Susann Friedel
- Genetics top 5%
- Psychiatry and Mental health top 5%
- Cognitive Neuroscience top 5%
- Endocrine and Autonomic Systems top 2%
- Cellular and Molecular Neuroscience top 5%
- Co-authors
- Johannes HebebrandAnke HinneyH. SchäferAstrid DempfleHelmut RemschmidtGünter BrönnerAndré ScheragTimo D. Müller
- Topics
- Attention Deficit Hyperactivity Disorder (8 papers)Genetic Associations and Epidemiology (6 papers)Autism Spectrum Disorder Research (5 papers)
- Partner nations
- GermanyUnited KingdomUnited States
In The Last Decade
Susann Friedel
27 papers receiving 1.6k citations
Peers
Comparison fields: 5 of 84
- Genetics 491
- Psychiatry and Mental health 407
- Cognitive Neuroscience 343
- Endocrine and Autonomic Systems 325
- Cellular and Molecular Neuroscience 293
Countries citing papers authored by Susann Friedel
This map shows the geographic impact of Susann Friedel's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Susann Friedel with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Susann Friedel more than expected).
Fields of papers citing papers by Susann Friedel
This network shows the impact of papers produced by Susann Friedel. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Susann Friedel. The network helps show where Susann Friedel may publish in the future.
Co-authorship network of co-authors of Susann Friedel
This figure shows the co-authorship network connecting the top 25 collaborators of Susann Friedel. A scholar is included among the top collaborators of Susann Friedel based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Susann Friedel. Susann Friedel is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 21 | |
| 2 | 30 | |
| 3 | 18 | |
| 4 | 44 | |
| 5 | 114 | |
| 6 | 27 | |
| 7 | 26 | |
| 8 | 25 | |
| 9 | 384 | |
| 10 | 107 | |
| 11 | 32 | |
| 12 | 37 | |
| 13 | 134 | |
| 14 | 89 | |
| 15 | 123 | |
| 16 | 29 | |
| 17 | 52 | |
| 18 | 112 | |
| 19 | 53 | |
| 20 | 47 |
About Susann Friedel
Susann Friedel is a scholar working on Endocrine and Autonomic Systems, Psychiatry and Mental health and Genetics, having authored 27 papers that have together received 1.6k indexed citations. Recurring topics across this work include Attention Deficit Hyperactivity Disorder (8 papers), Genetic Associations and Epidemiology (6 papers) and Autism Spectrum Disorder Research (5 papers). The work is most often cited by research in Endocrine and Autonomic Systems (325 citations), Psychiatry and Mental health (407 citations) and Cognitive Neuroscience (343 citations). Susann Friedel has collaborated with scholars based in Germany, United Kingdom and United States. Frequent co-authors include Johannes Hebebrand, Anke Hinney, H. Schäfer, Astrid Dempfle, Helmut Remschmidt, Günter Brönner, André Scherag, Timo D. Müller, Thuy Trang Nguyen and Harald Grallert. Their work appears in journals such as PLoS ONE, The Journal of Clinical Endocrinology & Metabolism and PEDIATRICS.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.