Tamás Kozicz

8.2k total citations
177 papers, 5.8k citations indexed

About

Tamás Kozicz is a scholar working on Behavioral Neuroscience, Physiology and Molecular Biology. According to data from OpenAlex, Tamás Kozicz has authored 177 papers receiving a total of 5.8k indexed citations (citations by other indexed papers that have themselves been cited), including 73 papers in Behavioral Neuroscience, 61 papers in Physiology and 50 papers in Molecular Biology. Recurrent topics in Tamás Kozicz's work include Stress Responses and Cortisol (73 papers), Neuroendocrine regulation and behavior (40 papers) and Adipose Tissue and Metabolism (34 papers). Tamás Kozicz is often cited by papers focused on Stress Responses and Cortisol (73 papers), Neuroendocrine regulation and behavior (40 papers) and Adipose Tissue and Metabolism (34 papers). Tamás Kozicz collaborates with scholars based in Netherlands, United States and Hungary. Tamás Kozicz's co-authors include Eric W. Roubos, Akira Arimura, Éva Morava, Balázs Gaszner, Lu Xu, Hitoshi Yanaihara, Sándor Vigh, Tim L. Emmerzaal, Miklós Palkovits and Zane B. Andrews and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and PLoS ONE.

In The Last Decade

Tamás Kozicz

171 papers receiving 5.7k citations

Peers

Tamás Kozicz
Teresa M. Reyes United States
Jan Pieter Konsman France
Raffaella Molteni Italy
Jaideep S. Bains Canada
Jackson C. Bittencourt Brazil
Gert J. Ter Horst Netherlands
Deborah A. Finn United States
Michela Marinelli United States
Darin J. Knapp United States
Alexander Yassouridis Germany
Teresa M. Reyes United States
Tamás Kozicz
Citations per year, relative to Tamás Kozicz Tamás Kozicz (= 1×) peers Teresa M. Reyes

Countries citing papers authored by Tamás Kozicz

Since Specialization
Citations

This map shows the geographic impact of Tamás Kozicz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tamás Kozicz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tamás Kozicz more than expected).

Fields of papers citing papers by Tamás Kozicz

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tamás Kozicz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tamás Kozicz. The network helps show where Tamás Kozicz may publish in the future.

Co-authorship network of co-authors of Tamás Kozicz

This figure shows the co-authorship network connecting the top 25 collaborators of Tamás Kozicz. A scholar is included among the top collaborators of Tamás Kozicz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tamás Kozicz. Tamás Kozicz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kozicz, Tamás, et al.. (2025). The Therapeutic Future for Congenital Disorders of Glycosylation. Journal of Inherited Metabolic Disease. 48(2). e70011–e70011. 2 indexed citations
2.
Radenkovic, Silvia, et al.. (2024). Liposome-encapsulated mannose-1-phosphate therapy improves global N-glycosylation in different congenital disorders of glycosylation. Molecular Genetics and Metabolism. 142(2). 108487–108487. 8 indexed citations
3.
Radenkovic, Silvia, Teun M. Klein Gunnewiek, Tarun N. Bhatia, et al.. (2024). Neural and metabolic dysregulation in PMM2-deficient human in vitro neural models. Cell Reports. 43(3). 113883–113883. 7 indexed citations
4.
Krzyściak, Wirginia, Beata Bystrowska, Robert Chrzan, et al.. (2024). Association of Blood Metabolomics Biomarkers with Brain Metabolites and Patient-Reported Outcomes as a New Approach in Individualized Diagnosis of Schizophrenia. International Journal of Molecular Sciences. 25(4). 2294–2294. 6 indexed citations
5.
Ranatunga, Wasantha, et al.. (2024). Normal transferrin glycosylation does not rule out severe ALG1 deficiency. JIMD Reports. 65(3). 135–143. 1 indexed citations
6.
Kozicz, Tamás, et al.. (2024). Assessing age of onset and clinical symptoms over time in patients with heterozygous pathogenic DHDDS variants. Journal of Inherited Metabolic Disease. 47(5). 935–944. 1 indexed citations
7.
8.
Krzyściak, Wirginia, Beata Bystrowska, Anna N. Ligezka, et al.. (2023). The Association of the Oral Microbiota with the Effects of Acid Stress Induced by an Increase of Brain Lactate in Schizophrenia Patients. Biomedicines. 11(2). 240–240. 8 indexed citations
9.
Gardea‐Resendez, Manuel, Brandon J. Coombes, Marin Veldić, et al.. (2022). Antidepressants that increase mitochondrial energetics may elevate risk of treatment-emergent mania. Molecular Psychiatry. 28(3). 1020–1026. 8 indexed citations
10.
Popiela, Tadeusz, Wirginia Krzyściak, Fabio Pilato, et al.. (2022). The Assessment of Endovascular Therapies in Ischemic Stroke: Management, Problems and Future Approaches. Journal of Clinical Medicine. 11(7). 1864–1864. 13 indexed citations
11.
Saraswat, Mayank, et al.. (2022). N‐glycoproteomics reveals distinct glycosylation alterations in NGLY1‐deficient patient‐derived dermal fibroblasts. Journal of Inherited Metabolic Disease. 46(1). 76–91. 12 indexed citations
12.
Johnsen, Christin, Seul Kee Byeon, Wasantha Ranatunga, et al.. (2022). TRIT1 defect leads to a recognizable phenotype of myoclonic epilepsy, speech delay, strabismus, progressive spasticity, and normal lactate levels. Journal of Inherited Metabolic Disease. 45(6). 1039–1047. 8 indexed citations
13.
Emmerzaal, Tim L., Marin Veldić, Shamima Rahman, et al.. (2021). Effect of neuropsychiatric medications on mitochondrial function: For better or for worse. Neuroscience & Biobehavioral Reviews. 127. 555–571. 18 indexed citations
14.
Preston, Graeme, Tim L. Emmerzaal, Silvia Radenkovic, et al.. (2021). Cerebellar and multi-system metabolic reprogramming associated with trauma exposure and post-traumatic stress disorder (PTSD)-like behavior in mice. Neurobiology of Stress. 14. 100300–100300. 9 indexed citations
15.
Emmerzaal, Tim L., Bram Geenen, Viviènne Verweij, et al.. (2020). Chronic fluoxetine or ketamine treatment differentially affects brain energy homeostasis which is not exacerbated in mice with trait suboptimal mitochondrial function. European Journal of Neuroscience. 53(9). 2986–3001. 8 indexed citations
16.
Emmerzaal, Tim L., Graeme Preston, Bram Geenen, et al.. (2020). Impaired mitochondrial complex I function as a candidate driver in the biological stress response and a concomitant stress-induced brain metabolic reprogramming in male mice. Translational Psychiatry. 10(1). 176–176. 35 indexed citations
17.
Krzyściak, Wirginia, et al.. (2020). Oxidative-Antioxidant Imbalance and Impaired Glucose Metabolism in Schizophrenia. Biomolecules. 10(3). 384–384. 46 indexed citations
18.
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Homberg, Judith R., et al.. (2017). Epigenetic programming of the neuroendocrine stress response by adult life stress. Journal of Molecular Endocrinology. 59(1). R11–R31. 50 indexed citations
20.
Marcinkiewcz, Catherine A., Christopher M. Mazzone, Giuseppe D’Agostino, et al.. (2016). Serotonin engages an anxiety and fear-promoting circuit in the extended amygdala. Nature. 537(7618). 97–101. 316 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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