Takahiro Itoshima

436 total citations
11 papers, 374 citations indexed

About

Takahiro Itoshima is a scholar working on Oncology, Molecular Biology and Biotechnology. According to data from OpenAlex, Takahiro Itoshima has authored 11 papers receiving a total of 374 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Oncology, 5 papers in Molecular Biology and 3 papers in Biotechnology. Recurrent topics in Takahiro Itoshima's work include Cancer-related Molecular Pathways (5 papers), Cancer Research and Treatments (3 papers) and Virus-based gene therapy research (2 papers). Takahiro Itoshima is often cited by papers focused on Cancer-related Molecular Pathways (5 papers), Cancer Research and Treatments (3 papers) and Virus-based gene therapy research (2 papers). Takahiro Itoshima collaborates with scholars based in Japan, United States and Switzerland. Takahiro Itoshima's co-authors include Jack A. Roth, Noriaki Tanaka, Toshiyoshi Fujiwara, Yoshihiko Kadowaki, Takuya Fukazawa, Jianghua Shao, Futoshi Uno, Shunsuke Kagawa, Yoshiko Takata and Tomoki Yamatsuji and has published in prestigious journals such as The Journal of Immunology, Oncogene and Brain Research.

In The Last Decade

Takahiro Itoshima

9 papers receiving 364 citations

Peers

Takahiro Itoshima
Comparison fields: 5 of 59
  • Molecular Biology 243
  • Oncology 172
  • Cancer Research 84
  • Immunology 60
  • Genetics 59
Replace Jana Wendt with:
Jana Wendt Germany
Swati Sathe United States
Tineke Timmer Netherlands
Maya Ameyar France
Hong Shik Yun South Korea
Anju Thomas United States
G. Pons Spain
Carrol Terleth Netherlands
M. Matsushima Japan
Ayumi Tsukamoto Japan
Jana Wendt Germany View profile →
Citations per field, relative to Takahiro Itoshima
Takahiro Itoshima · 1×
Citations per year, relative to Takahiro Itoshima
Takahiro Itoshima · 1×

Countries citing papers authored by Takahiro Itoshima

Since Specialization
Citations

This map shows the geographic impact of Takahiro Itoshima's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Takahiro Itoshima with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Takahiro Itoshima more than expected).

Fields of papers citing papers by Takahiro Itoshima

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Takahiro Itoshima. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Takahiro Itoshima. The network helps show where Takahiro Itoshima may publish in the future.

Co-authorship network of co-authors of Takahiro Itoshima

This figure shows the co-authorship network connecting the top 25 collaborators of Takahiro Itoshima. A scholar is included among the top collaborators of Takahiro Itoshima based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Takahiro Itoshima. Takahiro Itoshima is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
# Work Indexed citations
1 22
2 124
3 33
4 122
5
Induction of apoptosis in human esophageal cancer cells by sequential transfer of the wild-type p53 and E2F-1 genes: involvement of p53 accumulation via ARF-mediated MDM2 down-regulation.
36
6
Induction of differentiation-dependent apoptosis in human esophageal squamous cell carcinoma by adenovirus-mediated p21sdi1 gene transfer.
13
7
[A case of breast cancer metastasis to the skin and bone 13 years after, and to the brain 17 years after mastectomy].
0
8 1
9 22
10 0
11 1

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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