Thomas J. Lindell
- Molecular Biology top 10%
- Genetics top 10%
- Plant Science
- Cell Biology top 10%
- Immunology
- Co-authors
- William J. RutterFanyela WeinbergPaul W. MorrisRobert G. RoederWilliam ThornburgMark R. HausslerJoseph E. ZerwekhEarle Stellwagen
- Topics
- RNA and protein synthesis mechanisms (7 papers)DNA and Nucleic Acid Chemistry (5 papers)Enzyme function and inhibition (4 papers)
- Cited by
- AgingMolecular BiologyCell Biology
- Partner nations
- United States
In The Last Decade
Thomas J. Lindell
29 papers receiving 1.4k citations
Hit Papers
Peers
Comparison fields: 5 of 108
- Molecular Biology 1.1k
- Genetics 218
- Plant Science 162
- Cell Biology 130
- Immunology 120
Countries citing papers authored by Thomas J. Lindell
This map shows the geographic impact of Thomas J. Lindell's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas J. Lindell with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas J. Lindell more than expected).
Fields of papers citing papers by Thomas J. Lindell
This network shows the impact of papers produced by Thomas J. Lindell. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas J. Lindell. The network helps show where Thomas J. Lindell may publish in the future.
Co-authorship network of co-authors of Thomas J. Lindell
This figure shows the co-authorship network connecting the top 25 collaborators of Thomas J. Lindell. A scholar is included among the top collaborators of Thomas J. Lindell based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas J. Lindell. Thomas J. Lindell is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 14 | |
| 2 | 6 | |
| 3 | 5 | |
| 4 | 2 | |
| 5 | 7 | |
| 6 | 19 | |
| 7 | 18 | |
| 8 | 3 | |
| 9 | 32 | |
| 10 | 96 | |
| 11 | 7 | |
| 12 | 3 | |
| 13 | 86 | |
| 14 | 55 | |
| 15 | 7 | |
| 16 | Alpha-amanitin inhibition of mouse brain form II ribonucleic acid polymerase and passive avoidance retention. | 13 |
| 17 | 4 | |
| 18 | 174 | |
| 19 | Specific Inhibition of Nuclear RNA Polymerase II by α-Amanitinbreakdown → | 762 |
| 20 | 53 |
About Thomas J. Lindell
Thomas J. Lindell is a scholar working on Aging, Molecular Biology and Pharmacology, having authored 30 papers that have together received 1.6k indexed citations. Recurring topics across this work include RNA and protein synthesis mechanisms (7 papers), DNA and Nucleic Acid Chemistry (5 papers) and Enzyme function and inhibition (4 papers). The work is most often cited by research in Aging (33 citations), Molecular Biology (1.1k citations) and Cell Biology (130 citations). Thomas J. Lindell has collaborated with scholars based in United States. Frequent co-authors include William J. Rutter, Fanyela Weinberg, Paul W. Morris, Robert G. Roeder, William Thornburg, Mark R. Haussler, Joseph E. Zerwekh, Earle Stellwagen, Robert F. Weaver and S. P. Blatti. Their work appears in journals such as Nature, Science and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.