Tal Ilani

1.8k citations
26 papers · 1.3k · h-index 18

Impact in

Papers in

    • Glycosylation and Glycoproteins Research 4
    • Receptor Mechanisms and Signaling 4
    • Endoplasmic Reticulum Stress and Disease 6
    • Cellular transport and secretion 4

Tal Ilani

26 papers receiving 1.3k citations

Peers

Tal Ilani
Comparison fields: 5 of 119
  • Biological Psychiatry 111
  • Structural Biology 36
  • Immunology and Allergy 118
  • Immunology 295
  • Cell Biology 201
Replace Tomas Malinauskas with:
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Tal Ilani relative to Tomas Malinauskas United Kingdom Tomas Malinauskas's profile →
Citations per field
00.5×4.5×
Tomas Malinauskas · 1×
Citations per year

Countries citing papers authored by Tal Ilani

Since Specialization
Citations

This map shows the geographic impact of Tal Ilani's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tal Ilani with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tal Ilani more than expected).

Fields of papers citing papers by Tal Ilani

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tal Ilani. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tal Ilani. The network helps show where Tal Ilani may publish in the future.

Co-authors

The 25 scholars most cited alongside Tal Ilani, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Tal Ilani Line = papers co-authored together Tal Ilani links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 26 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2009184
2 2001142
3 2013139
4 2017110
5 2020102
6 200487
7 200786
8 201780
9 200346
10 201541
11 202240
12 201539
13 201938
14 201031
15 201323
16 200223
17 202222
18 201617
19 201817
20 202016

About Tal Ilani

Tal Ilani is a scholar working on Molecular Biology, Cell Biology, Immunology, Immunology and Allergy and Biological Psychiatry, having authored 26 papers that have together received 1.3k indexed citations. Recurring topics across this work include Endoplasmic Reticulum Stress and Disease (6 papers), Galectins and Cancer Biology (4 papers), Cell Adhesion Molecules Research (4 papers), Glycosylation and Glycoproteins Research (4 papers), Receptor Mechanisms and Signaling (4 papers), Cellular transport and secretion (4 papers), Tryptophan and brain disorders (3 papers) and Monoclonal and Polyclonal Antibodies Research (3 papers). The work is most often cited by research in Biological Psychiatry (111 citations), Structural Biology (36 citations), Immunology and Allergy (118 citations), Immunology (295 citations) and Cell Biology (201 citations). Tal Ilani has collaborated with scholars based in Israel, United States and Singapore. Frequent co-authors include Deborah Fass, Sara Fuchs, Rael D. Strous, Anthony Bretscher, Gaia Vasiliver-Shamis, Michael L. Dustin, Santosha A. Vardhana, David Margulies, Iris Grossman and Leila Motiei. Their work appears in journals such as Cell, Journal of Molecular Biology, The FASEB Journal, The Journal of Cell Biology and Journal of the American Chemical Society.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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