T. L. Dutka

1.4k citations
29 papers · 1.1k indexed · h-index 20
Topics
Ion channel regulation and function (12 papers)Muscle activation and electromyography studies (10 papers)Muscle Physiology and Disorders (9 papers)

In The Last Decade

T. L. Dutka

27 papers receiving 1.1k citations

Peers

T. L. Dutka
Comparison fields: 5 of 81
  • Molecular Biology 569
  • Physiology 394
  • Cell Biology 281
  • Rehabilitation 275
  • Biomedical Engineering 235
Replace Masanobu Wada with:
Masanobu Wada Japan
C. R. Lamboley Australia
Joshua N. Edwards Australia
Stefano Sartini Italy
James S.C. Gilchrist Canada
Steve Reiken United States
Heng Wu United States
Agnes Modin Sweden
S. R. Max United States
Jenny J. Yeh United States
T. L. Dutka relative to Masanobu Wada Japan Masanobu Wada's profile →
Citations per field
00.5×1.5×2.5×
Masanobu Wada · 1×
Citations per year

Countries citing papers authored by T. L. Dutka

Since Specialization
Citations

This map shows the geographic impact of T. L. Dutka's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by T. L. Dutka with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites T. L. Dutka more than expected).

Fields of papers citing papers by T. L. Dutka

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by T. L. Dutka. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by T. L. Dutka. The network helps show where T. L. Dutka may publish in the future.

Co-authorship network of co-authors of T. L. Dutka

This figure shows the co-authorship network connecting the top 25 collaborators of T. L. Dutka. A scholar is included among the top collaborators of T. L. Dutka based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with T. L. Dutka. T. L. Dutka is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 0
2 4
3 1
4 0
5 14
6 7
7 41
8 90
9 102
10 28
11 27
12 76
13 52
14 20
15 54
16 48
17 64
18 81
19 40
20 45

About T. L. Dutka

T. L. Dutka is a scholar working on Rehabilitation, Complementary and alternative medicine and Cellular and Molecular Neuroscience, having authored 29 papers that have together received 1.1k indexed citations. Recurring topics across this work include Ion channel regulation and function (12 papers), Muscle activation and electromyography studies (10 papers) and Muscle Physiology and Disorders (9 papers). The work is most often cited by research in Rehabilitation (275 citations), Complementary and alternative medicine (188 citations) and Cell Biology (281 citations). T. L. Dutka has collaborated with scholars based in Australia, United States and Kazakhstan. Frequent co-authors include Graham D. Lamb, Robyn M. Murphy, C. R. Lamboley, Michael J. McKenna, Giuseppe S. Posterino, Leonard J. Cole, Esther Verburg, Deanna Horvath, James R. Bell and L.M.D. Delbridge. Their work appears in journals such as PLoS ONE, The Journal of Physiology and Journal of Applied Physiology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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