Sumiko Kiryu‐Seo

3.1k total citations
64 papers, 2.5k citations indexed

About

Sumiko Kiryu‐Seo is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Oncology. According to data from OpenAlex, Sumiko Kiryu‐Seo has authored 64 papers receiving a total of 2.5k indexed citations (citations by other indexed papers that have themselves been cited), including 42 papers in Molecular Biology, 28 papers in Cellular and Molecular Neuroscience and 17 papers in Oncology. Recurrent topics in Sumiko Kiryu‐Seo's work include Neurogenesis and neuroplasticity mechanisms (17 papers), Nerve injury and regeneration (14 papers) and Signaling Pathways in Disease (11 papers). Sumiko Kiryu‐Seo is often cited by papers focused on Neurogenesis and neuroplasticity mechanisms (17 papers), Nerve injury and regeneration (14 papers) and Signaling Pathways in Disease (11 papers). Sumiko Kiryu‐Seo collaborates with scholars based in Japan, United States and United Kingdom. Sumiko Kiryu‐Seo's co-authors include Hiroshi Kiyama, Saya Nakagomi, Kazuhiko Namikawa, Bruce D. Trapp, Ryuichi Kato, Grahame J. Kidd, Nobuhiko Ohno, Hitoshi Komuro, Yasuhiro Suzuki and Kenichi Nagata and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Journal of Neuroscience.

In The Last Decade

Sumiko Kiryu‐Seo

63 papers receiving 2.5k citations

Peers

Countries citing papers authored by Sumiko Kiryu‐Seo

Since Specialization

Citations

This map shows the geographic impact of Sumiko Kiryu‐Seo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sumiko Kiryu‐Seo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sumiko Kiryu‐Seo more than expected).

Fields of papers citing papers by Sumiko Kiryu‐Seo

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sumiko Kiryu‐Seo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sumiko Kiryu‐Seo. The network helps show where Sumiko Kiryu‐Seo may publish in the future.

Co-authorship network of co-authors of Sumiko Kiryu‐Seo

This figure shows the co-authorship network connecting the top 25 collaborators of Sumiko Kiryu‐Seo. A scholar is included among the top collaborators of Sumiko Kiryu‐Seo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sumiko Kiryu‐Seo. Sumiko Kiryu‐Seo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Absence of the axon initial segment in sensory neuron enhances resistance to amyotrophic lateral sclerosis 2025 ·Brain ·(unknown), Sumiko Kiryu‐Seo, (unknown), Koji Wakatsuki, Yoshitaka Tashiro, Motosuke Tsutsumi, M. Ataka, Yohei Iguchi, Tomomi Nemoto, Ryōsuke Takahashi, Masahisa Katsuno, Hiroshi Kiyama	0
2	Impaired disassembly of the axon initial segment restricts mitochondrial entry into damaged axons 2022 ·The EMBO Journal ·Sumiko Kiryu‐Seo, (unknown), Yoshitaka Tashiro, Takeshi Yoshimura, Yohei Iguchi, Masahisa Katsuno, Ryōsuke Takahashi, Hiroshi Kiyama	16
3	Brain injury triggers cell‐type‐specific and time‐dependent endoplasmic reticulum stress responses 2022 ·Glia ·(unknown), Mika Takarada‐Iemata, (unknown), Takashi Tamatani, Hiroshi Ishii, Tsuyoshi Hattori, Sumiko Kiryu‐Seo, Hiroshi Kiyama, Osamu Hori	8
4	Axonal injury alters the extracellular glial environment of the axon initial segment and allows substantial mitochondrial influx into axon initial segment 2021 ·The Journal of Comparative Neurology ·Hiromi Tamada, Sumiko Kiryu‐Seo, (unknown), Hiroshi Kiyama	10
5	New Insights of a Neuronal Peptidase DINE/ECEL1: Nerve Development, Nerve Regeneration and Neurogenic Pathogenesis 2018 ·Neurochemical Research ·Sumiko Kiryu‐Seo, Kenichi Nagata, Takaomi C. Saido, Hiroshi Kiyama	12
6	Damage-induced neuronal endopeptidase (DINE) enhances axonal regeneration potential of retinal ganglion cells after optic nerve injury 2017 ·Cell Death and Disease ·(unknown), Sumiko Kiryu‐Seo, Sakiko Matsumoto, Hiroshi Kiyama	30
7	Three‐dimensional analysis of somatic mitochondrial dynamics in fission‐deficient injured motor neurons using FIB/SEM 2017 ·The Journal of Comparative Neurology ·Hiromi Tamada, Sumiko Kiryu‐Seo, (unknown), Keisuke Ohta, Naotada Ishihara, Masatoshi Nomura, Katsuyoshi Mihara, Kei‐ichiro Nakamura, Hiroshi Kiyama	15
8	Motor Nerve Arborization Requires Proteolytic Domain of Damage-Induced Neuronal Endopeptidase (DINE) during Development 2016 ·Journal of Neuroscience ·Sakiko Matsumoto, Sumiko Kiryu‐Seo, Hiroshi Kiyama	12
9	ECEL1 mutation implicates impaired axonal arborization of motor nerves in the pathogenesis of distal arthrogryposis 2016 ·Acta Neuropathologica ·Kenichi Nagata, Sumiko Kiryu‐Seo, Hiromi Tamada, (unknown), Hiroshi Kiyama, Takaomi C. Saido	16
10	Mitochondrial fission is an acute and adaptive response in injured motor neurons 2016 ·Scientific Reports ·Sumiko Kiryu‐Seo, Hiromi Tamada, (unknown), (unknown), Naotada Ishihara, Masatoshi Nomura, Katsuyoshi Mihara, Hiroshi Kiyama	47
11	Damage-Induced Neuronal Endopeptidase Is Critical for Presynaptic Formation of Neuromuscular Junctions 2010 ·Journal of Neuroscience ·Kenichi Nagata, Sumiko Kiryu‐Seo, Mitsuyo Maeda, Kayo Yoshida, Takashi Morita, Hiroshi Kiyama	33
12	Chronic stress elicits prolonged activation of α‐MSH secretion and subsequent degeneration of melanotroph 2009 ·Journal of Neurochemistry ·(unknown), (unknown), Hiroyuki Konishi, (unknown), Hiroyoshi Séi, Sumiko Kiryu‐Seo, Masaaki Tanaka, Yasuyoshi Watanabe, Hiroshi Kiyama	31
13	Altered expression of Smad family members in injured motor neurons of rat 2006 ·Brain Research ·Noriko Okuyama, Sumiko Kiryu‐Seo, Hiroshi Kiyama	18
14	Expression of damage‐induced neuronal endopeptidase (DINE) mRNA in peri‐infarct cortical and thalamic neurons following middle cerebral artery occlusion 2004 ·Journal of Neurochemistry ·(unknown), Sumiko Kiryu‐Seo, Mitsuyo Maeda, Michinari Muraoka, Masamitsu Ishii, Hiroshi Kiyama	25
15	Cavernous nerve injury elicits GAP-43 mRNA expression but not regeneration of injured pelvic ganglion neurons 2003 ·Brain Research ·Ryuichi Kato, Sumiko Kiryu‐Seo, Yoshikazu Sato, Shin‐ichi Hisasue, Taiji Tsukamoto, Hiroshi Kiyama	19
16	Collapsin response mediator protein‐2 accelerates axon regeneration of nerve‐injured motor neurons of rat 2003 ·Journal of Neurochemistry ·Yasuhíro Suzuki, Saya Nakagomi, Kazuhiko Namikawa, Sumiko Kiryu‐Seo, Naoyuki Inagaki, Kozo Kaibuchi, Hitoshi Aizawa, (unknown), Hiroshi Kiyama	70
17	Endothelin-converting enzymes and endothelin receptor B messenger RNAs are expressed in different neural cell species and these messenger RNAs are coordinately induced in neurons and astrocytes respectively following nerve injury 2000 ·Neuroscience ·Saya Nakagomi, Sumiko Kiryu‐Seo, Hiroshi Kiyama	58
18	Up-regulation of thioredoxin expression in motor neurons after nerve injury 1998 ·Molecular Brain Research ·(unknown), (unknown), Sumiko Kiryu‐Seo, (unknown), Kazuhiko Namikawa, Junji Yodoi, Hiroshi Kiyama	27
19	Alternative expression of Shc family members in nerve-injured motoneurons 1998 ·Molecular Brain Research ·Katsuhisa Tanabe, Sumiko Kiryu‐Seo, Takeshi Nakamura, Nozomu Mori, Hiroaki Tsujino, Takahiro Ochi, Hiroshi Kiyama	24
20	Selective Upregulation of Cytokine Receptor Subchain and Their Intracellular Signalling Molecules After Peripheral Nerve Injury 1997 ·European Journal of Neuroscience ·Gui Lan Yao, Hidemasa Kato, (unknown), Sumiko Kiryu‐Seo, Hiroshi Kiyama	45

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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