Ting‐Fen Tsai
Impact in
- Aging top 2%
- Cancer Research top 2%
- MicroRNA in disease regulation
- Cancer-related molecular mechanisms research
Papers in
- Aging 12
- Genetics, Aging, and Longevity in Model Organisms 12
- Co-authors
- Yi‐Fan ChenArthur L. BeaudetCheng-Heng KaoMing‐Ta HsuJan BresslerChih‐Hao WangYong-hui JiangShih-Feng Tsai
- Journals
- Hepatology (6 papers)International Journal of Molecular Sciences (5 papers)Aging Cell (4 papers)Autophagy (3 papers)genesis (3 papers)
- Partner nations
- TaiwanUnited StatesChina
In The Last Decade
Ting‐Fen Tsai
106 papers receiving 4.6k citations
Hit Papers
Peers
Comparison fields: 5 of 125
- Aging 122
- Cancer Research 977
- Hepatology 454
- Molecular Biology 3.0k
- Cell Biology 612
Countries citing papers authored by Ting‐Fen Tsai
This map shows the geographic impact of Ting‐Fen Tsai's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ting‐Fen Tsai with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ting‐Fen Tsai more than expected).
Fields of papers citing papers by Ting‐Fen Tsai
This network shows the impact of papers produced by Ting‐Fen Tsai. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ting‐Fen Tsai. The network helps show where Ting‐Fen Tsai may publish in the future.
Co-authors
The 25 scholars most cited alongside Ting‐Fen Tsai, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 0 | |
| 2 | 2025 | 0 | |
| 3 | 2025 | 3 | |
| 4 | 2025 | 8 | |
| 5 | 2025 | 0 | |
| 6 | 2025 | 1 | |
| 7 | 2024 | 3 | |
| 8 | 2024 | 1 | |
| 9 | 2023 | 4 | |
| 10 | 2023 | 2 | |
| 11 | 2021 | 15 | |
| 12 | 2021 | 17 | |
| 13 | 2020 | 11 | |
| 14 | 2017 | 60 | |
| 15 | Autophagy Regulates Human Hepatocellular Carcinoma Tumorigenesis through Selective Degradation of Cyclin D1 | 2016 | 2 |
| 16 | 2015 | 23 | |
| 17 | 2009 | 28 | |
| 18 | 2008 | 73 | |
| 19 | 2007 | 76 | |
| 20 | 1988 | 43 |
About Ting‐Fen Tsai
Ting‐Fen Tsai is a scholar working on Aging, Clinical Biochemistry, Molecular Biology, Cell Biology and Hepatology, having authored 110 papers that have together received 4.6k indexed citations. Recurring topics across this work include Mitochondrial Function and Pathology (26 papers), Epigenetics and DNA Methylation (16 papers), Endoplasmic Reticulum Stress and Disease (15 papers), Genetics, Aging, and Longevity in Model Organisms (12 papers), Liver Disease Diagnosis and Treatment (10 papers), Autophagy in Disease and Therapy (9 papers), Hepatitis B Virus Studies (8 papers) and Genetics and Neurodevelopmental Disorders (8 papers). The work is most often cited by research in Aging (122 citations), Cancer Research (977 citations), Hepatology (454 citations), Molecular Biology (3.0k citations) and Cell Biology (612 citations). Ting‐Fen Tsai has collaborated with scholars based in Taiwan, United States and China. Frequent co-authors include Yi‐Fan Chen, Arthur L. Beaudet, Cheng-Heng Kao, Ming‐Ta Hsu, Jan Bressler, Chih‐Hao Wang, Yong-hui Jiang, Shih-Feng Tsai, Zhao‐Qing Shen and Yau‐Huei Wei. Their work appears in journals such as Hepatology, International Journal of Molecular Sciences, Aging Cell, Autophagy and genesis.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.