Syue‐Ting Chen

440 total citations
17 papers, 331 citations indexed

About

Syue‐Ting Chen is a scholar working on Molecular Biology, Immunology and Organic Chemistry. According to data from OpenAlex, Syue‐Ting Chen has authored 17 papers receiving a total of 331 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 7 papers in Immunology and 2 papers in Organic Chemistry. Recurrent topics in Syue‐Ting Chen's work include Glycosylation and Glycoproteins Research (9 papers), Galectins and Cancer Biology (5 papers) and Connective Tissue Growth Factor Research (2 papers). Syue‐Ting Chen is often cited by papers focused on Glycosylation and Glycoproteins Research (9 papers), Galectins and Cancer Biology (5 papers) and Connective Tissue Growth Factor Research (2 papers). Syue‐Ting Chen collaborates with scholars based in Taiwan. Syue‐Ting Chen's co-authors include Min‐Chuan Huang, Mei‐Chun Lin, Hsueh‐Fen Juan, Ting‐Chun Kuo, Pei‐Jen Lou, John Huang, Ying-Yu Liao, Yu‐Wen Tien, Chia‐Lang Hsu and Hsin‐Yi Wu and has published in prestigious journals such as Oncogene, International Journal of Molecular Sciences and American Journal of Physiology-Cell Physiology.

In The Last Decade

Syue‐Ting Chen

16 papers receiving 329 citations

Peers

Syue‐Ting Chen

IMMUN

ONCOL

Martín Enrique Rabassa Argentina

Aiko Murai Japan

Amritha Kidiyoor United States

Ashu Shah United States

Nadia Le Marer United Kingdom

Yukiko Kariya Japan

Po-Chien Chou Taiwan

Aleksandra M. Dudek-Perić Belgium

Annabelle Chow Canada

Michael Zepp Germany

Martín Enrique Rabassa Argentina

Syue‐Ting Chen

233 ×1.0

97 ×0.8

IMMUN

103 ×1.3

ONCOL

37 ×0.6

24 ×0.6

Citations per year, relative to Syue‐Ting Chen Syue‐Ting Chen (= 1×) peers Martín Enrique Rabassa

Countries citing papers authored by Syue‐Ting Chen

Since Specialization

Citations

This map shows the geographic impact of Syue‐Ting Chen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Syue‐Ting Chen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Syue‐Ting Chen more than expected).

Fields of papers citing papers by Syue‐Ting Chen

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Syue‐Ting Chen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Syue‐Ting Chen. The network helps show where Syue‐Ting Chen may publish in the future.

Co-authorship network of co-authors of Syue‐Ting Chen

This figure shows the co-authorship network connecting the top 25 collaborators of Syue‐Ting Chen. A scholar is included among the top collaborators of Syue‐Ting Chen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Syue‐Ting Chen. Syue‐Ting Chen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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17 of 17 papers shown

Chen, Syue‐Ting, Shu-Yuan Hsu, Hsin‐Ching Sung, et al.. (2025). Growth differentiation factor 15 is a glucose-downregulated gene acting as the cross talk between stroma and cancer cells of the human bladder. American Journal of Physiology-Cell Physiology. 328(2). C557–C573. 1 indexed citations

Chen, Syue‐Ting, Yu‐Hsiang Lin, Chen‐Pang Hou, et al.. (2024). Glucose Upregulates ChREBP via Phosphorylation of AKT and AMPK to Modulate MALT1 and WISP1 Expression. Journal of Cellular Physiology. 240(1). e31478–e31478. 2 indexed citations

Chen, Syue‐Ting, Shu-Yuan Hsu, Hsin‐Ching Sung, et al.. (2024). The C-X-C Motif Chemokine Ligand 5, Which Exerts an Antioxidant Role by Inducing HO-1 Expression, Is C-X-C Motif Chemokine Receptor 2-Dependent in Human Prostate Stroma and Cancer Cells. Antioxidants. 13(12). 1489–1489.

Lin, Neng‐Yu, Jian‐Jr Lee, Syue‐Ting Chen, et al.. (2023). Truncation of GalNAc-type O-glycans Suppresses CD44-mediated Osteoclastogenesis and Bone Metastasis in Breast Cancer. Molecular Cancer Research. 21(7). 664–674. 6 indexed citations

Chen, Syue‐Ting, Hsin‐Ching Sung, Shu-Yuan Hsu, et al.. (2023). Androgen Receptor Upregulates Mucosa-Associated Lymphoid Tissue 1 to Induce NF-κB Activity via Androgen-Dependent and -Independent Pathways in Prostate Carcinoma Cells. International Journal of Molecular Sciences. 24(7). 6245–6245. 6 indexed citations

Lin, Neng‐Yu, Syue‐Ting Chen, Meng‐Yao Lu, et al.. (2022). C1GALT1 expression predicts a favorable prognosis and suppresses malignant phenotypes via TrkA signaling in neuroblastoma. Oncogenesis. 11(1). 8–8. 11 indexed citations

Lin, Neng‐Yu, Syue‐Ting Chen, Ji‐Shiang Hung, et al.. (2022). GALNT2 promotes invasiveness of colorectal cancer cells partly through AXL. Molecular Oncology. 17(1). 119–133. 14 indexed citations

Hou, Chen‐Pang, Ke-Hung Tsui, Syue‐Ting Chen, et al.. (2022). The Upregulation of Caffeic Acid Phenethyl Ester on Growth Differentiation Factor 15 Inhibits Transforming Growth Factor β/Smad Signaling in Bladder Carcinoma Cells. Biomedicines. 10(7). 1625–1625. 3 indexed citations

Chen, Syue‐Ting, Hsin‐Ching Sung, Shu-Yuan Hsu, et al.. (2022). WNT1 Inducible Signaling Pathway Protein 1 Is a Stroma-Specific Secreting Protein Inducing a Fibroblast Contraction and Carcinoma Cell Growth in the Human Prostate. International Journal of Molecular Sciences. 23(19). 11437–11437. 8 indexed citations

10.

Sung, Hsin‐Ching, Syue‐Ting Chen, Shu-Yuan Hsu, et al.. (2022). Metallothionein 2A with Antioxidant and Antitumor Activity Is Upregulated by Caffeic Acid Phenethyl Ester in Human Bladder Carcinoma Cells. Antioxidants. 11(8). 1509–1509. 13 indexed citations

11.

Kuo, Ting‐Chun, Ming‐Hsun Wu, Shih‐Hung Yang, et al.. (2021). C1GALT1 high expression is associated with poor survival of patients with pancreatic ductal adenocarcinoma and promotes cell invasiveness through integrin αv. Oncogene. 40(7). 1242–1254. 29 indexed citations

12.

Lee, Po‐Chu, Syue‐Ting Chen, Ting‐Chun Kuo, et al.. (2020). C1GALT1 is associated with poor survival and promotes soluble Ephrin A1-mediated cell migration through activation of EPHA2 in gastric cancer. Oncogene. 39(13). 2724–2740. 45 indexed citations

13.

Chen, Syue‐Ting, Ting‐Chun Kuo, Ying-Yu Liao, et al.. (2018). Silencing of MUC20 suppresses the malignant character of pancreatic ductal adenocarcinoma cells through inhibition of the HGF/MET pathway. Oncogene. 37(46). 6041–6053. 41 indexed citations

14.

Lin, Mei‐Chun, Hsin‐Yi Wu, Syue‐Ting Chen, et al.. (2018). C1GALT1 predicts poor prognosis and is a potential therapeutic target in head and neck cancer. Oncogene. 37(43). 5780–5793. 54 indexed citations

15.

Lin, Mei‐Chun, et al.. (2017). Therapeutic vaccine targeting Epstein-Barr virus latent protein, LMP1, suppresses LMP1-expressing tumor growth and metastasis in vivo. BMC Cancer. 17(1). 18–18. 27 indexed citations

16.

Chen, Syue‐Ting, Min‐Chuan Huang, John Huang, et al.. (2017). GALNT6 expression enhances aggressive phenotypes of ovarian cancer cells by regulating EGFR activity. Oncotarget. 8(26). 42588–42601. 38 indexed citations

17.

Chen, Chi‐Hau, Ming‐Kwang Shyu, Shuwei Wang, et al.. (2015). MUC20 promotes aggressive phenotypes of epithelial ovarian cancer cells via activation of the integrin β1 pathway. Gynecologic Oncology. 140(1). 131–137. 33 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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