Thomas E. Lloyd

10.4k total citations · 1 hit paper
92 papers, 4.9k citations indexed

About

Thomas E. Lloyd is a scholar working on Molecular Biology, Epidemiology and Neurology. According to data from OpenAlex, Thomas E. Lloyd has authored 92 papers receiving a total of 4.9k indexed citations (citations by other indexed papers that have themselves been cited), including 44 papers in Molecular Biology, 42 papers in Epidemiology and 30 papers in Neurology. Recurrent topics in Thomas E. Lloyd's work include Inflammatory Myopathies and Dermatomyositis (38 papers), Muscle Physiology and Disorders (19 papers) and Amyotrophic Lateral Sclerosis Research (19 papers). Thomas E. Lloyd is often cited by papers focused on Inflammatory Myopathies and Dermatomyositis (38 papers), Muscle Physiology and Disorders (19 papers) and Amyotrophic Lateral Sclerosis Research (19 papers). Thomas E. Lloyd collaborates with scholars based in United States, Spain and United Kingdom. Thomas E. Lloyd's co-authors include Hugo J. Bellen, Andrew L. Mammen, Lisa Christopher‐Stine, Yi Zhou, J. Paul Taylor, Iago Pinal‐Fernandez, Giuseppa Pennetta, Elaine Seto, Katherine Pak and Julie J. Paik and has published in prestigious journals such as Nature, Cell and Journal of Clinical Investigation.

In The Last Decade

Thomas E. Lloyd

91 papers receiving 4.8k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Stress Granule Assembly Disrupts Nucleocytoplasmic Transport

2018 279 citations Ke Zhang, Kai Ruan et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Thomas E. Lloyd United States	37	2.4k	1.6k	1.3k	1.1k	1.0k	92	4.9k
Conrad C. Weihl United States	36	2.4k 1.0×	1.0k 0.7×	1.1k 0.9×	1.0k 1.0×	736 0.7×	100	4.0k
Brian Harding United Kingdom	46	2.8k 1.2×	959 0.6×	309 0.2×	806 0.8×	909 0.9×	162	6.9k
Eduard Gallardo Spain	36	2.2k 0.9×	443 0.3×	435 0.3×	1.7k 1.6×	1.1k 1.1×	133	4.6k
Murat Günel United States	43	2.5k 1.0×	491 0.3×	482 0.4×	2.6k 2.4×	563 0.5×	135	6.6k
Hirotomo Saitsu Japan	43	4.2k 1.7×	675 0.4×	721 0.6×	276 0.3×	849 0.8×	430	7.7k
Haluk Topaloğlu Türkiye	49	5.5k 2.3×	353 0.2×	1.5k 1.2×	1.2k 1.1×	1.8k 1.8×	261	8.2k
Felipe A. Court Chile	42	3.2k 1.3×	1.1k 0.7×	1.5k 1.2×	1.1k 1.1×	2.4k 2.3×	88	6.3k
William F. Simonds United States	45	4.0k 1.6×	1.0k 0.7×	675 0.5×	393 0.4×	1.3k 1.3×	138	6.5k
Wolfgang Roggendorf Germany	38	1.5k 0.6×	457 0.3×	462 0.4×	1.2k 1.1×	702 0.7×	109	4.7k
Sara Mole United Kingdom	41	3.0k 1.2×	1.1k 0.7×	2.5k 1.9×	722 0.7×	432 0.4×	147	7.2k

Countries citing papers authored by Thomas E. Lloyd

Since Specialization

Citations

This map shows the geographic impact of Thomas E. Lloyd's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas E. Lloyd with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas E. Lloyd more than expected).

Fields of papers citing papers by Thomas E. Lloyd

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas E. Lloyd. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas E. Lloyd. The network helps show where Thomas E. Lloyd may publish in the future.

Co-authorship network of co-authors of Thomas E. Lloyd

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas E. Lloyd. A scholar is included among the top collaborators of Thomas E. Lloyd based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas E. Lloyd. Thomas E. Lloyd is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Montagne, Janelle M., et al.. (2025). Characterizing local antibody responses in the muscle of inclusion body myositis patients. Journal of Autoimmunity. 154. 103437–103437. 1 indexed citations

Maguire, Sarah, Joel J. Credle, Boyoung Cha, et al.. (2025). Dual probe ligation in situ hybridization with rolling-circle amplification for high-plex spatial transcriptomics. Biochemistry and Biophysics Reports. 43. 102207–102207.

Wischnewski, Sven, et al.. (2025). Emerging mechanisms and therapeutics in inflammatory muscle diseases. Trends in Pharmacological Sciences. 46(3). 249–263. 4 indexed citations

Ikenaga, Chiseko, Katherine E. Irwin, Irika R. Sinha, et al.. (2025). Loss of TDP‐43 Splicing Repression Occurs in Myonuclei of Inclusion Body Myositis Patients. Annals of Neurology. 97(4). 629–641. 1 indexed citations

Lynch, Eileen M., Sara K. Pittman, Jil Daw, et al.. (2024). Seeding-competent TDP-43 persists in human patient and mouse muscle. Science Translational Medicine. 16(775). eadp5730–eadp5730. 6 indexed citations

Sahana, T. G., et al.. (2023). c-Jun N-Terminal Kinase Promotes Stress Granule Assembly and Neurodegeneration in C9orf72-Mediated ALS and FTD. Journal of Neuroscience. 43(17). 3186–3197. 9 indexed citations

Britson, Kyla A., Jonathan P. Ling, Kerstin E. Braunstein, et al.. (2022). Loss of TDP-43 function and rimmed vacuoles persist after T cell depletion in a xenograft model of sporadic inclusion body myositis. Science Translational Medicine. 14(628). eabi9196–eabi9196. 34 indexed citations

Sun, Congshan, Suraj Kannan, In Young Choi, et al.. (2022). Human pluripotent stem cell-derived myogenic progenitors undergo maturation to quiescent satellite cells upon engraftment. Cell stem cell. 29(4). 610–619.e5. 17 indexed citations

Casal-Domínguez, María, Iago Pinal‐Fernandez, Katherine Pak, et al.. (2021). Performance of the 2017 European Alliance of Associations for Rheumatology/American College of Rheumatology Classification Criteria for Idiopathic Inflammatory Myopathies in Patients With Myositis‐Specific Autoantibodies. Arthritis & Rheumatology. 74(3). 508–517. 29 indexed citations

10.

Pinal‐Fernandez, Iago, Katherine Pak, Albert Gil‐Vila, et al.. (2021). Anti‐Cortactin Autoantibodies Are Associated With Key Clinical Features in Adult Myositis But Are Rarely Present in Juvenile Myositis. Arthritis & Rheumatology. 74(2). 358–364. 6 indexed citations

11.

McCray, Brett A., Jeremy M. Sullivan, William H. Aisenberg, et al.. (2021). Neuropathy-causing TRPV4 mutations disrupt TRPV4-RhoA interactions and impair neurite extension. Nature Communications. 12(1). 1444–1444. 31 indexed citations

12.

Ho, Lap, Danyue Zhao, Kenjiro Ono, et al.. (2018). Heterogeneity in gut microbiota drive polyphenol metabolism that influences α-synuclein misfolding and toxicity. The Journal of Nutritional Biochemistry. 64. 170–181. 56 indexed citations

13.

Johnson, Cheilonda, Paul C. Rosen, Thomas E. Lloyd, et al.. (2017). Exploration of the MUC5B promoter variant and ILD risk in patients with autoimmune myositis. Respiratory Medicine. 130. 52–54. 24 indexed citations

14.

Amici, David R., Iago Pinal‐Fernandez, Davi A. G. Mázala, et al.. (2017). Calcium dysregulation, functional calpainopathy, and endoplasmic reticulum stress in sporadic inclusion body myositis. Acta Neuropathologica Communications. 5(1). 24–24. 36 indexed citations

15.

Zhang, Ke, Jonathan C. Grima, Jeffery D. Rothstein, & Thomas E. Lloyd. (2016). Nucleocytoplasmic transport inC9orf72-mediated ALS/FTD. Nucleus. 7(2). 132–137. 23 indexed citations

16.

Herrmann, D., Rita Horváth, Janet E. Sowden, et al.. (2014). Synaptotagmin 2 Mutations Cause an Autosomal-Dominant Form of Lambert-Eaton Myasthenic Syndrome and Nonprogressive Motor Neuropathy. The American Journal of Human Genetics. 95(4). 472–472. 2 indexed citations

17.

Wong, Ching‐On, Kuchuan Chen, Yong Lin, et al.. (2014). A TRPV Channel in Drosophila Motor Neurons Regulates Presynaptic Resting Ca2+ Levels, Synapse Growth, and Synaptic Transmission. Neuron. 84(4). 764–777. 50 indexed citations

18.

Lloyd, Thomas E., James B. Machamer, Sarah E. Collins, et al.. (2012). The p150Glued CAP-Gly Domain Regulates Initiation of Retrograde Transport at Synaptic Termini. Neuron. 74(2). 344–360. 106 indexed citations

19.

Christopher‐Stine, Lisa, Sharon R. Ghazarian, Katherine Pak, et al.. (2012). Antibody levels correlate with creatine kinase levels and strength in anti–3‐hydroxy‐3‐methylglutaryl‐coenzyme A reductase–associated autoimmune myopathy. Arthritis & Rheumatism. 64(12). 4087–4093. 189 indexed citations

20.

Fergestad, Tim, et al.. (1999). Syntaxin 1A Interacts with Multiple Exocytic Proteins to Regulate Neurotransmitter Release In Vivo. Neuron. 23(3). 593–605. 170 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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