Telmo Catarino

593 total citations
11 papers, 390 citations indexed

About

Telmo Catarino is a scholar working on Molecular Biology, Immunology and Rheumatology. According to data from OpenAlex, Telmo Catarino has authored 11 papers receiving a total of 390 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 5 papers in Immunology and 2 papers in Rheumatology. Recurrent topics in Telmo Catarino's work include Glycosylation and Glycoproteins Research (4 papers), Immune Cell Function and Interaction (3 papers) and T-cell and B-cell Immunology (2 papers). Telmo Catarino is often cited by papers focused on Glycosylation and Glycoproteins Research (4 papers), Immune Cell Function and Interaction (3 papers) and T-cell and B-cell Immunology (2 papers). Telmo Catarino collaborates with scholars based in Portugal, United States and France. Telmo Catarino's co-authors include Pedro Gonçalves, Fátima Martel, Salomé S. Pinho, Celso A. Reis, Naoyuki Taniguchi, Ana M. Dias, Helena Pópulo, Paula Soares, Ricardo Celestino and Márcia S. Pereira and has published in prestigious journals such as Proceedings of the National Academy of Sciences, SHILAP Revista de lepidopterología and PLoS ONE.

In The Last Decade

Telmo Catarino

10 papers receiving 388 citations

Peers

Telmo Catarino
Neha M. Akella United States
Robert H. Keith United States
Xue Lv China
J B Stoudemire United States
Beverly S. Adler United States
Telmo Catarino
Citations per year, relative to Telmo Catarino Telmo Catarino (= 1×) peers Kazuhiro Nagahira

Countries citing papers authored by Telmo Catarino

Since Specialization
Citations

This map shows the geographic impact of Telmo Catarino's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Telmo Catarino with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Telmo Catarino more than expected).

Fields of papers citing papers by Telmo Catarino

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Telmo Catarino. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Telmo Catarino. The network helps show where Telmo Catarino may publish in the future.

Co-authorship network of co-authors of Telmo Catarino

This figure shows the co-authorship network connecting the top 25 collaborators of Telmo Catarino. A scholar is included among the top collaborators of Telmo Catarino based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Telmo Catarino. Telmo Catarino is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
2.
Pereira, Márcia S., Cecília Durães, Telmo Catarino, et al.. (2020). Genetic Variants of the MGAT5 Gene Are Functionally Implicated in the Modulation of T Cells Glycosylation and Plasma IgG Glycome Composition in Ulcerative Colitis. Clinical and Translational Gastroenterology. 11(4). e00166–e00166. 22 indexed citations
3.
Dias, Ana M., Alexandra Correia, Márcia S. Pereira, et al.. (2018). Metabolic control of T cell immune response through glycans in inflammatory bowel disease. Proceedings of the National Academy of Sciences. 115(20). E4651–E4660. 86 indexed citations
4.
Pereira, Márcia S., Cecília Durães, Telmo Catarino, et al.. (2018). P850 Genetic variants of MGAT5 gene are associated with ulcerative colitis severity and response to therapy. Journal of Crohn s and Colitis. 12(supplement_1). S546–S547. 1 indexed citations
5.
Pereira, Cristiana, Cecília Durães, Rosa Coelho, et al.. (2017). Association between Polymorphisms in Antioxidant Genes and Inflammatory Bowel Disease. PLoS ONE. 12(1). e0169102–e0169102. 20 indexed citations
6.
Gonçalves, Pedro, et al.. (2017). The Effect of Inflammatory Status on Butyrate and Folate Uptake by Tumoral (Caco-2) and Non-Tumoral (IEC-6) Intestinal Epithelial Cells.. SHILAP Revista de lepidopterología. 19(Suppl 1). 96–105. 8 indexed citations
7.
Carvalho, Sandra, Telmo Catarino, Ana M. Dias, et al.. (2015). Preventing E-cadherin aberrant N-glycosylation at Asn-554 improves its critical function in gastric cancer. Oncogene. 35(13). 1619–1631. 99 indexed citations
8.
Vinagre, João, Vasco Pinto, Ricardo Celestino, et al.. (2014). Telomerase promoter mutations in cancer: an emerging molecular biomarker?. Archiv für Pathologische Anatomie und Physiologie und für Klinische Medicin. 465(2). 119–133. 91 indexed citations
9.
Gonçalves, Pedro, et al.. (2012). Inhibition of butyrate uptake by the primary bile salt chenodeoxycholic acid in intestinal epithelial cells. Journal of Cellular Biochemistry. 113(9). 2937–2947. 17 indexed citations
10.
Gonçalves, Pedro, et al.. (2012). The effect of oxidative stress upon the intestinal uptake of folic acid: in vitro studies with Caco-2 cells. Cell Biology and Toxicology. 28(6). 369–381. 22 indexed citations
11.
Gonçalves, Pedro, et al.. (2012). The effect of oxidative stress upon the intestinal epithelial uptake of butyrate. European Journal of Pharmacology. 699(1-3). 88–100. 24 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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