T. Inaba

6.1k total citations · 1 hit paper
122 papers, 5.2k citations indexed

About

T. Inaba is a scholar working on Pharmacology, Molecular Biology and Oncology. According to data from OpenAlex, T. Inaba has authored 122 papers receiving a total of 5.2k indexed citations (citations by other indexed papers that have themselves been cited), including 55 papers in Pharmacology, 25 papers in Molecular Biology and 25 papers in Oncology. Recurrent topics in T. Inaba's work include Pharmacogenetics and Drug Metabolism (45 papers), Drug Transport and Resistance Mechanisms (24 papers) and Metabolism and Genetic Disorders (23 papers). T. Inaba is often cited by papers focused on Pharmacogenetics and Drug Metabolism (45 papers), Drug Transport and Resistance Mechanisms (24 papers) and Metabolism and Genetic Disorders (23 papers). T. Inaba collaborates with scholars based in Canada, Japan and United States. T. Inaba's co-authors include W. Kalow, S V Otton, W Kalow, Rachel F. Tyndale, David J. Stewart, M Jurima, Frank J. Gonzalez, Edward M. Sellers, Harry V. Gelboin and Urs Meyer and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of the American Chemical Society and Journal of Biological Chemistry.

In The Last Decade

T. Inaba

120 papers receiving 5.0k citations

Hit Papers

Cytochrome P-450 hPCN3, a Novel Cytochrome P-450 IIIA Gen... 1989 2026 2001 2013 1989 100 200 300 400

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
T. Inaba Canada 37 2.7k 1.3k 1.0k 899 778 122 5.2k
Felix Bochner Australia 42 1.3k 0.5× 826 0.6× 710 0.7× 1.2k 1.3× 1.1k 1.4× 162 5.3k
M. S. Lennard United Kingdom 36 2.4k 0.9× 1.1k 0.8× 774 0.7× 847 0.9× 421 0.5× 114 4.6k
Juliette Säwe Sweden 37 1.5k 0.6× 664 0.5× 555 0.5× 602 0.7× 1.4k 1.8× 77 4.5k
David E. Moody United States 41 1.1k 0.4× 516 0.4× 1.5k 1.4× 714 0.8× 779 1.0× 192 5.5k
Geoffrey T. Tucker United Kingdom 58 4.9k 1.8× 2.8k 2.1× 1.8k 1.8× 1.8k 2.0× 2.0k 2.6× 190 12.0k
Takashi Ishizaki Japan 54 4.0k 1.5× 2.2k 1.7× 1.7k 1.7× 1.3k 1.4× 1.4k 1.8× 296 10.0k
Tatsuji Iga Japan 39 1.6k 0.6× 1.8k 1.4× 1.4k 1.4× 1.1k 1.2× 830 1.1× 340 5.8k
W. Kalow Canada 57 4.0k 1.5× 1.6k 1.2× 3.2k 3.1× 2.9k 3.2× 1.2k 1.6× 237 11.1k
Kan Chiba Japan 41 2.8k 1.0× 2.0k 1.6× 1.6k 1.5× 1.0k 1.2× 891 1.1× 158 7.2k
David G. Shand United States 53 2.2k 0.8× 1.3k 1.0× 1.6k 1.6× 2.1k 2.4× 962 1.2× 150 10.6k

Countries citing papers authored by T. Inaba

Since Specialization
Citations

This map shows the geographic impact of T. Inaba's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by T. Inaba with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites T. Inaba more than expected).

Fields of papers citing papers by T. Inaba

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by T. Inaba. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by T. Inaba. The network helps show where T. Inaba may publish in the future.

Co-authorship network of co-authors of T. Inaba

This figure shows the co-authorship network connecting the top 25 collaborators of T. Inaba. A scholar is included among the top collaborators of T. Inaba based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with T. Inaba. T. Inaba is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Fukuen, Shuichi, Tsuyoshi Fukuda, Yuka Ikenaga, et al.. (2002). Novel detection assay by PCR???RFLP and frequency of the CYP3A5 SNPs, CYP3A5 *3 and *6, in a Japanese population. Pharmacogenetics. 12(4). 331–334. 155 indexed citations
2.
Fukuen, Shuichi, Tsuyoshi Fukuda, Hideyasu Matsuda, et al.. (2002). Identification of the novel splicing variants for the hPXR in human livers. Biochemical and Biophysical Research Communications. 298(3). 433–438. 53 indexed citations
3.
Fukuda, Tsuyoshi, et al.. (1999). Relationship between mRNA Levels Quantified by Reverse Transcription-Competitive PCR and Metabolic Activity of CYP3A4 and CYP2E1 in Human Liver. Biochemical and Biophysical Research Communications. 262(2). 499–503. 75 indexed citations
4.
Inaba, T., et al.. (1997). Estimation of the human hepatic microsomal cytochrome P-450 species responsible to the thrmieihadione metabolism. The Japanese Journal of Pharmacology. 73. 115–115. 4 indexed citations
5.
Otton, S V, S. Ball, Siu Wah Cheung, et al.. (1996). Venlafaxine oxidationin vitrois catalysed by CYP2D6. British Journal of Clinical Pharmacology. 41(2). 149–156. 189 indexed citations
6.
Wu, Di, S V Otton, Paul E. Morrow, et al.. (1993). Human hepatic cytochrome P450 2D6-like activity in nonhuman primates: catalytic characterization in vitro.. Journal of Pharmacology and Experimental Therapeutics. 266(2). 715–719. 21 indexed citations
7.
Sasa, Hidenori, T. Inaba, Chikako Tanaka, So Tokunaga, & Yoshinobu Wakisaka. (1990). Study on thymic remnant in the neck in rats: Effects of 6-aminonicotinamide on the incidence in rat fetuses : Abstracts of Papers Presented at the Thirtieth Annual Meeting of the Japanese Teratology Society Miyazaki, Japan, July 12-13, 1990. Congenital Anomalies. 30(3). 255–256. 1 indexed citations
8.
Muralidharan, G., K.K. Midha, G. McKay, E. M. Hawes, & T. Inaba. (1989). Selectivein vivoinhibition by quinidine of methoxyphenamine oxidation in rat models of human debrisoquine polymorphism. Xenobiotica. 19(2). 189–197. 11 indexed citations
9.
Tyndale, Rachel F., T. Inaba, & W Kalow. (1989). Evidence in humans for variant allozymes of the nondeficient sparteine/debrisoquine monooxygenase (P45OIID 1) in vitro.. Drug Metabolism and Disposition. 17(3). 334–340. 25 indexed citations
10.
Inaba, T., W. Kalow, Toshiyuki Someya, et al.. (1989). Haloperidol reduction can be assayed in human red blood cells. Canadian Journal of Physiology and Pharmacology. 67(11). 1468–1469. 26 indexed citations
11.
Inaba, T., Alan R. Tait, Masahiro Nakano, W. A. Mahon, & W Kalow. (1988). Metabolism of diazepam in vitro by human liver. Independent variability of N-demethylation and C3-hydroxylation.. Drug Metabolism and Disposition. 16(4). 605–608. 31 indexed citations
13.
Inaba, T., et al.. (1983). Human metabolism of antipyrine labelled with14C in the pyrazolone ring or in the N-methyl group. Xenobiotica. 13(3). 155–162. 17 indexed citations
14.
Inaba, T., Alexander A. Vinks, S V Otton, & W. Kalow. (1983). Comparative pharmacogenetics of sparteine and debrisoquine. Clinical Pharmacology & Therapeutics. 33(3). 394–399. 33 indexed citations
15.
Inaba, T., Haruto Uchino, D. Kadar, & W. Kalow. (1981). Antipyrine metabolites in two populations.. PubMed. 32(2). 235–44. 5 indexed citations
16.
Tang, B., T. Inaba, & W. Kalow. (1977). N-Hydroxylation of pentobarbital in man.. Drug Metabolism and Disposition. 5(1). 71–74. 13 indexed citations
17.
Mahon, W. A., et al.. (1976). Biliary elimination of diazepam in man. Clinical Pharmacology & Therapeutics. 19(4). 443–450. 22 indexed citations
18.
Mahon, W. A., Michael Mayersohn, & T. Inaba. (1976). Disposition kinetics of two oral forms of quinidine. Clinical Pharmacology & Therapeutics. 19(5part1). 566–575. 30 indexed citations
19.
Inaba, T. & W. Kalow. (1975). Salivary excretion of amobarbital in man. Clinical Pharmacology & Therapeutics. 18(5part1). 558–562. 32 indexed citations
20.
Brien, James F. & T. Inaba. (1974). Determination of low levels of 5,5-diphenylhydantoin in serum by gas-liquid chromatography. Journal of Chromatography A. 88(2). 265–270. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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