Thomas Deller

18.8k total citations · 1 hit paper
228 papers, 11.0k citations indexed

About

Thomas Deller is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Developmental Neuroscience. According to data from OpenAlex, Thomas Deller has authored 228 papers receiving a total of 11.0k indexed citations (citations by other indexed papers that have themselves been cited), including 162 papers in Cellular and Molecular Neuroscience, 78 papers in Molecular Biology and 71 papers in Developmental Neuroscience. Recurrent topics in Thomas Deller's work include Neuroscience and Neuropharmacology Research (104 papers), Neurogenesis and neuroplasticity mechanisms (69 papers) and Neuroinflammation and Neurodegeneration Mechanisms (31 papers). Thomas Deller is often cited by papers focused on Neuroscience and Neuropharmacology Research (104 papers), Neurogenesis and neuroplasticity mechanisms (69 papers) and Neuroinflammation and Neurodegeneration Mechanisms (31 papers). Thomas Deller collaborates with scholars based in Germany, United States and Netherlands. Thomas Deller's co-authors include Michael Frotscher, Andreas Vlachos, Domenico Del Turco, Udo Rüb, Ulrike Müller, Martin Körte, Guido J. Burbach, Carola A. Haas, Mathias Jucker and Stephan W. Schwarzacher and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Nature Communications.

In The Last Decade

Thomas Deller

222 papers receiving 10.9k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Not just amyloid: physiological functions of the amyloid precursor protein family

2017 439 citations Thomas Deller, Martin Körte et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Thomas Deller Germany	59	5.9k	4.0k	2.5k	2.5k	1.9k	228	11.0k
Giles E. Hardingham United Kingdom	55	6.6k 1.1×	7.2k 1.8×	1.8k 0.7×	2.0k 0.8×	1.4k 0.7×	133	12.5k
Martin Körte Germany	53	6.3k 1.1×	4.9k 1.2×	3.3k 1.3×	2.5k 1.0×	2.9k 1.5×	142	13.2k
Andrea Volterra Italy	49	9.3k 1.6×	5.0k 1.2×	2.5k 1.0×	5.5k 2.2×	1.8k 1.0×	90	14.7k
Mike Dragunow New Zealand	72	9.2k 1.6×	7.4k 1.8×	2.7k 1.1×	3.1k 1.3×	2.2k 1.2×	231	18.2k
Baljit S. Khakh United States	64	7.4k 1.3×	5.4k 1.3×	2.0k 0.8×	5.4k 2.2×	2.0k 1.1×	130	16.1k
Brian A. MacVicar Canada	70	7.7k 1.3×	6.3k 1.6×	2.1k 0.8×	4.3k 1.8×	1.3k 0.7×	157	15.5k
Wen‐Biao Gan United States	44	6.4k 1.1×	3.1k 0.8×	1.9k 0.8×	5.2k 2.1×	1.8k 1.0×	90	13.9k
Christian Steinhäuser Germany	56	7.1k 1.2×	5.1k 1.3×	1.4k 0.6×	3.8k 1.6×	2.3k 1.2×	160	11.6k
Masahiro Fukaya Japan	52	5.8k 1.0×	4.9k 1.2×	1.1k 0.4×	1.8k 0.7×	2.0k 1.1×	135	10.4k
Giorgio Carmignoto Italy	52	9.4k 1.6×	4.8k 1.2×	1.9k 0.7×	3.9k 1.6×	1.7k 0.9×	90	13.3k

Countries citing papers authored by Thomas Deller

Since Specialization

Citations

This map shows the geographic impact of Thomas Deller's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Deller with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Deller more than expected).

Fields of papers citing papers by Thomas Deller

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas Deller. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Deller. The network helps show where Thomas Deller may publish in the future.

Co-authorship network of co-authors of Thomas Deller

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas Deller. A scholar is included among the top collaborators of Thomas Deller based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas Deller. Thomas Deller is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Drakew, Alexander, et al.. (2025). Dendritic retraction contributes to spine density recovery following denervation in vitro. Neurobiology of Disease. 216. 107136–107136.

Key, Jana, Luis E. Almaguer-Mederos, Suzana Gispert, et al.. (2025). Conditional ATXN2L-Null in Adult Frontal Cortex CamK2a+ Neurons Does Not Cause Cell Death but Restricts Spontaneous Mobility and Affects the Alternative Splicing Pathway. Cells. 14(19). 1532–1532. 1 indexed citations

Trautmann, Sandra, Tobias Schmid, Bernhard Brüne, et al.. (2021). Prodromal sensory neuropathy in Pink1 ^−/− SNCA ^A53T double mutant Parkinson mice. Neuropathology and Applied Neurobiology. 47(7). 1060–1079. 13 indexed citations

Lenz, Maximilian, et al.. (2021). All-trans retinoic acid induces synaptopodin-dependent metaplasticity in mouse dentate granule cells. eLife. 10. 13 indexed citations

Lenz, Maximilian, et al.. (2021). All-trans retinoic acid induces synaptic plasticity in human cortical neurons. eLife. 10. 48 indexed citations

Degenhardt, Karoline, Jessica Wagner, Angelos Skodras, et al.. (2020). Medin aggregation causes cerebrovascular dysfunction in aging wild-type mice. Proceedings of the National Academy of Sciences. 117(38). 23925–23931. 26 indexed citations

Deller, Thomas, Domenico Del Turco, Désirée Griesemer, et al.. (2019). GABA is a modulator, rather than a classical transmitter, in the medial nucleus of the trapezoid body–lateral superior olive sound localization circuit. The Journal of Physiology. 597(8). 2269–2295. 16 indexed citations

O’Sullivan, Gregory A., Peter Jedlička, Hongxing Chen, et al.. (2016). Forebrain-specific loss of synaptic GABAA receptors results in altered neuronal excitability and synaptic plasticity in mice. Molecular and Cellular Neuroscience. 72. 101–113. 12 indexed citations

Pierre, Sandra, Bona Linke, J. Suo, et al.. (2016). GPVI and Thromboxane Receptor on Platelets Promote Proinflammatory Macrophage Phenotypes during Cutaneous Inflammation. Journal of Investigative Dermatology. 137(3). 686–695. 54 indexed citations

10.

Lenz, Maximilian, Denise Becker, Joab Chapman, et al.. (2014). Systemic inflammation is associated with a reduction in Synaptopodin expression in the mouse hippocampus. Experimental Neurology. 261. 230–235. 34 indexed citations

11.

Vlachos, Andreas, Suneel Reddy‐Alla, Theofilos Papadopoulos, Thomas Deller, & Heinrich Betz. (2012). Homeostatic Regulation of Gephyrin Scaffolds and Synaptic Strength at Mature Hippocampal GABAergic Postsynapses†. Cerebral Cortex. 23(11). 2700–2711. 53 indexed citations

12.

Zeller, Nicolas, Geert Loo, Doron Merkler, et al.. (2011). IκB kinase 2 determines oligodendrocyte loss by non-cell-autonomous activation of NF-κB in the central nervous system. Brain. 134(4). 1184–1198. 84 indexed citations

13.

Kögel, Donat, Thomas Deller, & Christian Behl. (2011). Roles of amyloid precursor protein family members in neuroprotection, stress signaling and aging. Experimental Brain Research. 217(3-4). 471–479. 74 indexed citations

14.

Werno, Christian, Tobias Schmid, Ekaterini Copanaki, et al.. (2010). Roles of Hypoxia-Inducible Factor-1α (Hif-1α) Versus Hif-2α in the Survival of Hepatocellular Tumor Spheroids. Hepatology. 51(6). 2183–2192. 107 indexed citations

15.

Turco, Domenico Del, et al.. (2009). AnkyrinG is required to maintain axo-dendritic polarity in vivo. Proceedings of the National Academy of Sciences. 106(41). 17564–17569. 157 indexed citations

16.

Seidel, Kay, Peter Bauer, Wilfred F.A. den Dunnen, et al.. (2009). Widespread thalamic and cerebellar degeneration in a patient with a complicated hereditary spastic paraplegia (HSP). Annals of Anatomy - Anatomischer Anzeiger. 191(2). 203–211. 9 indexed citations

17.

Stanke, Matthias, Markus Geißen, Guido J. Burbach, et al.. (2005). Target-dependent specification of the neurotransmitter phenotype:cholinergic differentiation of sympathetic neurons is mediated in vivo by gp130 signaling. Development. 133(1). 141–150. 95 indexed citations

18.

Deller, Thomas, Martin Körte, Sophie Chabanis, et al.. (2003). Synaptopodin-deficient mice lack a spine apparatus and show deficits in synaptic plasticity. Proceedings of the National Academy of Sciences. 100(18). 10494–10499. 226 indexed citations

19.

Woods, Alisa G., et al.. (2002). CNTF and CNTF receptor alpha are constitutively expressed by astrocytes in the mouse brain. Glia. 37(4). 374–378. 63 indexed citations

20.

Deller, Thomas, Alexander Drakew, & Michael Frotscher. (1999). Different Primary Target Cells Are Important for Fiber Lamination in the Fascia Dentata: A Lesson from Reeler Mutant Mice. Experimental Neurology. 156(2). 239–253. 51 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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