Terry S. Elton

8.9k total citations · 1 hit paper
89 papers, 6.5k citations indexed

About

Terry S. Elton is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Cancer Research. According to data from OpenAlex, Terry S. Elton has authored 89 papers receiving a total of 6.5k indexed citations (citations by other indexed papers that have themselves been cited), including 65 papers in Molecular Biology, 24 papers in Cardiology and Cardiovascular Medicine and 18 papers in Cancer Research. Recurrent topics in Terry S. Elton's work include Receptor Mechanisms and Signaling (22 papers), MicroRNA in disease regulation (16 papers) and Renin-Angiotensin System Studies (12 papers). Terry S. Elton is often cited by papers focused on Receptor Mechanisms and Signaling (22 papers), MicroRNA in disease regulation (16 papers) and Renin-Angiotensin System Studies (12 papers). Terry S. Elton collaborates with scholars based in United States, China and Switzerland. Terry S. Elton's co-authors include Mickey M. Martin, Nathan K. Evanson, Naduviladath Vishvanath Chandrasekharan, Daniel L. Simmons, Katarina Roos, Raymond Reeves, Suzanne Oparil, David S. Feldman, Narasimham L. Parinandi and Y. F. Chen and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Circulation.

In The Last Decade

Terry S. Elton

87 papers receiving 6.3k citations

Hit Papers

1 papers align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

COX-3, a cyclooxygenase-1 variant inhibited by acetaminophen and other analgesic/antipyretic drugs: Cloning, structure, and expression

2002 1.5k citations Terry S. Elton et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Terry S. Elton United States	33	3.4k	1.8k	1.1k	1.0k	775	89	6.5k
Juan Miguel Redondo Spain	51	3.8k 1.1×	1.1k 0.6×	526 0.5×	567 0.6×	617 0.8×	135	6.7k
De‐Pei Liu China	49	4.1k 1.2×	1.1k 0.6×	1.1k 1.0×	284 0.3×	1.3k 1.7×	242	8.0k
John Hwa United States	48	3.6k 1.1×	482 0.3×	840 0.8×	611 0.6×	624 0.8×	130	6.6k
Alfredo Criollo Chile	45	4.5k 1.3×	1.1k 0.6×	593 0.6×	506 0.5×	915 1.2×	75	8.9k
Yong Zhang China	49	5.4k 1.6×	2.9k 1.6×	968 0.9×	328 0.3×	828 1.1×	256	8.6k
Michael Gekle Germany	51	3.6k 1.1×	1.2k 0.6×	652 0.6×	326 0.3×	551 0.7×	241	8.0k
Goo Taeg Oh South Korea	45	3.3k 1.0×	640 0.3×	496 0.5×	744 0.7×	897 1.2×	175	6.9k
Agapios Sachinidis Germany	47	4.2k 1.3×	697 0.4×	955 0.9×	311 0.3×	796 1.0×	275	7.5k
Grant M. Hatch Canada	50	4.1k 1.2×	651 0.4×	500 0.5×	550 0.5×	1.1k 1.4×	187	6.6k
Jifeng Zhang United States	53	5.3k 1.6×	1.4k 0.8×	1.4k 1.3×	397 0.4×	1.7k 2.1×	227	9.5k

Countries citing papers authored by Terry S. Elton

Since Specialization

Citations

This map shows the geographic impact of Terry S. Elton's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Terry S. Elton with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Terry S. Elton more than expected).

Fields of papers citing papers by Terry S. Elton

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Terry S. Elton. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Terry S. Elton. The network helps show where Terry S. Elton may publish in the future.

Co-authorship network of co-authors of Terry S. Elton

This figure shows the co-authorship network connecting the top 25 collaborators of Terry S. Elton. A scholar is included among the top collaborators of Terry S. Elton based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Terry S. Elton. Terry S. Elton is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Wang, Xinyi, Vı́ctor Agmo Hernández, Milon Mondal, et al.. (2024). Use of CRISPR/Cas9 with Homology-Directed Repair to Gene-Edit Topoisomerase II β in Human Leukemia K562 Cells: Generation of a Resistance Phenotype. Journal of Pharmacology and Experimental Therapeutics. 389(2). 186–196. 1 indexed citations

Wang, Xinyi, et al.. (2023). Strategies to Circumvent Topoisomerase IIα Intron 19 Intronic Polyadenylation (IPA) in Acquired Etoposide Resistance Human Leukemia K562 Cells. Journal of Pharmacology and Experimental Therapeutics. 385. 251–251.

Elton, Terry S., et al.. (2022). Intronic Polyadenylation in Acquired Cancer Drug Resistance Circumvented by Utilizing CRISPR/Cas9 with Homology-Directed Repair: The Tale of Human DNA Topoisomerase IIα. Cancers. 14(13). 3148–3148. 8 indexed citations

Hernández, Vı́ctor Agmo, et al.. (2022). Effects of hsa-miR-9-3p and hsa-miR-9-5p on Topoisomerase II β Expression in Human Leukemia K562 Cells with Acquired Resistance to Etoposide. Journal of Pharmacology and Experimental Therapeutics. 384(2). 265–276. 5 indexed citations

Hernández, Vı́ctor Agmo, et al.. (2021). CRISPR/Cas9 Genome Editing of the Human Topoisomerase IIα Intron 19 5′ Splice Site Circumvents Etoposide Resistance in Human Leukemia K562 Cells. Molecular Pharmacology. 99(3). 226–241. 11 indexed citations

Kanagasabai, Ragu, et al.. (2018). The Novel C-terminal Truncated 90-kDa Isoform of Topoisomerase IIα (TOP2α/90) Is a Determinant of Etoposide Resistance in K562 Leukemia Cells via Heterodimerization with the TOP2α/170 Isoform. Molecular Pharmacology. 93(5). 515–525. 13 indexed citations

Elton, Terry S. & Jack C. Yalowich. (2015). Experimental procedures to identify and validate specific mRNA targets of miRNAs. PubMed. 14. 758–90. 21 indexed citations

Elton, Terry S., Mahmood Khan, & Dmitry Terentyev. (2011). MicroRNAs in cardiovascular disease. F1000 Medicine Reports. 3. 10–10. 14 indexed citations

Terentyev, Dmitry, Radmila Terentyeva, Andriy E. Belevych, et al.. (2009). Abstract 2635: Phosphorylation of Ryanodine Receptor by Both PKA and CAMKII is Required for Induction of Arrhythmogenic Disturbances in Myocyte Ca Cycling Caused by miR-1. Circulation. 120. 1 indexed citations

10.

Wexler, Randy, Terry S. Elton, Christopher A. Taylor, Adam Pleister, & David S. Feldman. (2009). Physician reported perception in the treatment of high blood pressure does not correspond to practice. BMC Family Practice. 10(1). 23–23. 29 indexed citations

11.

Wexler, Randy, Terry S. Elton, Adam Pleister, & David S. Feldman. (2009). Barriers to Blood Pressure Control as Reported by African American Patients. Journal of the National Medical Association. 101(6). 597–603. 27 indexed citations

12.

Thacker, Tyler C., Xueyuan Zhou, Jacob D. Estes, et al.. (2008). Follicular Dendritic Cells and Human Immunodeficiency Virus Type 1 Transcription in CD4⁺T Cells. Journal of Virology. 83(1). 150–158. 39 indexed citations

13.

Mihai, Cosmin, Maqsood A. Chotani, Terry S. Elton, & Gunjan Agarwal. (2008). Mapping of DDR1 Distribution and Oligomerization on the Cell Surface by FRET Microscopy. Journal of Molecular Biology. 385(2). 432–445. 75 indexed citations

14.

Sethupathy, Praveen, Christelle Borel, Maryline Gagnebin, et al.. (2007). Human microRNA-155 on Chromosome 21 Differentially Interacts with Its Polymorphic Target in the AGTR1 3′ Untranslated Region: A Mechanism for Functional Single-Nucleotide Polymorphisms Related to Phenotypes. The American Journal of Human Genetics. 81(2). 405–413. 305 indexed citations

15.

Martin, Mickey M., et al.. (2001). Identification and characterization of functional angiotensin II type 1 receptors on immortalized human fetal aortic vascular smooth muscle cells. Molecular and Cellular Endocrinology. 183(1-2). 81–91. 10 indexed citations

16.

Lazarov, Mirella, Mickey M. Martin, Barry M. Willardson, & Terry S. Elton. (2000). Molecular cloning and characterization of the human phosducin-like protein (hPhLP) promoter. Biochimica et Biophysica Acta (BBA) - Gene Structure and Expression. 1492(2-3). 460–464.

17.

Martin, Mickey M., C. Roger White, Huaibin Li, Patrick J. Miller, & Terry S. Elton. (1995). A functional comparison of the rat type-1 angiotensin II receptors (AT1AR and AT1BR). Regulatory Peptides. 60(2-3). 135–147. 24 indexed citations

18.

Elton, Terry S., et al.. (1995). Molecular Cloning of a cDNA Encoding Putative Molt-Inhibiting Hormone from the Blue Crab, Callinectes sapidus. Biochemical and Biophysical Research Communications. 209(3). 1126–1131. 57 indexed citations

19.

Martin, Mickey M., et al.. (1994). The Genomic Organization and Functional Analysis of the Promoter for the Human Angiotensin II Type 1 Receptor. Biochemical and Biophysical Research Communications. 204(3). 1039–1046. 23 indexed citations

20.

Reeves, Raymond & Terry S. Elton. (1987). Non-histone chromatin proteins that recognize specific sequences of DNA. Journal of Chromatography B Biomedical Sciences and Applications. 418. 73–95. 5 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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