Thomas G. Moens

2.0k total citations · 1 hit paper
13 papers, 975 citations indexed

About

Thomas G. Moens is a scholar working on Neurology, Molecular Biology and Genetics. According to data from OpenAlex, Thomas G. Moens has authored 13 papers receiving a total of 975 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Neurology, 6 papers in Molecular Biology and 6 papers in Genetics. Recurrent topics in Thomas G. Moens's work include Amyotrophic Lateral Sclerosis Research (10 papers), Neurogenetic and Muscular Disorders Research (6 papers) and Parkinson's Disease Mechanisms and Treatments (3 papers). Thomas G. Moens is often cited by papers focused on Amyotrophic Lateral Sclerosis Research (10 papers), Neurogenetic and Muscular Disorders Research (6 papers) and Parkinson's Disease Mechanisms and Treatments (3 papers). Thomas G. Moens collaborates with scholars based in United Kingdom, United States and Belgium. Thomas G. Moens's co-authors include Adrian M. Isaacs, Linda Partridge, Teresa Niccoli, Charlotte Ridler, Pietro Fratta, Sarah Mizielinska, Sebastian Grönke, Stuart Pickering‐Brown, Melissa Cabecinha and Elizabeth Fisher and has published in prestigious journals such as Science, The Lancet and Brain.

In The Last Decade

Thomas G. Moens

13 papers receiving 970 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

C9orf72 repeat expansions cause neurodegeneration in Drosophila through arginine-rich proteins

2014 510 citations Sarah Mizielinska, Sebastian Grönke et al. Science profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Thomas G. Moens United Kingdom	7	593	497	339	218	208	13	975
Lillian M. Daughrity United States	9	740 1.2×	564 1.1×	432 1.3×	223 1.0×	194 0.9×	12	1.1k
Martin H. Schludi Germany	7	770 1.3×	498 1.0×	419 1.2×	185 0.8×	214 1.0×	7	972
Anna M. Blokhuis Netherlands	7	655 1.1×	451 0.9×	412 1.2×	152 0.7×	151 0.7×	7	929
Jeannie Chew United States	8	615 1.0×	491 1.0×	342 1.0×	186 0.9×	158 0.8×	8	894
Sharon Carmona United States	4	652 1.1×	512 1.0×	422 1.2×	153 0.7×	221 1.1×	4	977
Cheryl Leystra‐Lantz Canada	17	888 1.5×	592 1.2×	458 1.4×	245 1.1×	154 0.7×	24	1.2k
Julien Couthouis United States	16	418 0.7×	595 1.2×	175 0.5×	217 1.0×	186 0.9×	22	1.1k
Jacqueline Dols Germany	8	408 0.7×	445 0.9×	236 0.7×	200 0.9×	165 0.8×	8	788
Nailah Siddique United States	14	525 0.9×	383 0.8×	295 0.9×	152 0.7×	199 1.0×	20	938
Caroline Stetler United States	12	764 1.3×	647 1.3×	332 1.0×	469 2.2×	192 0.9×	12	1.3k

Countries citing papers authored by Thomas G. Moens

Since Specialization

Citations

This map shows the geographic impact of Thomas G. Moens's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas G. Moens with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas G. Moens more than expected).

Fields of papers citing papers by Thomas G. Moens

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas G. Moens. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas G. Moens. The network helps show where Thomas G. Moens may publish in the future.

Co-authorship network of co-authors of Thomas G. Moens

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas G. Moens. A scholar is included among the top collaborators of Thomas G. Moens based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas G. Moens. Thomas G. Moens is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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13 of 13 papers shown

Moens, Thomas G., Sandrine Da Cruz, Manuela Neumann, et al.. (2025). Amyotrophic lateral sclerosis caused by FUS mutations: advances with broad implications. The Lancet Neurology. 24(2). 166–178. 11 indexed citations

Scheveneels, Wendy, Katarina Stoklund Dittlau, Arun Pal, et al.. (2024). PP2A and GSK3 act as modifiers of FUS-ALS by modulating mitochondrial transport. Acta Neuropathologica. 147(1). 41–41. 5 indexed citations

Decker, Mathias De, Pavol Zelina, Thomas G. Moens, et al.. (2024). C21ORF2 mutations point towards primary cilia dysfunction in amyotrophic lateral sclerosis. Brain. 148(3). 803–816. 5 indexed citations

Masrori, Pegah, Simona Ospitalieri, Karin Forsberg, et al.. (2022). Respiratory onset of amyotrophic lateral sclerosis in a pregnant woman with a novel SOD1 mutation. European Journal of Neurology. 29(4). 1279–1283. 3 indexed citations

Kedersha, Nancy, Natalia Rivkin, Yehuda M. Danino, et al.. (2020). Spatio-Temporal Proteomic Analysis of Stress Granule Disassembly Using APEX Reveals Regulation by SUMOylation and Links to ALS Pathogenesis. SSRN Electronic Journal. 2 indexed citations

Moens, Thomas G., Teresa Niccoli, Katherine Wilson, et al.. (2019). C9orf72 arginine-rich dipeptide proteins interact with ribosomal proteins in vivo to induce a toxic translational arrest that is rescued by eIF1A. Acta Neuropathologica. 137(3). 487–500. 83 indexed citations

Moens, Thomas G., Sarah Mizielinska, Teresa Niccoli, et al.. (2018). Sense and antisense RNA are not toxic in Drosophila models of C9orf72-associated ALS/FTD. Acta Neuropathologica. 135(3). 445–457. 47 indexed citations

Moens, Thomas G., Linda Partridge, & Adrian M. Isaacs. (2017). Genetic models of C9orf72: what is toxic?. Current Opinion in Genetics & Development. 44. 92–101. 42 indexed citations

Balendra, Rubika, Sarah Mizielinska, Thomas G. Moens, et al.. (2016). Molecular mechanisms and therapeutic strategies in amyotrophic lateral sclerosis caused by C9orf72 mutations. The Lancet. 387. S13–S13. 1 indexed citations

10.

Matarín, Mar, Derviş A. Salih, Marina V. Yasvoina, et al.. (2015). A Genome-wide Gene-Expression Analysis and Database in Transgenic Mice during Development of Amyloid or Tau Pathology. Cell Reports. 10(4). 633–644. 193 indexed citations

11.

Mizielinska, Sarah, Sebastian Grönke, Teresa Niccoli, et al.. (2014). C9orf72 repeat expansions cause neurodegeneration in Drosophila through arginine-rich proteins. Science. 345(6201). 1192–1194. 510 indexed citations breakdown →

12.

Hallett, Penelope J., Thomas G. Moens, Gaynor A. Smith, et al.. (2014). Enhanced ubiquitin-dependent degradation by Nedd4 protects against α-synuclein accumulation and toxicity in animal models of Parkinson's disease. Neurobiology of Disease. 64. 79–87. 70 indexed citations

13.

Messiaen, Ludwine, Georges De Bruyne, Erwin R. Boghaert, et al.. (1991). Spontaneous acquisition of tumorigenicity and invasiveness by mouse lens explant cells during culture in vitro. In Vitro Cellular & Developmental Biology - Animal. 27(5). 369–380. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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