Susan E. Waltz

4.5k total citations
114 papers, 3.3k citations indexed

About

Susan E. Waltz is a scholar working on Molecular Biology, Immunology and Hepatology. According to data from OpenAlex, Susan E. Waltz has authored 114 papers receiving a total of 3.3k indexed citations (citations by other indexed papers that have themselves been cited), including 51 papers in Molecular Biology, 31 papers in Immunology and 29 papers in Hepatology. Recurrent topics in Susan E. Waltz's work include Liver physiology and pathology (29 papers), Axon Guidance and Neuronal Signaling (21 papers) and Wnt/β-catenin signaling in development and cancer (13 papers). Susan E. Waltz is often cited by papers focused on Liver physiology and pathology (29 papers), Axon Guidance and Neuronal Signaling (21 papers) and Wnt/β-catenin signaling in development and cancer (13 papers). Susan E. Waltz collaborates with scholars based in United States, Australia and Taiwan. Susan E. Waltz's co-authors include Belinda E. Peace, Purnima K. Wagh, Sandra J. Friezner Degen, Jerilyn K. Gray, Peterson Pathrose, Glendon M. Zinser, Mike A. Leonis, Megan N. Thobe, Alex B. Lentsch and William D. Stuart and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Journal of Biological Chemistry.

In The Last Decade

Susan E. Waltz

110 papers receiving 3.2k citations

Peers

Susan E. Waltz
Comparison fields: 5 of 135
  • Molecular Biology 1.5k
  • Immunology 1.0k
  • Hepatology 832
  • Oncology 817
  • Pathology and Forensic Medicine 331
Ronald Hoffman United States
Wan Yu China
Charles S. Abrams United States
Jingsong Zhao United States
William H. Sherman United States
Peter J. Richards Switzerland
Giorgio Giacomo Galli United States
Kristiina Aittomäki Finland
Michael Ng Hong Kong
Allen Eaves Canada
Ronald Hoffman United States View profile →
Citations per field, relative to Susan E. Waltz
Susan E. Waltz · 1×
Citations per year, relative to Susan E. Waltz
Susan E. Waltz · 1×

Countries citing papers authored by Susan E. Waltz

Since Specialization
Citations

This map shows the geographic impact of Susan E. Waltz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Susan E. Waltz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Susan E. Waltz more than expected).

Fields of papers citing papers by Susan E. Waltz

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Susan E. Waltz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Susan E. Waltz. The network helps show where Susan E. Waltz may publish in the future.

Co-authorship network of co-authors of Susan E. Waltz

This figure shows the co-authorship network connecting the top 25 collaborators of Susan E. Waltz. A scholar is included among the top collaborators of Susan E. Waltz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Susan E. Waltz. Susan E. Waltz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
# Title Journal Authors Indexed citations
1 Molecular portraits of cell cycle checkpoint kinases in cancer evolution, progression, and treatment responsiveness Science Advances Aloran Mazumder, Jonathan T. Lei et al. 10
2 RON-augmented cholesterol biosynthesis in breast cancer metastatic progression and recurrence Oncogene Sara Vicente-Muñoz, Susanne I. Wells et al. 10
3 Macrophage-mediated RON signaling supports breast cancer growth and progression through modulation of IL-35 Oncogene Nancy M. Benight, Susan E. Waltz et al. 15
4 Tumor cell intrinsic RON signaling suppresses innate immune responses in breast cancer through inhibition of IRAK4 signaling Cancer Letters Nancy M. Benight, Susan E. Waltz et al. 13
5 Glutaminase inhibition with telaglenastat (CB-839) improves treatment response in combination with ionizing radiation in head and neck squamous cell carcinoma models Cancer Letters Sunil Krishnan, Kathryn E. Aziz et al. 70
6 Prostate Epithelial RON Signaling Promotes M2 Macrophage Activation to Drive Prostate Tumor Growth and Progression Molecular Cancer Research Juozas Vasiliauskas, Peterson Pathrose et al. 24
7 Defective transcription elongation in a subset of cancers confers immunotherapy resistance Nature Communications Vishnu Modur, Navneet Singh et al. 17
8 Therapeutic Considerations for Ron Receptor Expression in Prostate Cancer. Europe PMC (PubMed Central) Susan E. Waltz et al. 2
9 Tumor Cell Autonomous RON Receptor Expression Promotes Prostate Cancer Growth Under Conditions of Androgen Deprivation Neoplasia Andrew M. Paluch, Kakajan Komurov et al. 13
10 Myeloid-Specific Expression of Ron Receptor Kinase Promotes Prostate Tumor Growth Cancer Research Devikala Gurusamy, Jerilyn K. Gray et al. 45
11 Ron receptor tyrosine kinase signaling as a therapeutic target Expert Opinion on Therapeutic Targets Nancy M. Benight, Susan E. Waltz 32
12 Targeting Tyrosine Phosphorylation of PCNA Inhibits Prostate Cancer Growth Molecular Cancer Therapeutics Huajun Zhao, Yuan-Hung Lo et al. 76
13 β-Catenin is required for Ron receptor-induced mammary tumorigenesis Oncogene Purnima K. Wagh, Jerilyn K. Gray et al. 47
14 Therapeutic Implications of a Human Neutralizing Antibody to the Macrophage-Stimulating Protein Receptor Tyrosine Kinase (RON), a c-MET Family Member Cancer Research Karen E. Rabenau, Dan Lu et al. 105
15 Short-form Ron receptor is required for normal IFN-γ production in concanavalin A-induced acute liver injury American Journal of Physiology-Gastrointestinal and Liver Physiology Mike A. Leonis, Arlene E. Dent et al. 12
16 Ron-mediated cytoplasmic signaling is dispensable for viability but is required to limit inflammatory responses Journal of Clinical Investigation Susan E. Waltz, Laura Eaton et al. 5
17 Point mutations and overexpression of Ron induce transformation, tumor formation, and metastasis Oncogene Belinda E. Peace, Michael Hughes et al. 68
18 On the Universality of Human Rights Susan E. Waltz 0
19 Tunisia's League and the Pursuit of Human Rights Susan E. Waltz 2
20 Islamist appeal in Tunisia The Middle East Journal Susan E. Waltz 40

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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