Tal Teitz

2.6k total citations · 1 hit paper
31 papers, 1.9k citations indexed

About

Tal Teitz is a scholar working on Molecular Biology, Neurology and Oncology. According to data from OpenAlex, Tal Teitz has authored 31 papers receiving a total of 1.9k indexed citations (citations by other indexed papers that have themselves been cited), including 26 papers in Molecular Biology, 10 papers in Neurology and 9 papers in Oncology. Recurrent topics in Tal Teitz's work include Cell death mechanisms and regulation (12 papers), Neuroblastoma Research and Treatments (10 papers) and Hearing, Cochlea, Tinnitus, Genetics (5 papers). Tal Teitz is often cited by papers focused on Cell death mechanisms and regulation (12 papers), Neuroblastoma Research and Treatments (10 papers) and Hearing, Cochlea, Tinnitus, Genetics (5 papers). Tal Teitz collaborates with scholars based in United States, Israel and Japan. Tal Teitz's co-authors include Vincent J. Kidd, Jill M. Lahti, Virginia Valentine, José Grenet, Elio F. Vanin, A. Thomas Look, Dwayne G. Stupack, David Mikolon, Dan Canaani and Peter J. Houghton and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Nature Medicine.

In The Last Decade

Tal Teitz

30 papers receiving 1.9k citations

Hit Papers

Caspase 8 is deleted or silenced preferentially in childh... 2000 2026 2008 2017 2000 200 400 600

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Tal Teitz United States 19 1.5k 484 465 414 277 31 1.9k
Arturo Sala United Kingdom 29 1.6k 1.1× 272 0.6× 1.1k 2.3× 402 1.0× 340 1.2× 55 2.3k
Behnam Nabet United States 22 3.1k 2.1× 95 0.2× 619 1.3× 356 0.9× 237 0.9× 41 3.6k
Elizabeth E. Reczek United States 9 2.1k 1.4× 364 0.8× 1.2k 2.6× 513 1.2× 305 1.1× 10 3.2k
Ronald K. Gary United States 18 1.5k 1.0× 443 0.9× 296 0.6× 235 0.6× 103 0.4× 31 2.1k
Peter J. Hurlin United States 27 1.6k 1.1× 84 0.2× 580 1.2× 296 0.7× 166 0.6× 47 2.0k
Steve Silletti United States 24 1.5k 1.0× 117 0.2× 598 1.3× 705 1.7× 300 1.1× 49 2.8k
Maria-Magdalena Georgescu United States 17 1.8k 1.2× 72 0.1× 339 0.7× 237 0.6× 495 1.8× 29 2.5k
Kersi Pestonjamasp United States 19 1.2k 0.8× 153 0.3× 177 0.4× 104 0.3× 350 1.3× 36 1.9k
Ken Aldape United States 15 1.3k 0.9× 149 0.3× 540 1.2× 743 1.8× 252 0.9× 21 2.4k
Jakob Lovén United States 15 2.5k 1.7× 127 0.3× 435 0.9× 636 1.5× 231 0.8× 17 3.1k

Countries citing papers authored by Tal Teitz

Since Specialization
Citations

This map shows the geographic impact of Tal Teitz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tal Teitz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tal Teitz more than expected).

Fields of papers citing papers by Tal Teitz

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tal Teitz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tal Teitz. The network helps show where Tal Teitz may publish in the future.

Co-authorship network of co-authors of Tal Teitz

This figure shows the co-authorship network connecting the top 25 collaborators of Tal Teitz. A scholar is included among the top collaborators of Tal Teitz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tal Teitz. Tal Teitz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Ingersoll, Matthew A., et al.. (2024). Trametinib, a MEK1/2 Inhibitor, Protects Mice from Cisplatin- and Noise-Induced Hearing Loss. Pharmaceuticals. 17(6). 735–735.
2.
Ingersoll, Matthew A., et al.. (2024). KSR1 knockout mouse model demonstrates MAPK pathway's key role in cisplatin- and noise-induced hearing loss. Journal of Neuroscience. 44(18). e2174232024–e2174232024. 5 indexed citations
3.
Ingersoll, Matthew A., Regina G. Kelmann, Huizhan Liu, et al.. (2023). Dabrafenib protects from cisplatin-induced hearing loss in a clinically relevant mouse model. JCI Insight. 8(24). 5 indexed citations
4.
Ingersoll, Matthew A., Zhenhang Xu, Marisa Zallocchi, et al.. (2020). BRAF inhibition protects against hearing loss in mice. Science Advances. 6(49). 40 indexed citations
5.
Hazlitt, Robert A., Tal Teitz, Shiyong Diao, et al.. (2018). Development of Second-Generation CDK2 Inhibitors for the Prevention of Cisplatin-Induced Hearing Loss. Journal of Medicinal Chemistry. 61(17). 7700–7709. 26 indexed citations
6.
Xu, Heng, Giles Robinson, Jie Huang, et al.. (2015). Common variants in ACYP2 influence susceptibility to cisplatin-induced hearing loss. Nature Genetics. 47(3). 263–266. 92 indexed citations
7.
Teitz, Tal, Marcus B. Valentine, Kejin Zhu, et al.. (2013). Th-MYCN Mice with Caspase-8 Deficiency Develop Advanced Neuroblastoma with Bone Marrow Metastasis. Cancer Research. 73(13). 4086–4097. 48 indexed citations
8.
Teitz, Tal, Jennifer J. Stanke, Sara M. Federico, et al.. (2011). Preclinical Models for Neuroblastoma: Establishing a Baseline for Treatment. PLoS ONE. 6(4). e19133–e19133. 70 indexed citations
9.
Barbero, Simone, Ainhoa Mielgo, Vicente A. Torres, et al.. (2009). Caspase-8 Association with the Focal Adhesion Complex Promotes Tumor Cell Migration and Metastasis. Cancer Research. 69(9). 3755–3763. 112 indexed citations
10.
Stupack, Dwayne G., Tal Teitz, David Mikolon, et al.. (2006). Potentiation of neuroblastoma metastasis by loss of caspase-8. Nature. 439(7072). 95–99. 210 indexed citations
11.
Teitz, Tal, Dong Liu, Virginia Valentine, et al.. (2002). Caspase-9 and Apaf-1 are expressed and functionally active in human neuroblastoma tumor cell lines with 1p36 LOH and amplified MYCN. Oncogene. 21(12). 1848–1858. 37 indexed citations
12.
Takita, Junko, Hong Wei Yang, Yuyan Chen, et al.. (2001). Allelic imbalance on chromosome 2q and alterations of the caspase 8 gene in neuroblastoma. Oncogene. 20(32). 4424–4432. 75 indexed citations
13.
Takita, Junko, Hong Yang, Fumio Bessho, et al.. (2000). Absent or reduced expression of thecaspase 8 gene occurs frequently in neuroblastoma, but not commonly in Ewing sarcoma or rhabdomyosarcoma. Medical and Pediatric Oncology. 35(6). 541–543. 33 indexed citations
14.
Kidd, Vincent J., Jill M. Lahti, & Tal Teitz. (2000). Proteolytic regulation of apoptosis. Seminars in Cell and Developmental Biology. 11(3). 191–201. 74 indexed citations
15.
Teitz, Tal, Elio F. Vanin, José Grenet, et al.. (2000). Caspase 8 is deleted or silenced preferentially in childhood neuroblastomas with amplification of MYCN. Nature Medicine. 6(5). 529–535. 623 indexed citations breakdown →
16.
Teitz, Tal, Judy C. Chang, Yuet Wai Kan, & T. S. Benedict Yen. (1995). Thymic epithelial neoplasms in transgenic mice expressing SV40 T antigen under the control of an erythroid‐specific enhancer. The Journal of Pathology. 177(3). 309–315. 5 indexed citations
17.
Teitz, Tal, T. S. Benedict Yen, Judy C. Chang, & Yuet Wai Kan. (1994). SV40 T Antigen Directed by a Powerful Erythroid Enhancer–Promoter Produced Sarcomas and Pancreatic Tumors But Not Erythroid-Specific Tumors in Transgenic Mice. DNA and Cell Biology. 13(7). 705–710. 3 indexed citations
18.
Teitz, Tal, Mary Bakhanashvili, Tova Naiman, et al.. (1990). Expression of the cDNA for the beta subunit of human casein kinase II confers partial UV resistance on xeroderma pigmentosum cells. Mutation Research/DNA Repair. 236(1). 85–97. 52 indexed citations
20.
Yang‐Feng, Teresa L., et al.. (1990). Assignment of the human casein kinase II β-subunit gene to 6p12→p21. Genomics. 8(4). 741–742. 16 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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