TA Springer
- Immunology and Allergy top 1%
- Cell Adhesion Molecules Research 6
- Hematology top 2%
- Platelet Disorders and Treatments 3
- Immunology top 5%
- Immune Cell Function and Interaction 4
- Mast cells and histamine 3
- Complement system in diseases 3
- Immunodeficiency and Autoimmune Disorders 2
- Internal Medicine top 10%
- Genetics top 10%
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- Glycosylation and Glycoproteins Research 3
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- Pneumocystis jirovecii pneumonia detection and treatment 2
- Co-authors
- Tatsuo KinashiDF BaintonKiyoshi TakatsuKarl WeltePaul E. HarrisP RalphM A MooreR. A. Harrison
- Journals
- Blood (10 papers)PubMed (3 papers)
- Partner nations
- United StatesUnited KingdomJapan
In The Last Decade
TA Springer
13 papers receiving 1.3k citations
Peers
Comparison fields: 5 of 79
- Immunology and Allergy 582
- Hematology 527
- Immunology 587
- Internal Medicine 50
- Genetics 81
Countries citing papers authored by TA Springer
This map shows the geographic impact of TA Springer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by TA Springer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites TA Springer more than expected).
Fields of papers citing papers by TA Springer
This network shows the impact of papers produced by TA Springer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by TA Springer. The network helps show where TA Springer may publish in the future.
Co-authorship network
The 23 scholars most cited alongside TA Springer, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 1996 | 344 | |
| 2 | 1996 | 395 | |
| 3 | 1995 | 93 | |
| 4 | 1994 | 115 | |
| 5 | 1994 | 17 | |
| 6 | 1994 | 154 | |
| 7 | Leukocyte complement receptors and adhesion proteins in the inflammatory response: insights from an experiment of nature. | 1986 | 17 |
| 8 | 1985 | 80 | |
| 9 | The LFA-1, Mac-1 glycoprotein family and its deficiency in an inherited disease. | 1985 | 39 |
| 10 | 1985 | 11 | |
| 11 | 1983 | 2 | |
| 12 | 1983 | 92 | |
| 13 | Detergent-soluble products of the HLA region. | 1977 | 3 |
About TA Springer
TA Springer is a scholar working on Immunology and Allergy, Immunology, Hematology, Pharmaceutical Science and Genetics, having authored 13 papers that have together received 1.4k indexed citations. Recurring topics across this work include Cell Adhesion Molecules Research (6 papers), Immune Cell Function and Interaction (4 papers), Glycosylation and Glycoproteins Research (3 papers), Mast cells and histamine (3 papers), Platelet Disorders and Treatments (3 papers), Complement system in diseases (3 papers), Immunodeficiency and Autoimmune Disorders (2 papers) and Pneumocystis jirovecii pneumonia detection and treatment (2 papers). The work is most often cited by research in Immunology and Allergy (582 citations), Hematology (527 citations), Immunology (587 citations), Internal Medicine (50 citations) and Genetics (81 citations). TA Springer has collaborated with scholars based in United States, United Kingdom and Japan. Frequent co-authors include Tatsuo Kinashi, DF Bainton, Kiyoshi Takatsu, Karl Welte, Paul E. Harris, P Ralph, M A Moore, R. A. Harrison, P J Lachmann and Mark Walport. Their work appears in journals such as Blood and PubMed.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.