Tibor Gyuris

565 citations
14 papers · 461 indexed · h-index 10
Topics
Protein Kinase Regulation and GTPase Signaling (2 papers)Peptidase Inhibition and Analysis (2 papers)Genetics and Neurodevelopmental Disorders (2 papers)

In The Last Decade

Tibor Gyuris

14 papers receiving 453 citations

Peers

Tibor Gyuris
Comparison fields: 5 of 71
  • Molecular Biology 327
  • Oncology 80
  • Neurology 78
  • Cancer Research 72
  • Physiology 50
Replace Cécile Couriaud with:
Cécile Couriaud France
Shuo Huang United States
А. Г. Бабич Germany
Siobhán Q. Gregg United States
Edward A. Felinski United States
Yun‐Zheng Le United States
Natalia Martín-Martín Spain
Janet E. Holley United Kingdom
Francis Amara Canada
Enric Poch Spain
Tibor Gyuris relative to Cécile Couriaud France Cécile Couriaud's profile →
Citations per field
00.5×4.7×
Cécile Couriaud · 1×
Citations per year

Countries citing papers authored by Tibor Gyuris

Since Specialization
Citations

This map shows the geographic impact of Tibor Gyuris's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tibor Gyuris with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tibor Gyuris more than expected).

Fields of papers citing papers by Tibor Gyuris

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tibor Gyuris. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tibor Gyuris. The network helps show where Tibor Gyuris may publish in the future.

Co-authorship network of co-authors of Tibor Gyuris

This figure shows the co-authorship network connecting the top 25 collaborators of Tibor Gyuris. A scholar is included among the top collaborators of Tibor Gyuris based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tibor Gyuris. Tibor Gyuris is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

14 of 14 papers shown
#WorkIndexed citations
1 1
2 43
3 12
4 78
5 44
6 96
7 4
8
AMG 479, a fully human anti IGF-1 receptor monoclonal antibody, is efficacious against Ewing’s sarcoma and osteosarcoma xenografts
6
9 33
10 17
11 9
12 4
13 21
14 93

About Tibor Gyuris

Tibor Gyuris is a scholar working on Immunology and Allergy, Clinical Biochemistry and Nephrology, having authored 14 papers that have together received 461 indexed citations. Recurring topics across this work include Protein Kinase Regulation and GTPase Signaling (2 papers), Peptidase Inhibition and Analysis (2 papers) and Genetics and Neurodevelopmental Disorders (2 papers). The work is most often cited by research in Neurology (78 citations), Cancer Research (72 citations) and Molecular Biology (327 citations). Tibor Gyuris has collaborated with scholars based in United States, Hungary and Belgium. Frequent co-authors include T Torchia, D. Serban, Mark Rogers, Michael Scott, Stanley B. Prusiner, Miklós Péterfy, Yang Li, Hui Tian, Jennifer Weiszmann and Hélène Baribault. Their work appears in journals such as Genes & Development, Blood and The Journal of Immunology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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