diagnostics, and Control Strategies. Afro-Egyptian Journal of Infectious and Endemic Diseases. 0(0). 0–0. 2 indexed citations
2.
Ngari, Moses M., et al.. (2024). Nutritional status of people who inject drugs in Coastal Kenya: a cross-sectional study. BMC Nutrition. 10(1). 55–55.
3.
Were, Tom, et al.. (2024). Assemblages and Subassemblages of Giardia duodenalis in Rural Western, Kenya: Association with Sources, Signs, and Symptoms. Journal of Parasitology Research. 2024. 1–8. 4 indexed citations
5.
Were, Tom, et al.. (2023). Hepatic Function and its Association with Clinical Outcomes in Non-Adherent HIV-1 Adults. Afro-Egyptian Journal of Infectious and Endemic Diseases. 0(0). 0–0. 1 indexed citations
7.
Were, Tom, et al.. (2022). The burden and types of anaemia among HIV infected, ART-naive injection substance users in Kenya. African Health Sciences. 22(1). 431–42. 2 indexed citations
8.
9.
Were, Tom, et al.. (2021). Cytokine profiles in highly active antiretroviral treatment non-adherent, adherent and naive HIV-1 infected patients in Western Kenya. African Health Sciences. 21(4). 1584–92. 7 indexed citations
11.
Ahmed, Aabid, et al.. (2015). Serum adiponectin in HIV-1 and hepatitis C virus mono- and co-infected Kenyan injection drug users. Endocrine Connections. 4(4). 223–232. 5 indexed citations
12.
Nyamache, Anthony Kebira, et al.. (2015). Hepatitis B virus sero-profiles and genotypes in HIV-1 infected and uninfected injection and Non-injection drug users from coastal Kenya. BMC Infectious Diseases. 15(1). 299–299. 21 indexed citations
13.
Kempaiah, Prakasha, Samuel B. Anyona, Evans Raballah, et al.. (2012). Reduced interferon (IFN)-α conditioned by IFNA2 (−173) and IFNA8 (−884) haplotypes is associated with enhanced susceptibility to severe malarial anemia and longitudinal all-cause mortality. Human Genetics. 131(8). 1375–1391. 14 indexed citations
14.
Ayodo, George, Tom Were, Samuel B. Anyona, et al.. (2012). Factors associated with non-adherence to Artemisinin-based combination therapy (ACT) to malaria in a rural population from holoendemic region of western Kenya. BMC Infectious Diseases. 12(1). 143–143. 36 indexed citations
15.
Ong’echa, John Michael, Evans Raballah, Prakasha Kempaiah, et al.. (2011). Polymorphic variability in the 3' untranslated region (UTR) of IL12B is associated with susceptibility to severe anaemia in Kenyan children with acute Plasmodium falciparum malaria. BMC Genetics. 12(1). 69–69. 13 indexed citations
16.
Novelli, Enrico M., James B. Hittner, Gregory C. Davenport, et al.. (2010). Clinical predictors of severe malarial anaemia in a holoendemic Plasmodium falciparum transmission area. British Journal of Haematology. 149(5). 711–721. 34 indexed citations
17.
Ouma, Collins, Tom Were, Yuvadee Mahakunkijcharoen, et al.. (2010). Haplotypes of IL12B promoter polymorphisms condition susceptibility to severe malaria and functional changes in cytokine levels in Thai adults. Immunogenetics. 62(6). 345–356. 24 indexed citations
18.
Were, Tom, Gregory C. Davenport, James B. Hittner, et al.. (2009). Naturally acquired hemozoin by monocytes promotes suppression of RANTES in children with malarial anemia through an IL-10-dependent mechanism. Microbes and Infection. 11(8-9). 811–819. 23 indexed citations
19.
Ouma, Collins, Gregory C. Davenport, Tom Were, et al.. (2008). Haplotypes of IL-10 promoter variants are associated with susceptibility to severe malarial anemia and functional changes in IL-10 production. Human Genetics. 124(5). 515–524. 68 indexed citations
20.
Otieno, R.O, Collins Ouma, John Michael Ong’echa, et al.. (2006). Increased severe anemia in HIV-1-exposed and HIV-1-positive infants and children during acute malaria. AIDS. 20(2). 275–280. 110 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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