Thomas D. Corso

837 total citations
17 papers, 712 citations indexed

About

Thomas D. Corso is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Neurology. According to data from OpenAlex, Thomas D. Corso has authored 17 papers receiving a total of 712 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Cellular and Molecular Neuroscience, 8 papers in Molecular Biology and 4 papers in Neurology. Recurrent topics in Thomas D. Corso's work include Neuroscience and Neuropharmacology Research (7 papers), Epigenetics and DNA Methylation (3 papers) and Memory and Neural Mechanisms (3 papers). Thomas D. Corso is often cited by papers focused on Neuroscience and Neuropharmacology Research (7 papers), Epigenetics and DNA Methylation (3 papers) and Memory and Neural Mechanisms (3 papers). Thomas D. Corso collaborates with scholars based in United States, Poland and France. Thomas D. Corso's co-authors include Michael A. Collins, Edward J. Neafsey, John W. Olney, David F. Wozniak, Tatyana Tenkova, Andrew S. Fix, Ewa K. Stachowiak, Nuri B. Farber, Michal K. Stachowiak and T.C. Der and has published in prestigious journals such as Brain Research, Journal of Neurochemistry and Journal of Neuropathology & Experimental Neurology.

In The Last Decade

Thomas D. Corso

17 papers receiving 692 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Thomas D. Corso United States 12 373 246 159 136 129 17 712
Renata Leke Brazil 16 349 0.9× 219 0.9× 84 0.5× 127 0.9× 141 1.1× 22 861
Analı́a Reinés Argentina 17 244 0.7× 272 1.1× 57 0.4× 134 1.0× 142 1.1× 29 775
Hussam Jourdi United States 15 424 1.1× 234 1.0× 66 0.4× 100 0.7× 50 0.4× 18 692
Karen Zheng United States 4 373 1.0× 236 1.0× 94 0.6× 65 0.5× 52 0.4× 4 652
Isabelle Leroux‐Nicollet France 20 600 1.6× 344 1.4× 101 0.6× 95 0.7× 96 0.7× 40 1.0k
Mustafa Q. Hameed United States 12 486 1.3× 270 1.1× 188 1.2× 82 0.6× 146 1.1× 22 921
Keiko Ikemoto Japan 18 504 1.4× 335 1.4× 125 0.8× 33 0.2× 81 0.6× 63 853
Alejandra Bernardi United States 13 394 1.1× 305 1.2× 49 0.3× 107 0.8× 31 0.2× 20 720
B Tabakoff United States 13 825 2.2× 532 2.2× 107 0.7× 80 0.6× 162 1.3× 19 1.1k
Hélène Scarna France 18 429 1.2× 461 1.9× 201 1.3× 28 0.2× 80 0.6× 31 1.0k

Countries citing papers authored by Thomas D. Corso

Since Specialization
Citations

This map shows the geographic impact of Thomas D. Corso's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas D. Corso with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas D. Corso more than expected).

Fields of papers citing papers by Thomas D. Corso

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas D. Corso. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas D. Corso. The network helps show where Thomas D. Corso may publish in the future.

Co-authorship network of co-authors of Thomas D. Corso

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas D. Corso. A scholar is included among the top collaborators of Thomas D. Corso based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas D. Corso. Thomas D. Corso is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Corso, Thomas D., et al.. (2019). Accessory mammillary bodies formed by the enlarged lateral mammillary nuclei: cytoarchitecture. Brain Structure and Function. 224(5). 1971–1974. 2 indexed citations
2.
Kucinski, Aaron, Scott R. Wersinger, Ewa K. Stachowiak, et al.. (2013). Neuronal nicotinic receptor agonists ameliorate spontaneous motor asymmetries and motor discoordination in a unilateral mouse model of Parkinson's disease. Pharmacology Biochemistry and Behavior. 111. 1–10. 12 indexed citations
3.
Stachowiak, Michal K., Aaron Kucinski, Sridhar Narla, et al.. (2012). Schizophrenia: A neurodevelopmental disorder — Integrative genomic hypothesis and therapeutic implications from a transgenic mouse model. Schizophrenia Research. 143(2-3). 367–376. 37 indexed citations
4.
5.
Klejbor, Ilona, Aaron Kucinski, Scott R. Wersinger, et al.. (2009). Serotonergic hyperinnervation and effective serotonin blockade in an FGF receptor developmental model of psychosis. Schizophrenia Research. 113(2-3). 308–321. 18 indexed citations
6.
Klejbor, Ilona, Jason Myers, Thomas D. Corso, et al.. (2006). Fibroblast growth factor receptor signaling affects development and function of dopamine neurons – inhibition results in a schizophrenia‐like syndrome in transgenic mice. Journal of Neurochemistry. 97(5). 1243–1258. 61 indexed citations
7.
Corso, Thomas D., German Torres, Indrajit Roy, et al.. (2005). Assessment of viral and non-viral gene transfer into adult rat brains using HSV-1, calcium phosphate and PEI-based methods.. PubMed. 64(3). 130–44. 6 indexed citations
8.
9.
Tabbaa, Sawsan, Thomas D. Corso, Lawrence C. Jenkins, et al.. (2005). In vivo gene transfer to the brain cortex using a single injection of HSV-1 vector into the medial septum.. PubMed. 64(4). 273–81. 2 indexed citations
10.
Corso, Thomas D., et al.. (1998). Brain Neuronal Degeneration Caused by Episodic Alcohol Intoxication in Rats: Effects of Nimodipine, 6,7‐Dinitro‐quinoxaline‐2,3‐dione, and MK‐801. Alcoholism Clinical and Experimental Research. 22(1). 217–224. 22 indexed citations
11.
Corso, Thomas D., et al.. (1998). Brain Neuronal Degeneration Caused by Episodic Alcohol Intoxication in Rats. Alcoholism Clinical and Experimental Research. 22(1). 217–217. 4 indexed citations
12.
Corso, Thomas D., Hassan Mostafa, Michael A. Collins, & Edward J. Neafsey. (1998). Brain neuronal degeneration caused by episodic alcohol intoxication in rats: effects of nimodipine, 6,7-dinitro-quinoxaline-2,3-dione, and MK-801.. PubMed. 22(1). 217–24. 52 indexed citations
13.
Wozniak, David F., Krikor Dikranian, Masahiko Ishimaru, et al.. (1998). Disseminated Corticolimbic Neuronal Degeneration Induced in Rat Brain by MK-801: Potential Relevance to Alzheimer's Disease. Neurobiology of Disease. 5(5). 305–322. 76 indexed citations
14.
Corso, Thomas D., Michael A. Sesma, Tatyana Tenkova, et al.. (1997). Multifocal brain damage induced by phencyclidine is augmented by pilocarpine. Brain Research. 752(1-2). 1–14. 92 indexed citations
15.
Wozniak, David F., et al.. (1996). MK-801 neurotoxicity in male mice: histologic effects and chronic impairment in spatial learning. Brain Research. 707(2). 165–179. 75 indexed citations
16.
Collins, Michael A., Thomas D. Corso, & Edward J. Neafsey. (1996). Neuronal Degeneration in Rat Cerebrocortical and Olfactory Regions During Subchronic “Binge” Intoxication with Ethanol: Possible Explanation for Olfactory Deficits in Alcoholics. Alcoholism Clinical and Experimental Research. 20(2). 284–292. 147 indexed citations
17.
Corso, Thomas D., et al.. (1995). Excitotoxic Cytopathology, Progression, and Reversibility of Thiamine Deficiency-induced Diencephalic Lesions. Journal of Neuropathology & Experimental Neurology. 54(2). 255–267. 79 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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