Tinisha McDonald

2.8k total citations
30 papers, 1.7k citations indexed

About

Tinisha McDonald is a scholar working on Hematology, Molecular Biology and Immunology. According to data from OpenAlex, Tinisha McDonald has authored 30 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Hematology, 12 papers in Molecular Biology and 10 papers in Immunology. Recurrent topics in Tinisha McDonald's work include Chronic Myeloid Leukemia Treatments (9 papers), Chronic Lymphocytic Leukemia Research (7 papers) and Acute Myeloid Leukemia Research (6 papers). Tinisha McDonald is often cited by papers focused on Chronic Myeloid Leukemia Treatments (9 papers), Chronic Lymphocytic Leukemia Research (7 papers) and Acute Myeloid Leukemia Research (6 papers). Tinisha McDonald collaborates with scholars based in United States, United Kingdom and China. Tinisha McDonald's co-authors include Ravi Bhatia, Tessa L. Holyoake, Stephen J. Forman, Allen Lin, Ling Li, YinWei Ho, Su Chu, Zhiqiang Wang, Liang Li and Lisheng Wang and has published in prestigious journals such as SHILAP Revista de lepidopterología, Blood and Gastroenterology.

In The Last Decade

Tinisha McDonald

28 papers receiving 1.7k citations

Peers

Tinisha McDonald
Comparison fields: 5 of 73
  • Hematology 868
  • Molecular Biology 753
  • Oncology 543
  • Genetics 372
  • Immunology 361
Armando G. Poeppl Canada
S. Tiong Ong United States
Todd M. Zimmerman United States
Denise Toscani Italy
Letha A. Phillips United States
Marina Bolzoni Italy
Denis M. Schewe Germany
Christina Matheny United States
Bryan Ciccarelli United States
Michael Hundemer Germany
Armando G. Poeppl Canada View profile →
Citations per field, relative to Tinisha McDonald
Tinisha McDonald · 1×
Citations per year, relative to Tinisha McDonald
Tinisha McDonald · 1×

Countries citing papers authored by Tinisha McDonald

Since Specialization
Citations

This map shows the geographic impact of Tinisha McDonald's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tinisha McDonald with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tinisha McDonald more than expected).

Fields of papers citing papers by Tinisha McDonald

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tinisha McDonald. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tinisha McDonald. The network helps show where Tinisha McDonald may publish in the future.

Co-authorship network of co-authors of Tinisha McDonald

This figure shows the co-authorship network connecting the top 25 collaborators of Tinisha McDonald. A scholar is included among the top collaborators of Tinisha McDonald based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tinisha McDonald. Tinisha McDonald is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
# Title Journal Authors Indexed citations
1 Activated natural killer cells predict poor clinical prognosis in high-risk B- and T-cell acute lymphoblastic leukemia Blood Caroline Duault, Anil Kumar et al. 39
2 Acute Myeloid Leukemia Cell-Intrinsic PD-1 Functions Promoting Leukemia Development By Recruiting SHP2 Blood Le Xuan Truong Nguyen, Fenglin Li et al. 2
3 Activated Natural Killer Cells Are Associated with Poor Clinical Prognosis in High-Risk B- and T- Cell Acute Lymphoblastic Leukemia Blood Caroline Duault, Anil Kumar et al. 1
4 Acute myeloid leukemia transforms the bone marrow niche into a leukemia-permissive microenvironment through exosome secretion Leukemia Bijender Kumar, Mayra García et al. 328
5 Acute Myeloid Leukemia-Derived Exosomes Transform Bone Marrow Niche into Leukemic Niche. Blood Bijender Kumar, Mayra García et al. 3
6 The Src and c-Kit kinase inhibitor dasatinib enhances p53-mediated targeting of human acute myeloid leukemia stem cells by chemotherapeutic agents Blood Cédric Dos Santos, Tinisha McDonald et al. 79
7 T cells expressing CD123-specific chimeric antigen receptors exhibit specific cytolytic effector functions and antitumor effects against human acute myeloid leukemia Blood Armen Mardiros, Cédric Dos Santos et al. 300
8 Pharmacological Inhibition Of The SIRT1 Deacetylase With The Small Molecule Inhibitor Tenovin-6 Enhances Ablation Of FLT3-ITD+ LSC In Combination With TKI Treatment Blood Ling Li, YinWei Ho et al. 2
9 Leukemia-Derived Exosomes Reorganize Bone Marrow Microenvironment In AML Blood Bijender Kumar, Lihong Weng et al. 2
10 Activation of p53 by SIRT1 Inhibition Enhances Elimination of CML Leukemia Stem Cells in Combination with Imatinib Cancer Cell Ling Li, Lisheng Wang et al. 327
11 CD123-Specific Chimeric Antigen Receptor Redirected T Cells Exhibit Potent Cytolytic Activity and Multiple Effector Functions Against Acute Myeloid Leukemia without Altering Normal Hematopoietic Colony Formation in Vitro Blood Armen Mardiros, Cédric Dos Santos et al. 1
12 Altered Hematopoietic Cell Gene Expression Precedes Development of Therapy-Related Myelodysplasia/Acute Myeloid Leukemia and Identifies Patients at Risk Cancer Cell Liang Li, Min Li et al. 65
13 Pharmacological Inhibition of the Stress-Related Deacetylase SIRT1 Enhances Eradication of CML stem Cells Blood Ling Li, Lisheng Wang et al. 0
14 A critical role for SHP2 in STAT5 activation and growth factor–mediated proliferation, survival, and differentiation of human CD34+ cells Blood Liang Li, Hardik Modi et al. 39
15 Persistence of Leukemia Stem Cells in Chronic Myelogenous Leukemia Patients in Complete Cytogenetic Remission on Imatinib Treatment for 5 Years Blood Su Chu, Allen Lin et al. 8
16 Role of BCR/ABL gene-expression levels in determining the phenotype and imatinib sensitivity of transformed human hematopoietic cells Blood Hardik Modi, Tinisha McDonald et al. 49
17 Expression of DLK/PREF1 Results in Inhibition of Human Myeloid Cell Differentiation and Proliferation through Distinct Molecular Mechanisms. Blood Liang Li, Tinisha McDonald et al. 5
18 Feasibility and Efficacy of Mobilization of BCR/ABL- PBSC in CML Patients Receiving Imatinib. Blood Ravi Bhatia, David S. Snyder et al. 2
19 Immune escape mechanisms of childhood ALL and a potential countering role for DC-like leukemia cells Cytotherapy Ping Han, Colin Story et al. 10
20 PYY and NPY stimulate isolated perfused canine ileal contractions by inhibiting VIP not no release Gastroenterology J.E.T. Fox‐Threlkeld, E.E. Daniel et al. 1

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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